Transgenic inhibition of glial NF-kappa B reduces pain behavior and inflammation after peripheral nerve injury.
about
p75NTR is highly expressed in vestibular schwannomas and promotes cell survival by activating nuclear transcription factor κB.In vivo luminescent imaging of NF-κB activity and NF-κB-related serum cytokine levels predict pain sensitivities in a rodent model of peripheral neuropathyStat3 inhibition attenuates mechanical allodynia through transcriptional regulation of chemokine expression in spinal astrocytes.PAR2-mediated upregulation of BDNF contributes to central sensitization in bone cancer pain.The VGF-derived peptide TLQP-21 contributes to inflammatory and nerve injury-induced hypersensitivityNF-κB in the mechanism of brain edema in acute liver failure: studies in transgenic mice.Nuclear factor-κB activation in Schwann cells regulates regeneration and remyelination.Neuropathic pain-induced depressive-like behavior and hippocampal neurogenesis and plasticity are dependent on TNFR1 signalingInteractions between the immune and nervous systems in pain.Glial NF-κB inhibition alters neuropeptide expression after sciatic nerve injury in mice.Stimulation of the neurotrophin receptor TrkB on astrocytes drives nitric oxide production and neurodegenerationSpinal TLR4 mediates the transition to a persistent mechanical hypersensitivity after the resolution of inflammation in serum-transferred arthritis.CXCR3 chemokine receptor signaling mediates itch in experimental allergic contact dermatitisGlial Fibrillary Acidic Protein-Expressing Glia in the Mouse Lung.Transgenic inhibition of astroglial NF-κB protects from optic nerve damage and retinal ganglion cell loss in experimental optic neuritis.Epigenetics and the transition from acute to chronic pain.Upregulation of cystathionine beta-synthetase expression by nuclear factor-kappa B activation contributes to visceral hypersensitivity in adult rats with neonatal maternal deprivation.Enhanced binding capability of nuclear factor-κB with demethylated P2X3 receptor gene contributes to cancer pain in rats.A genetically engineered thermally responsive sustained release curcumin depot to treat neuroinflammation.Chronic pain: emerging evidence for the involvement of epigenetics.Role of astrocytes in pain.NF-κB signaling pathways: role in nervous system physiology and pathology.Molecules involved in the crosstalk between immune- and peripheral nerve Schwann cells.Withaferin A Inhibits Nuclear Factor-κB-Dependent Pro-Inflammatory and Stress Response Pathways in the Astrocytes.Gene expression profile changes in rat dorsal horn after sciatic nerve injury.Molecular Mechanisms Involved in Schwann Cell PlasticitySpinal IFN-γ-induced protein-10 (CXCL10) mediates metastatic breast cancer-induced bone pain by activation of microglia in rat models.Toll-like receptor 4-mediated nuclear factor-κB activation in spinal cord contributes to chronic morphine-induced analgesic tolerance and hyperalgesia in rats.Upregulation of CX3CL1 mediated by NF-κB activation in dorsal root ganglion contributes to peripheral sensitization and chronic pain induced by oxaliplatin administration.NF-κB subunits are differentially distributed in cells of lumbar dorsal root ganglia in naïve and diabetic rats.Astrocytes play a key role in EAE pathophysiology by orchestrating in the CNS the inflammatory response of resident and peripheral immune cells and by suppressing remyelination.Involvement of placental growth factor in Wallerian degeneration.Combination of Mangifera indica L. extract supplementation plus methotrexate in rheumatoid arthritis patients: a pilot study.Antihyperalgesic effects of an aqueous stem bark extract of Mangifera indica L.: role of mangiferin isolated from the extract.Progesterone reduces the expression of spinal cyclooxygenase-2 and inducible nitric oxide synthase and prevents allodynia in a rat model of central neuropathic pain.Epigallocatechin-3-gallate treatment reduces thermal hyperalgesia after spinal cord injury by down-regulating RhoA expression in mice.Role of A3 adenosine receptor in diabetic neuropathy.Silencing of Id2 Alleviates Chronic Neuropathic Pain Following Chronic Constriction Injury.Cl- channel is required for CXCL10-induced neuronal activation and itch response in a murine model of allergic contact dermatitis.Valproate reduces neuroinflammation and neuronal death in a rat chronic constriction injury model
P2860
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P2860
Transgenic inhibition of glial NF-kappa B reduces pain behavior and inflammation after peripheral nerve injury.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Transgenic inhibition of glial ...... after peripheral nerve injury.
@ast
Transgenic inhibition of glial ...... after peripheral nerve injury.
@en
type
label
Transgenic inhibition of glial ...... after peripheral nerve injury.
@ast
Transgenic inhibition of glial ...... after peripheral nerve injury.
@en
prefLabel
Transgenic inhibition of glial ...... after peripheral nerve injury.
@ast
Transgenic inhibition of glial ...... after peripheral nerve injury.
@en
P2093
P2860
P1433
P1476
Transgenic inhibition of glial ...... after peripheral nerve injury.
@en
P2093
Daniel J Liebl
Eugene S Fu
Jacqueline Sagen
John R Bethea
Keith A Candiotti
Paul D Morton
Roberta Brambilla
Yan Ping Zhang
P2860
P304
P356
10.1016/J.PAIN.2010.01.001
P407
P577
2010-01-22T00:00:00Z