mTORC2 phosphorylation of Akt1: a possible mechanism for hydrogen sulfide-induced cardioprotection. - Wikidata - wikimedia - TriplyDB

mTORC2 phosphorylation of Akt1: a possible mechanism for hydrogen sulfide-induced cardioprotection.

mTORC2 phosphorylation of Akt1: a possible mechanism for hydrogen sulfide-induced cardioprotection.

http://www.wikidata.org/entity/Q33783826

about

Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury.Hydrogen sulfide preconditioning protects against myocardial ischemia/reperfusion injury in rats through inhibition of endo/sarcoplasmic reticulum stress Intravenous high mobility group box 1 upregulates the expression of HIF-1α in the myocardium via a protein kinase B-dependent pathway in rats following acute myocardial ischemia.Emerging role of hydrogen sulfide-microRNA crosstalk in cardiovascular diseases.The role of hydrogen sulfide in aging and age-related pathologies Discovery of Potential Therapeutic miRNA Targets in Cardiac Ischemia-Reperfusion Injury.H2 S inhibits apo(a) expression and secretion through PKCα/FXR and Akt/HNF4α pathways in HepG2 cells.Hydrogen Sulfide (HS)-Releasing Compounds: Therapeutic Potential in Cardiovascular Diseases

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P2860

mTORC2 phosphorylation of Akt1: a possible mechanism for hydrogen sulfide-induced cardioprotection.

http://www.wikidata.org/entity/Q33783826

description

2014 nî lūn-bûn

@nan

2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

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name

mTORC2 phosphorylation of Akt1 ...... fide-induced cardioprotection.

@ast

mTORC2 phosphorylation of Akt1 ...... fide-induced cardioprotection.

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type

label

mTORC2 phosphorylation of Akt1 ...... fide-induced cardioprotection.

@ast

mTORC2 phosphorylation of Akt1 ...... fide-induced cardioprotection.

@en

prefLabel

mTORC2 phosphorylation of Akt1 ...... fide-induced cardioprotection.

@ast

mTORC2 phosphorylation of Akt1 ...... fide-induced cardioprotection.

@en

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mTORC2 phosphorylation of Akt1 ...... fide-induced cardioprotection.

@en

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Arthur Mark Richards

http://www.w3.org/2001/XMLSchema#string

Daying Wang

http://www.w3.org/2001/XMLSchema#string

Karsheng Lew

http://www.w3.org/2001/XMLSchema#string

Peipei Wang

http://www.w3.org/2001/XMLSchema#string

Xiufang Gao

http://www.w3.org/2001/XMLSchema#string

Yue Zhou

http://www.w3.org/2001/XMLSchema#string

P2860

miR-22 forms a regulatory loop in PTEN/AKT pathway and modulates signaling kinetics

Active-site inhibitors of mTOR target rapamycin-resistant outputs of mTORC1 and mTORC2

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

PHLPP and a second isoform, PHLPP2, differentially attenuate the amplitude of Akt signaling by regulating distinct Akt isoforms

Mammalian TOR complex 2 controls the actin cytoskeleton and is rapamycin insensitive

Severe diabetes, age-dependent loss of adipose tissue, and mild growth deficiency in mice lacking Akt2/PKB beta

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

Regulation of protein kinase B/Akt-serine 473 phosphorylation by integrin-linked kinase: critical roles for kinase activity and amino acids arginine 211 and serine 343

H2S as a physiologic vasorelaxant: hypertension in mice with deletion of cystathionine gamma-lyase

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6

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24949720

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phosphorylation

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4064967

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