Fra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells. - Wikidata - wikimedia - TriplyDB

Fra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells.

Fra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells.

http://www.wikidata.org/entity/Q33781682

about

C-Fos expression is a molecular predictor of progression and survival in epithelial ovarian carcinoma Wound healing and inflammation genes revealed by array analysis of 'macrophageless' PU.1 null mice Regulatory pathways controlling the endovascular invasive trophoblast cell lineage Tumor suppressor p53 protein is a new target for the metastasis-associated Mts1/S100A4 protein: functional consequences of their interaction Elevated protein expression of cyclin D1 and Fra-1 but decreased expression of c-Myc in human colorectal adenocarcinomas overexpressing beta-catenin FOSL1 controls the assembly of endothelial cells into capillary tubes by direct repression of αv and β3 integrin transcription Fra-1 promotes growth and survival in RAS-transformed thyroid cells by controlling cyclin A transcription Diversity of gene expression in adenocarcinoma of the lung AP-1 differentially expressed proteins Krp1 and fibronectin cooperatively enhance Rho-ROCK-independent mesenchymal invasion by altering the function, localization, and activity of nondifferentially expressed proteins.Small molecule inhibitors targeting activator protein 1 (AP-1).c-FOS suppresses ovarian cancer progression by changing adhesion Regulation of a multigenic invasion programme by the transcription factor, AP-1: re-expression of a down-regulated gene, TSC-36, inhibits invasion.An association of a simultaneous nuclear and cytoplasmic localization of Fra-1 with breast malignancy.FRA-1 protein overexpression is a feature of hyperplastic and neoplastic breast disorders Modeling metastasis in the mouse.Emerging roles of ATF2 and the dynamic AP1 network in cancer.Characterization of Sp1, AP-1, CBF and KRC binding sites and minisatellite DNA as functional elements of the metastasis-associated mts1/S100A4 gene intronic enhancer.Dolichol-phosphate-mannose-3 (DPM3)/prostin-1 is a novel phospholipase C-gamma regulated gene negatively associated with prostate tumor invasion.Blockade of AP1 transactivation abrogates the abnormal expression of breast cancer-specific gene 1 in breast cancer cells.Transactivation of Fra-1 and consequent activation of AP-1 occur extracellular signal-regulated kinase dependently.Old players with a newly defined function: Fra-1 and c-Fos support growth of human malignant breast tumors by activating membrane biogenesis at the cytoplasm.A network of immediate early gene products propagates subtle differences in mitogen-activated protein kinase signal amplitude and duration.Role of Fos-related antigen 1 in the progression and prognosis of ductal breast carcinoma.Identify lymphatic metastasis-associated genes in mouse hepatocarcinoma cell lines using gene chip FRA-1 as a driver of tumour heterogeneity: a nexus between oncogenes and embryonic signalling pathways in cancer.Transcription factor Fos-related antigen 1 is an effective target for a breast cancer vaccine.Aberrantly expressed Fra-1 by IL-6/STAT3 transactivation promotes colorectal cancer aggressiveness through epithelial-mesenchymal transition.FOSL1 is integral to establishing the maternal-fetal interface Gene therapy for carcinoma of the breast.Pharmacology and anti-tumor activity of RWJ67657, a novel inhibitor of p38 mitogen activated protein kinase Invasion of normal human fibroblasts induced by v-Fos is independent of proliferation, immortalization, and the tumor suppressors p16INK4a and p53.Osteogenic capillaries orchestrate growth plate-independent ossification of the malleus.Identification of a pharmacologically tractable Fra-1/ADORA2B axis promoting breast cancer metastasis.Polo-like kinase 1 induces epithelial-to-mesenchymal transition and promotes epithelial cell motility by activating CRAF/ERK signaling.The PKCθ pathway participates in the aberrant accumulation of Fra-1 protein in invasive ER-negative breast cancer cells.The Bromodomain BET Inhibitor JQ1 Suppresses Tumor Angiogenesis in Models of Childhood Sarcoma A novel combination: ranpirnase and rosiglitazone induce a synergistic apoptotic effect by down-regulating Fra-1 and Survivin in cancer cells.Inhibition of AP-1 transcriptional activity blocks the migration, invasion, and experimental metastasis of murine osteosarcoma.Oral DNA vaccines target the tumor vasculature and microenvironment and suppress tumor growth and metastasis.Selective activator protein-1 inhibitor T-5224 prevents lymph node metastasis in an oral cancer model

P2860

Q24650289-75311AF4-5893-48EA-A2DD-6A18CF1B9769 Q24805609-D7F53DB0-5990-4EBD-9B66-38C11FCA2B2C Q26861033-F403BD06-BA58-441C-8D41-9D4897CF0ABF Q28208109-EC63DFB3-6B10-4D41-9921-E953EBC4F8E9 Q28220621-2FE26E57-6C76-4B49-97B4-5C3A96853014 Q28283351-A0529EA8-AE6B-4BF1-9887-988FDC3CDA0A Q28584124-CAA2978F-FBD8-47AC-8EE4-263DC15AE171 Q29616070-115FCF31-3E3E-4466-8CE7-AF1CCFA3EC30 Q30159929-13592174-0693-4D9B-8A78-F4E9045EB88E Q30398272-0222C044-FA6B-4759-9746-EFC96454A6C4 Q30570979-0DD94326-D259-4504-8B1B-E8B73B2DF8CC Q31833429-EF61BA4B-F9F5-498A-AA4C-31488127E869 Q33267756-5457C79E-43E7-4162-9174-2999C412F60D Q33270390-BD4542BE-2E1E-4727-8AC0-3F0B9E130150 Q33739285-E45B055A-3F23-4AE2-BE78-B1E1EA9BB5E4 Q33866973-4FDAEA73-EEB0-4136-A509-DF82BC9903FA Q33941301-4F24CB29-9499-4217-BBAE-256C901A4658 Q33952528-D5C22303-D97D-4082-978D-01C1D9A92F96 Q34134097-58E67CBB-497E-44E7-8204-6E2CFCD8D93B Q34440530-8AC3369B-8FB5-4A1D-A978-D1AB8F5E249B Q34542384-F1E7CD90-AFF6-4D12-95BE-7170B64A2234 Q34551716-D83F21BF-E9B6-4B64-B7C7-39BBDE6DB4BD Q34892464-D728335E-8833-4F6E-A14C-057D554FB4BC Q35010418-FCA55C7B-44F2-4053-AFED-1B2A85C4F0B5 Q35154337-1937B51F-E227-436C-BCBE-837A6626D426 Q35170949-BE8C7D4D-FD43-4B43-A742-2624172E7E02 Q35356072-802B58EE-5C60-4E55-90F4-089F598A455A Q35598846-68BB6C11-619C-41F9-B623-2ADB4749D476 Q35609061-0BEE986A-6796-47B0-A58C-C413F2E3C1DA Q36135818-C1E83441-209F-48D9-AE0C-5FBFB45ECA1F Q36232928-CEA6E3DD-9134-437B-9BD2-81B92FE85A7C Q36462661-68BEB962-BE98-4443-8118-04CEF3B6AFD9 Q36729649-C2B85A54-5B7E-4EEB-AC2D-E3D82A9D79B0 Q36739768-BA42AC18-A0DE-4E31-A57D-E60172650E19 Q36760840-88EFEEC2-E2DD-46D8-B4AC-C976790F0E80 Q36918007-5AE312BE-44E4-44B8-9F01-8C99E65EE2F1 Q36952781-F750E9D8-B32F-4A97-8037-A0B06622EF7E Q37073811-7F94AFF3-E066-44B2-BD27-BDC3777E0326 Q37118147-A254837D-02F3-41ED-9BFB-FEAE611659D9 Q37146678-0BAE7A0C-4A53-4061-8564-4A251E068EB1

P2860

Fra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells.

http://www.wikidata.org/entity/Q33781682

description

1998 nî lūn-bûn

@nan

1998 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1998 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

1998年の論文

@ja

1998年論文

@yue

1998年論文

@zh-hant

1998年論文

@zh-hk

1998年論文

@zh-mo

1998年論文

@zh-tw

1998年论文

@wuu

name

Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.

@ast

Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.

@en

type

label

Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.

@ast

Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.

@en

prefLabel

Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.

@ast

Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.

@en

P1433

Q33781682-AC73A10B-5BFB-42BA-BF88-FA992B1DF280

P1476

Q33781682-1DA472E0-D21E-46D7-8182-34737CA78068

P2093

Q33781682-358E744D-55AB-4B9B-A696-A92398B2F603

Q33781682-3F78215F-F02E-44FC-A901-F069446DA769

Q33781682-5BEBB0EA-B956-4869-B201-D18756BC4E2E

Q33781682-94BB3720-2723-4B05-9445-B823B3C66C59

Q33781682-CD812F99-D9CD-4058-9525-2AF005A5B728

Q33781682-D08AB06C-0E4A-4F45-AF6C-A7945AE8E0D1

Q33781682-FF6430F1-2D30-42CA-94F6-CAAABAB2911A

P2860

Q33781682-0192BF6C-F867-4E98-B330-43D58F22C701

Q33781682-025E16B9-8579-4428-BCAA-144E157F963E

Q33781682-0679666D-0C11-4253-855A-02E415161D24

Q33781682-086A1CE0-F64E-4442-8781-D2177465CE9B

Q33781682-0AF18A66-FD9E-40C2-A625-60CE21F03CE2

Q33781682-11B06B79-34DF-481B-A0DF-1A2150685809

Q33781682-13A1DB0D-AF64-4641-A57C-5C7A54F6130A

Q33781682-13F53714-1E46-470E-983A-5E18CB83E95C

Q33781682-15CD0EC5-2DED-446E-97BD-586802977703

Q33781682-15E6B9F1-2953-4397-BB18-703578FC287E

P304

Q33781682-BBDC4781-EEBC-4C67-B178-4E752F6F5903

P31

Q33781682-705FE316-911E-48D4-BD57-F1FFE11B61BF

P356

Q33781682-10F95236-AA9F-4F6A-9FFC-B4E5BF0A1283

P407

Q33781682-57818381-EA2B-4904-BF1D-7AF96B374068

P433

Q33781682-6CA5B743-3EE7-4DEF-8A8E-F4D6D2524FFE

P478

Q33781682-17AA1849-DF8D-4D2A-AABC-906BF1BFF870

P577

Q33781682-34611D55-084B-49D8-8542-0542B15FA2E4

P698

Q33781682-889E1A25-1EF5-4040-B335-0A736FFD537C

P921

Q33781682-9FBE4E36-BD2D-4394-9E52-D4D9C4ABAB72

P932

Q33781682-67F707A9-41DA-434B-849E-128939AC6F8E

P356

P1433

Molecular and Cellular Biology

P1476

Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.

@en

P2093

D Kramerov

http://www.w3.org/2001/XMLSchema#string

E Bock

http://www.w3.org/2001/XMLSchema#string

E Lukanidin

http://www.w3.org/2001/XMLSchema#string

E Tulchinsky

http://www.w3.org/2001/XMLSchema#string

M Grigorian

http://www.w3.org/2001/XMLSchema#string

O Kustikova

http://www.w3.org/2001/XMLSchema#string

V Berezin

http://www.w3.org/2001/XMLSchema#string

P2860

Interaction cloning: identification of a helix-loop-helix zipper protein that interacts with c-Fos

SV40-transformed human lung fibroblasts secrete a 92-kDa type IV collagenase which is identical to that secreted by normal human macrophages

A naturally occurring truncated form of FosB that inhibits Fos/Jun transcriptional activity

Cell-type specific DNA-protein interactions at the tissue-type plasminogen activator promoter in human endothelial and HeLa cells in vivo and in vitro

Cross-family dimerization of transcription factors Fos/Jun and ATF/CREB alters DNA binding specificity

Tight control of gene expression in mammalian cells by tetracycline-responsive promoters

Targeted disruption of the c-fos gene demonstrates c-fos-dependent and -independent pathways for gene expression stimulated by growth factors or oncogenes

The estrogen-dependent c-JunER protein causes a reversible loss of mammary epithelial cell polarity involving a destabilization of adherens junctions

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Cell migration: a physically integrated molecular process

P304

7095-7105

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1128/MCB.18.12.7095

http://www.w3.org/2001/XMLSchema#string

P407

P433

12

http://www.w3.org/2001/XMLSchema#string

P478

18

http://www.w3.org/2001/XMLSchema#string

P577

1998-12-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

9819396

http://www.w3.org/2001/XMLSchema#string

P921

P932

109291

http://www.w3.org/2001/XMLSchema#string