Cbfβ deletion in mice recapitulates cleidocranial dysplasia and reveals multiple functions of Cbfβ required for skeletal development. - Wikidata - wikimedia - TriplyDB

Cbfβ deletion in mice recapitulates cleidocranial dysplasia and reveals multiple functions of Cbfβ required for skeletal development.

Cbfβ deletion in mice recapitulates cleidocranial dysplasia and reveals multiple functions of Cbfβ required for skeletal development.

http://www.wikidata.org/entity/Q33767459

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Role of GATA binding protein 4 (GATA4) in the regulation of tooth development via GNAI3.The silencing of cathepsin K used in gene therapy for periodontal disease reveals the role of cathepsin K in chronic infection and inflammation.Loss of osteoblast Runx3 produces severe congenital osteopenia Deletion of IFT80 Impairs Epiphyseal and Articular Cartilage Formation Due to Disruption of Chondrocyte Differentiation.Targeting Atp6v1c1 Prevents Inflammation and Bone Erosion Caused by Periodontitis and Reveals Its Critical Function in Osteoimmunology Multiple essential MT1-MMP functions in tooth root formation, dentinogenesis, and tooth eruption.Pre-Osteoblasts Stimulate Migration of Breast Cancer Cells via the HGF/MET Pathway.Ac45 silencing mediated by AAV-sh-Ac45-RNAi prevents both bone loss and inflammation caused by periodontitis.A novel, complex RUNX2 gene mutation causes cleidocranial dysplasia Odanacatib, A Cathepsin K-Specific Inhibitor, Inhibits Inflammation and Bone Loss Caused by Periodontal Diseases Class A Scavenger Receptor Exacerbates Osteoclastogenesis by an Interleukin-6-Mediated Mechanism through ERK and JNK Signaling Pathways.The RUNX family: developmental regulators in cancer.Fate of growth plate hypertrophic chondrocytes: death or lineage extension?MiR-455-3p regulates early chondrogenic differentiation via inhibiting Runx2.CCAAT/Enhancer-binding Protein α (C/EBPα) Is Important for Osteoclast Differentiation and Activity.Transcriptional network systems in cartilage development and disease.Cbfβ governs osteoblast-adipocyte lineage commitment through enhancing β-catenin signaling and suppressing adipogenesis gene expression.Platelet-rich plasma inhibits RANKL-induced osteoclast differentiation through activation of Wnt pathway during bone remodeling.C/EBPα is Regulated by the RANK Cytoplasmic IVVY535-538 Motif and Stimulates Osteoclastogenesis more Strongly than c-Fos.RUNX2 binds CBFB Cbfb2 Isoform Dominates More Potent Cbfb1 and Is Required for Skeletal Development.Core Binding Factor β Plays a Critical Role During Chondrocyte Differentiation.Effect of corticision on orthodontic tooth movement in a rat model as assessed by RNA sequencing.Roles of Runx2 in Skeletal Development.GATA4 regulates osteoblastic differentiation and bone remodeling via p38-mediated signaling.Losartan Inhibits Vascular Calcification by Suppressing the BMP2 and Runx2 Expression in Rats In Vivo.

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Cbfβ deletion in mice recapitulates cleidocranial dysplasia and reveals multiple functions of Cbfβ required for skeletal development.

http://www.wikidata.org/entity/Q33767459

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Cbfβ deletion in mice recapitu ...... ired for skeletal development.

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Cbfβ deletion in mice recapitu ...... ired for skeletal development.

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type

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Cbfβ deletion in mice recapitu ...... ired for skeletal development.

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Cbfβ deletion in mice recapitu ...... ired for skeletal development.

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Cbfβ deletion in mice recapitu ...... ired for skeletal development.

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PNAS.1310617111

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Proceedings of the National Academy of Sciences of the United States of America

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Cbfβ deletion in mice recapitu ...... uired for skeletal development

@en

P2093

Christie Paulson

http://www.w3.org/2001/XMLSchema#string

Fei Tian

http://www.w3.org/2001/XMLSchema#string

Guochun Zhu

http://www.w3.org/2001/XMLSchema#string

Joel Jules

http://www.w3.org/2001/XMLSchema#string

Junqing Ma

http://www.w3.org/2001/XMLSchema#string

Lin Wang

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Mengrui Wu

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Wei Chen

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Xuedong Zhou

http://www.w3.org/2001/XMLSchema#string

Yi-Ping Li

http://www.w3.org/2001/XMLSchema#string

P2860

Mutations involving the transcription factor CBFA1 cause cleidocranial dysplasia

Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development

Cbfbeta interacts with Runx2 and has a critical role in bone development

The core-binding factor beta subunit is required for bone formation and hematopoietic maturation

Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

Absence of fetal liver hematopoiesis in mice deficient in transcriptional coactivator core binding factor beta.

Runx2, osx, and dspp in tooth development.

ATF4 is a substrate of RSK2 and an essential regulator of osteoblast biology; implication for Coffin-Lowry Syndrome.

Mouse alpha1(I)-collagen promoter is the best known promoter to drive efficient Cre recombinase expression in osteoblast.

Core-binding factor beta interacts with Runx2 and is required for skeletal development.

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