Monomethylarsonous acid induces transformation of human bladder cells.
about
Activation of Nrf2 by arsenite and monomethylarsonous acid is independent of Keap1-C151: enhanced Keap1-Cul3 interactionDevelopmental and genetic modulation of arsenic biotransformation: a gene by environment interaction?Epigenetic changes during cell transformationInorganic arsenic and human prostate cancerNutritional status has marginal influence on the metabolism of inorganic arsenic in pregnant Bangladeshi womenBreast-feeding protects against arsenic exposure in Bangladeshi infantsArsenic exposure and cancer mortality in a US-based prospective cohort: the strong heart studyLow level exposure to monomethyl arsonous acid-induced the over-production of inflammation-related cytokines and the activation of cell signals associated with tumor progression in a urothelial cell modelInterleukin-8 (IL-8) over-production and autocrine cell activation are key factors in monomethylarsonous acid [MMA(III)]-induced malignant transformation of urothelial cellsThe Use of Chemical-Chemical Interaction and Chemical Structure to Identify New Candidate Chemicals Related to Lung CancerMethylarsonous acid causes oxidative DNA damage in cells independent of the ability to biomethylate inorganic arsenicMonomethylarsonous acid produces irreversible events resulting in malignant transformation of a human bladder cell line following 12 weeks of low-level exposureInfluence of arsenate and arsenite on signal transduction pathways: an updateEpigenetic mediated transcriptional activation of WNT5A participates in arsenical-associated malignant transformationA transgenic Drosophila model for arsenic methylation suggests a metabolic rationale for differential dose-dependent toxicity endpoints.Involvement of N-6 adenine-specific DNA methyltransferase 1 (N6AMT1) in arsenic biomethylation and its role in arsenic-induced toxicityArsenic toxicology: translating between experimental models and human pathology.Global gene expression changes in human urothelial cells exposed to low-level monomethylarsonous acid.Impact of trivalent arsenicals on selenoprotein synthesisAgglomerates of aberrant DNA methylation are associated with toxicant-induced malignant transformation.Expression of AS3MT alters transcriptional profiles in human urothelial cells exposed to arsenite.Transcriptional Modulation of the ERK1/2 MAPK and NF-κB Pathways in Human Urothelial Cells After Trivalent Arsenical Exposure: Implications for Urinary Bladder Cancer.Arsenite activates NFκB through induction of C-reactive protein.Epigenetic remodeling during arsenical-induced malignant transformation.Arsenic-based antineoplastic drugs and their mechanisms of action.Hedgehog signaling regulates bladder cancer growth and tumorigenicityExposure to monomethylarsonous acid (MMA(III)) leads to altered selenoprotein synthesis in a primary human lung cell model.Oral exposure to inorganic arsenic: evaluation of its carcinogenic and non-carcinogenic effects.Arsenicals produce stable progressive changes in DNA methylation patterns that are linked to malignant transformation of immortalized urothelial cells.Acetylated H4K16 by MYST1 protects UROtsa cells from arsenic toxicity and is decreased following chronic arsenic exposure.Requirement of arsenic biomethylation for oxidative DNA damage.Differential binding of monomethylarsonous acid compared to arsenite and arsenic trioxide with zinc finger peptides and proteins.Expression Of Selected Pathway-Marker Genes In Human Urothelial Cells Exposed Chronically To A Non-Cytotoxic Concentration Of Monomethylarsonous Acid.Oxidative DNA damage after acute exposure to arsenite and monomethylarsonous acid in biomethylation-deficient human cells.Interdependent genotoxic mechanisms of monomethylarsonous acid: role of ROS-induced DNA damage and poly(ADP-ribose) polymerase-1 inhibition in the malignant transformation of urothelial cellsCell damage and death by autoschizis in human bladder (RT4) carcinoma cells resulting from treatment with ascorbate and menadione.Persistence of DNA damage following exposure of human bladder cells to chronic monomethylarsonous acidReactive oxygen species regulate properties of transformation in UROtsa cells exposed to monomethylarsonous acid by modulating MAPK signalingThe role of reactive oxygen species in arsenite and monomethylarsonous acid-induced signal transduction in human bladder cells: acute studies.Oxidative DNA damage enhances the carcinogenic potential of in vitro chronic arsenic exposures.
P2860
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P2860
Monomethylarsonous acid induces transformation of human bladder cells.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Monomethylarsonous acid induces transformation of human bladder cells.
@ast
Monomethylarsonous acid induces transformation of human bladder cells.
@en
type
label
Monomethylarsonous acid induces transformation of human bladder cells.
@ast
Monomethylarsonous acid induces transformation of human bladder cells.
@en
prefLabel
Monomethylarsonous acid induces transformation of human bladder cells.
@ast
Monomethylarsonous acid induces transformation of human bladder cells.
@en
P2093
P2860
P1476
Monomethylarsonous acid induces transformation of human bladder cells.
@en
P2093
A Jay Gandolfi
Bhumasamudram Jagadish
Eugene A Mash
Kylee E Eblin
Tiffany G Bredfeldt
P2860
P356
10.1016/J.TAAP.2006.04.011
P577
2006-06-27T00:00:00Z