Rapid host defense against Aspergillus fumigatus involves alveolar macrophages with a predominance of alternatively activated phenotype.
about
Update on the role of alternatively activated macrophages in asthmaFirst Line of Defense: Innate Cell-Mediated Control of Pulmonary AspergillosisSphingolipids as Regulators of the Phagocytic Response to Fungal InfectionsAntifungal Th Immunity: Growing up in FamilyFleA Expression in Aspergillus fumigatus Is Recognized by Fucosylated Structures on Mucins and Macrophages to Prevent Lung InfectionTH1/TH2 paradigm extended: macrophage polarization as an unappreciated pathogen-driven escape mechanism?Protective and pathogenic functions of macrophage subsetsCalcineurin Orchestrates Lateral Transfer of Aspergillus fumigatus during Macrophage Cell DeathPersistence versus escape: Aspergillus terreus and Aspergillus fumigatus employ different strategies during interactions with macrophages.Distinct innate immune phagocyte responses to Aspergillus fumigatus conidia and hyphae in zebrafish larvae.Rod-like microglia are restricted to eyes with laser-induced ocular hypertension but absent from the microglial changes in the contralateral untreated eye.Taenia taeniaeformis in rat favors protracted skin lesions caused by Sporothrix schenckii infection: Dectin-1 and IL-17 are dispensable for clearance of this fungusTemporal pattern of expression and colocalization of microglia/macrophage phenotype markers following brain ischemic injury in mice.MHC class I-specific antibody binding to nonhematopoietic cells drives complement activation to induce transfusion-related acute lung injury in miceRecruited alveolar macrophages, in response to airway epithelial-derived monocyte chemoattractant protein 1/CCl2, regulate airway inflammation and remodeling in allergic asthma.Sex differences in the response of the alveolar macrophage proteome to treatment with exogenous surfactant protein-A.Toll-like receptor-3 activation increases the vulnerability of the neonatal brain to hypoxia-ischemia.Dynamic Immune Cell Recruitment After Murine Pulmonary Aspergillus fumigatus Infection under Different Immunosuppressive Regimens.Contribution of alternatively activated macrophages to allergic lung inflammation: a tale of mice and men.Cellular immune reactions in the lung.Division of labor between lung dendritic cells and macrophages in the defense against pulmonary infections.The innate immune response to Aspergillus fumigatus at the alveolar surface.Acute Penicillium marneffei infection stimulates host M1/M2a macrophages polarization in BALB/C miceMannose receptor, C type 1 contributes to bacterial uptake by placental trophoblast giant cells.Diphenyl diselenide-modulation of macrophage activation: down-regulation of classical and alternative activation markers.IL-33 Signaling Regulates Innate IL-17A and IL-22 Production via Suppression of Prostaglandin E2 during Lung Fungal Infection.Three-dimensional confocal analysis of microglia/macrophage markers of polarization in experimental brain injury.Defective calcineurin/NFAT signaling in myeloid cells and susceptibility to aspergillosis in post-transplant patients.Isolation of mouse lung dendritic cells.Conidial Dihydroxynaphthalene Melanin of the Human Pathogenic Fungus Aspergillus fumigatus Interferes with the Host Endocytosis Pathway.Interleukin-33 drives activation of alveolar macrophages and airway inflammation in a mouse model of acute exacerbation of chronic asthma.The predominance of alternatively activated macrophages following challenge with cell wall peptide-polysaccharide after prior infection with Sporothrix schenckii.Macrophage polarization and allergic asthma.Tissue-Resident Macrophages in Fungal Infections.Organ-specific mechanisms linking innate and adaptive antifungal immunity.Host response to pulmonary fungal infections: A highlight on cell-driven immunity to Cryptococcus species and Aspergillus fumigatus.Differential strain-related tissue immune response to sublethal systemic Aspergillus fumigatus infection in mice.Acidic mammalian chitinase negatively affects immune responses during acute and chronic Aspergillus fumigatus exposure.Renal allograft recipients fail to increase interferon-γ during invasive fungal diseases.Macrophages inhibit Aspergillus fumigatus germination and neutrophil-mediated fungal killing
P2860
Q26746898-1EF25A43-F387-4018-8037-129D12A497D0Q26765933-BBF6C5C2-AD95-4B5F-9064-9A51703C1EB9Q26774635-792D3282-2AE7-48C9-ABBB-878F1D80BD5CQ27002920-BE5A6073-105A-427E-8CA0-45BEBC598097Q27314890-D2A63DD3-C055-42EF-AC26-E9303C6D5268Q27693193-A94500F2-1BD7-4ADD-B5F2-C46AC428D431Q29620351-9515D4C1-D455-4915-A66D-C3CA6929DA49Q30829038-4E67F177-C61F-4201-9A58-A7A30C9739D3Q34154790-C7303BCB-7149-4737-A421-D653BFD53D52Q34297696-A65F1B02-3889-46A4-B84C-3AAC476991C7Q34394275-CBE69AF9-D09D-46D3-BE72-EC2D4AE9015EQ34534019-A609F288-0EDB-4CCD-8F63-1CF8BE3E133AQ35652529-319AEA24-3AC4-420C-BD9F-7B3E03430D7CQ35669060-9C4EB65B-2CFE-4829-9679-0613AB9DBC7FQ35816421-6737E83C-8411-4037-B3DD-5227869C97B0Q36604498-F1D2EF4B-2724-458A-BDC4-3BAC699D36B9Q37020230-03DADC65-50B5-4B8E-8365-1407F2A9B4F1Q37088821-60ACC993-B94F-4D3D-B353-C0D26A7E771BQ37996251-5195185C-6D7B-4004-A3A6-16184320B9D4Q38070793-86D3209E-DCB4-4C68-8F3F-D058CB5227CDQ38095347-76F04F61-D929-4402-81A5-0D7C3919ADC7Q38454116-38D1428D-EC3B-4C67-9B90-6785637D8E36Q38618231-F716FB3E-2662-44F5-BDD1-74F27B0DCF3DQ39242561-F39E5742-7BE7-4750-BCE1-9CA40286F8FBQ39418089-F4F80412-9FC8-42D0-B228-D7BF5CD3719EQ40103200-5D5F6E33-F693-416C-9B2E-4B38FC00A278Q41976314-CEF14964-B352-475E-A59C-0E628272D310Q41996998-6D6C3C59-3829-4E44-9C95-47F42669CCD9Q42019096-2F533ECF-75D8-4A37-9099-23CDFD507404Q42085114-14789102-A0F9-4631-AA57-F249DBF37AF1Q42181941-36A64668-06E1-4315-B9F9-B0D0B5A6AC0AQ44694227-9634385F-20A2-4967-95BF-DA8687351998Q47608848-630535EA-146C-4C6E-B7CA-B5EEC75EE281Q47663189-58DF81F5-268E-4A42-B37B-74CA7A649BFFQ49795904-BA3B0D89-D6DD-439A-B951-D4C1B8F9DE0FQ50073021-BA279433-A90D-402B-945D-97027E63FAA7Q51548827-218BF2B2-8052-49B3-B2DE-671FD3E80FE1Q53689793-9D118F39-7188-4CED-B98B-9312CB1E3095Q54292395-28DEFD12-3193-4EA8-A8B0-983BC513D042Q58803073-405B657F-49FC-4926-A474-AA5D31477766
P2860
Rapid host defense against Aspergillus fumigatus involves alveolar macrophages with a predominance of alternatively activated phenotype.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Rapid host defense against Asp ...... rnatively activated phenotype.
@ast
Rapid host defense against Asp ...... rnatively activated phenotype.
@en
type
label
Rapid host defense against Asp ...... rnatively activated phenotype.
@ast
Rapid host defense against Asp ...... rnatively activated phenotype.
@en
prefLabel
Rapid host defense against Asp ...... rnatively activated phenotype.
@ast
Rapid host defense against Asp ...... rnatively activated phenotype.
@en
P2093
P2860
P50
P1433
P1476
Rapid host defense against Asp ...... rnatively activated phenotype.
@en
P2093
Anuradha Ray
Claudette M St Croix
Gregory A Gibson
Manohar Yarlagadda
Mingjian Fei
Prabir Ray
Shikha Bhatia
Shinobu Saijo
Zengbiao Qi
P2860
P304
P356
10.1371/JOURNAL.PONE.0015943
P407
P577
2011-01-05T00:00:00Z