Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.
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CD1d- and MR1-Restricted T Cells in SepsisIs the developmentally immature immune response in paediatric sepsis a recapitulation of immune tolerance?Global analysis of glycoproteins identifies markers of endotoxin tolerant monocytes and GPR84 as a modulator of TNFα expression.Mitochondrial DAMPs induce endotoxin tolerance in human monocytes: an observation in patients with myocardial infarction.TLR ligands, but not modulators of histone modifiers, can induce the complex immune response pattern of endotoxin tolerance in mammary epithelial cells.Proteogenomics of selective susceptibility to endotoxin using circulating acute phase biomarkers and bioassay development in sheep: a review.Down-Regulated Receptor Interacting Protein 140 Is Involved in Lipopolysaccharide-Preconditioning-Induced Inactivation of Kupffer Cells and Attenuation of Hepatic Ischemia Reperfusion InjuryRole of the RAGE Axis during the Immune Response after Severe Trauma: A Prospective Pilot Study.Inflammasome and toll-like receptor signaling in human monocytes after successful cardiopulmonary resuscitation.Neurons and astroglia govern microglial endotoxin tolerance through macrophage colony-stimulating factor receptor-mediated ERK1/2 signals.p21 mediates macrophage reprogramming through regulation of p50-p50 NF-κB and IFN-βTLR9-mediated inflammation drives a Ccr2-independent peripheral monocytosis through enhanced extramedullary monocytopoiesis.MiR-146b Mediates Endotoxin Tolerance in Human PhagocytesA System Dynamics Model to Predict the Human Monocyte Response to Endotoxins.Advanced glycation endproducts induce self- and cross-tolerance in monocytes.The Role of IFN-β during the Course of Sepsis Progression and Its Therapeutic Potential.Impaired Surface Expression of HLA-DR, TLR2, TLR4, and TLR9 in Ex Vivo-In Vitro Stimulated Monocytes from Severely Injured Trauma Patients.The immune response of the human brain to abdominal surgery.The gut-kidney axis in chronic renal failure: A new potential target for therapy.Circulating Monocytes Exhibit an Endotoxin Tolerance Status after Acute Ischemic Stroke: Mitochondrial DNA as a Putative Explanation for Poststroke Infections.Liver - guardian, modifier and target of sepsis.Monophosphoryl lipid A induces protection against LPS in medullary thick ascending limb through a TLR4-TRIF-PI3K signaling pathway.Quantitative Assessment of Visceral Obesity and Postoperative Colon Cancer Outcomes.Expression of X-linked Toll-like receptor 4 signaling genes in female vs. male neonates.Galactomannan Downregulates the Inflammation Responses in Human Macrophages via NFκB2/p100.Spleen contributes significantly to increased circulating levels of fibroblast growth factor 23 in response to lipopolysaccharide-induced inflammation.In vivo attenuation and genetic evolution of a ST247-SCCmecI MRSA clone after 13 years of pathogenic bronchopulmonary colonization in a patient with cystic fibrosis: implications of the innate immune response.Endotoxin-induced acute lung injury in mice is protected by 5,7-dihydroxy-8-methoxyflavone via inhibition of oxidative stress and HIF-1α.Peripheral immune tolerance alleviates the intracranial lipopolysaccharide injection-induced neuroinflammation and protects the dopaminergic neurons from neuroinflammation-related neurotoxicity.Host resistance to endotoxic shock requires the neuroendocrine regulation of group 1 innate lymphoid cells.Age-related cognitive impairment is associated with long-term neuroinflammation and oxidative stress in a mouse model of episodic systemic inflammation.Endotoxin Preconditioning Reprograms S1 Tubules and Macrophages to Protect the Kidney.PD-L1 Overexpression During Endotoxin Tolerance Impairs the Adaptive Immune Response in Septic Patients via HIF1α.Altered immune responses in broiler chicken husbandry workers and their association with endotoxin exposure.Peripheral Blood Monocyte Tolerance Alleviates Intraperitoneal Lipopolysaccharides-Induced Neuroinflammation in Rats Via Upregulating the CD200R Expression.Reduced ex vivo release of pro-inflammatory cytokines and elevated plasma interleukin-6 are inflammatory signatures of post-stroke delirium.Low doses of LPS exacerbate the inflammatory response and trigger death on TLR3-primed human monocytes.Impaired Inflammatory Response to LPS in Type 2 Diabetes Mellitus.The immunosuppressive face of sepsis early on intensive care unit-A large-scale microarray meta-analysis.Thalidomide analog CC-122 induces a refractory state in monocytes from patients with diffuse large B cell lymphoma
P2860
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P2860
Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.
description
2013 nî lūn-bûn
@nan
2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.
@ast
Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.
@en
type
label
Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.
@ast
Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.
@en
prefLabel
Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.
@ast
Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.
@en
P2860
P921
P356
P1433
P1476
Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.
@en
P2093
Carlos del Fresno
Eduardo López-Collazo
P2860
P2888
P356
10.1186/CC13110
P577
2013-11-14T00:00:00Z
P5875
P6179
1035611076