Persistence and expression of the herpes simplex virus genome in the absence of immediate-early proteins.
about
A live-attenuated HSV-2 ICP0 virus elicits 10 to 100 times greater protection against genital herpes than a glycoprotein D subunit vaccineICP0 dismantles microtubule networks in herpes simplex virus-infected cellsHerpes simplex virus 2 ICP0 mutant viruses are avirulent and immunogenic: implications for a genital herpes vaccineICP0 antagonizes ICP4-dependent silencing of the herpes simplex virus ICP0 geneFunctional interaction between class II histone deacetylases and ICP0 of herpes simplex virus type 1PML contributes to a cellular mechanism of repression of herpes simplex virus type 1 infection that is inactivated by ICP0Functional inaccessibility of quiescent herpes simplex virus genomesProduction of immunogenic West Nile virus-like particles using a herpes simplex virus 1 recombinant vectorNeuroanatomy goes viral!The molecular basis of herpes simplex virus latencyThe potential link between PML NBs and ICP0 in regulating lytic and latent infection of HSV-1A viral E3 ligase targets RNF8 and RNF168 to control histone ubiquitination and DNA damage responsesSafety and biodistribution studies of an HSV multigene vector following intracranial delivery to non-human primates.Methods, potentials, and limitations of gene delivery to regenerate central nervous system cellsFlip-Flop HSV-BAC: bacterial artificial chromosome based system for rapid generation of recombinant herpes simplex virus vectors using two independent site-specific recombinases.ICP0 induces the accumulation of colocalizing conjugated ubiquitin.Herpes simplex virus type 1 vector-mediated expression of nerve growth factor protects dorsal root ganglion neurons from peroxide toxicityThe herpes simplex virus type 1 regulatory protein ICP27 is required for the prevention of apoptosis in infected human cells.Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycleEngineered HSV vector achieves safe long-term transgene expression in the central nervous system.ATR and ATRIP are recruited to herpes simplex virus type 1 replication compartments even though ATR signaling is disabled.Transcription of the herpes simplex virus 1 genome during productive and quiescent infection of neuronal and nonneuronal cells.Recombinant herpes simplex virus type 1 engineered for targeted binding to erythropoietin receptor-bearing cells.Long-term transgene expression in mice infected with a herpes simplex virus type 1 mutant severely impaired for immediate-early gene expression.Pseudotyping of glycoprotein D-deficient herpes simplex virus type 1 with vesicular stomatitis virus glycoprotein G enables mutant virus attachment and entry.ICP22 and the UL13 protein kinase are both required for herpes simplex virus-induced modification of the large subunit of RNA polymerase II.Perturbation of cell cycle progression and cellular gene expression as a function of herpes simplex virus ICP0.Truncation of the C-terminal acidic transcriptional activation domain of herpes simplex virus VP16 renders expression of the immediate-early genes almost entirely dependent on ICP0Herpes simplex virus triggers and then disarms a host antiviral response.Reactivation of the human cytomegalovirus major immediate-early regulatory region and viral replication in embryonal NTera2 cells: role of trichostatin A, retinoic acid, and deletion of the 21-base-pair repeats and modulator.Efficient activation of viral genomes by levels of herpes simplex virus ICP0 insufficient to affect cellular gene expression or cell survival.Herpes simplex virus ICP27 activation of stress kinases JNK and p38.Viral vectors for neurotrophic factor delivery: a gene therapy approach for neurodegenerative diseases of the CNSGene delivery with viral vectors for cerebrovascular diseases.HSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part II. Vector systems and applicationsNuclear IFI16 induction of IRF-3 signaling during herpesviral infection and degradation of IFI16 by the viral ICP0 proteinPhosphorylation of transcription factor Sp1 during herpes simplex virus type 1 infectionPan-HSV-2 IgG antibody in vaccinated mice and guinea pigs correlates with protection against herpes simplex virus 2.Pivotal roles of cGAS-cGAMP signaling in antiviral defense and immune adjuvant effectsNuclear interferon-inducible protein 16 promotes silencing of herpesviral and transfected DNA.
P2860
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P2860
Persistence and expression of the herpes simplex virus genome in the absence of immediate-early proteins.
description
1998 nî lūn-bûn
@nan
1998 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Persistence and expression of ...... e of immediate-early proteins.
@ast
Persistence and expression of ...... e of immediate-early proteins.
@en
type
label
Persistence and expression of ...... e of immediate-early proteins.
@ast
Persistence and expression of ...... e of immediate-early proteins.
@en
prefLabel
Persistence and expression of ...... e of immediate-early proteins.
@ast
Persistence and expression of ...... e of immediate-early proteins.
@en
P2093
P2860
P1433
P1476
Persistence and expression of ...... e of immediate-early proteins.
@en
P2093
L A Samaniego
L Neiderhiser
N A DeLuca
P2860
P304
P577
1998-04-01T00:00:00Z