AP-1 factors play an important role in transformation induced by the v-rel oncogene.
about
A novel interferon regulatory factor (IRF), IRF-10, has a unique role in immune defense and is induced by the v-Rel oncoprotein.Mutational analysis of the v-Rel dimerization interface reveals a critical role for v-Rel homodimers in transformationDominant negative c-jun inhibits activation of the cyclin D1 and cyclin E kinase complexesReticuloendotheliosis virus strain T induces miR-155, which targets JARID2 and promotes cell survivalRegulation of a multigenic invasion programme by the transcription factor, AP-1: re-expression of a down-regulated gene, TSC-36, inhibits invasion.Multiple mutations contribute to the oncogenicity of the retroviral oncoprotein v-Rel.Mapping of a serine-rich domain essential for the transcriptional, antiapoptotic, and transforming activities of the v-Rel oncoprotein.Interferon regulatory factor 4 contributes to transformation of v-Rel-expressing fibroblastsCell transformation by v-Rel reveals distinct roles of AP-1 family members in Rel/NF-kappaB oncogenesis.ERK and JNK activation is essential for oncogenic transformation by v-Rel.Differential regulation of the inhibitor of apoptosis ch-IAP1 by v-rel and the proto-oncogene c-rel.Epidermal growth factor receptor signaling synergizes with Hedgehog/GLI in oncogenic transformation via activation of the MEK/ERK/JUN pathway.Mutations in the v-Rel transactivation domain indicate altered phosphorylation and identify a subset of NF-kappaB-regulated cell death inhibitors important for v-Rel transforming activity.The transcription factor AP-1 is required for EGF-induced activation of rho-like GTPases, cytoskeletal rearrangements, motility, and in vitro invasion of A431 cells.Repression of B-cell linker (BLNK) and B-cell adaptor for phosphoinositide 3-kinase (BCAP) is important for lymphocyte transformation by rel proteins.CAPERalpha is a novel Rel-TAD-interacting factor that inhibits lymphocyte transformation by the potent Rel/NF-kappaB oncoprotein v-Rel.The activation of TC10, a Rho small GTPase, contributes to v-Rel-mediated transformation.Activation of gga-miR-155 by reticuloendotheliosis virus T strain and its contribution to transformation.Introduction of wild-type patched gene suppresses the oncogenic potential of human squamous cell carcinoma cell lines including A431.Characterization of ATF2 in Rel/NFκB oncogenesis reveals its role in the regulation of Ras signaling.
P2860
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P2860
AP-1 factors play an important role in transformation induced by the v-rel oncogene.
description
1998 nî lūn-bûn
@nan
1998 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
AP-1 factors play an important role in transformation induced by the v-rel oncogene.
@ast
AP-1 factors play an important role in transformation induced by the v-rel oncogene.
@en
type
label
AP-1 factors play an important role in transformation induced by the v-rel oncogene.
@ast
AP-1 factors play an important role in transformation induced by the v-rel oncogene.
@en
prefLabel
AP-1 factors play an important role in transformation induced by the v-rel oncogene.
@ast
AP-1 factors play an important role in transformation induced by the v-rel oncogene.
@en
P2093
P2860
P356
P1476
AP-1 factors play an important role in transformation induced by the v-rel oncogene.
@en
P2093
P2860
P304
P356
10.1128/MCB.18.5.2997
P407
P577
1998-05-01T00:00:00Z