Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver - Wikidata - wikimedia - TriplyDB

Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver

Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver

http://www.wikidata.org/entity/Q33799804

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The redox state of free nicotinamide-adenine dinucleotide phosphate in the cytoplasm of rat liver The impact of obesity, sex, and diet on hepatic glucose production in cats A general introduction to the biochemistry of mitochondrial fatty acid β-oxidation Dihydroxyacetone phosphate. Its structure and reactivity with -glycerophosphate dehydrogenase, aldolase and triose phosphate isomerase and some possible metabolic implications Mechanisms of fatty acid-induced inhibition of glucose uptake Fatty acids-stress attenuates gluconeogenesis induction and glucose production in primary hepatocytes Transplantation of non-visceral fat to the visceral cavity improves glucose tolerance in mice: investigation of hepatic lipids and insulin sensitivity.The effects of free fatty acids on gluconeogenesis and glycogenolysis in normal subjects.Demonstration of a critical role for free fatty acids in mediating counterregulatory stimulation of gluconeogenesis and suppression of glucose utilization in humans.Adrenergic mechanisms contribute to the late phase of hypoglycemic glucose counterregulation in humans by stimulating lipolysis.Liver and kidney metabolism during prolonged starvation Acute effects of insulin-like growth factor I on glucose and amino acid metabolism in the awake fasted rat. Comparison with insulin.Glucose and free fatty acid metabolism in non-insulin-dependent diabetes mellitus. Evidence for multiple sites of insulin resistance Role of hepatic autoregulation in defense against hypoglycemia in humans.Removal of intra-abdominal visceral adipose tissue improves glucose tolerance in rats: role of hepatic triglyceride storage.Increased lipolysis and its consequences on gluconeogenesis in non-insulin-dependent diabetes mellitus.Mechanism of increased gluconeogenesis in noninsulin-dependent diabetes mellitus. Role of alterations in systemic, hepatic, and muscle lactate and alanine metabolism Lower body adipose tissue removal decreases glucose tolerance and insulin sensitivity in mice with exposure to high fat diet.Elevated TCA cycle function in the pathology of diet-induced hepatic insulin resistance and fatty liver.Interrelations between hepatic fatty acid oxidation and gluconeogenesis: a possible regulatory role of carnitine palmityltransferase.Profound hypoglycemia in starved, ghrelin-deficient mice is caused by decreased gluconeogenesis and reversed by lactate or fatty acids.Dynamic modeling of methylprednisolone effects on body weight and glucose regulation in rats.PKM2 phosphorylates histone H3 and promotes gene transcription and tumorigenesis Combined intervention with pioglitazone and n-3 fatty acids in metformin-treated type 2 diabetic patients: improvement of lipid metabolism.Studies of the mechanism of the hypoglycemic action of 4-pentenoic acid.Metabolic alterations following visceral fat removal and expansion: Beyond anatomic location.Partial resistance to peroxisome proliferator-activated receptor-alpha agonists in ZDF rats is associated with defective hepatic mitochondrial metabolism.Effect of fatty acids on glucose production and utilization in man.Banting Lecture. From the triumvirate to the ominous octet: a new paradigm for the treatment of type 2 diabetes mellitus Inhibiting gluconeogenesis prevents fatty acid-induced increases in endogenous glucose production Causal linkage between insulin suppression of lipolysis and suppression of liver glucose output in dogs.Comparison of the direct and indirect effects of epinephrine on hepatic glucose production.Indirect effect of insulin to suppress endogenous glucose production is dominant, even with hyperglucagonemia.Caloric restriction reverses hepatic insulin resistance in aging rats by decreasing visceral fat.Abnormal renal and hepatic glucose metabolism in type 2 diabetes mellitus.Getting the message across: mechanisms of physiological cross talk by adipose tissue.CEACAM1 loss links inflammation to insulin resistance in obesity and non-alcoholic steatohepatitis (NASH).Integrated Regulation of Hepatic Lipid and Glucose Metabolism by Adipose Triacylglycerol Lipase and FoxO Proteins.Insulin therapy in maturity-onset diabetes.Abdominal obesity and risk.

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P2860

Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver

http://www.wikidata.org/entity/Q33799804

description

1966 nî lūn-bûn

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1966 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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1966 թվականի հուլիսին հրատարակված գիտական հոդված

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1966年の論文

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1966年論文

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1966年論文

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1966年論文

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1966年論文

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1966年論文

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1966年论文

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Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver

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Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver

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Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver

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Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver

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Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver

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Proceedings of the National Academy of Sciences of the United States of America

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Mechanism for the stimulation of gluconeogenesis by fatty acids in perfused rat liver

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Felts PW

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Kreisberg RA

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Williamson JR

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P2860

Palmityl-coenzyme A inhibition of the citrate-condensing enzyme

The dominant role of the liver in plasma protein synthesis; a direct study of the isolated perfused rat liver with the aid of lysine-epsilon-C14.

Paths of carbon in gluconeogenesis and lipogenesis: the role of mitochondria in supplying precursors of phosphoenolpyruvate

The effects of adenine nucleotides on carbohydrate metabolism in pigeon-liver homogenates.

SOME PROPERTIES OF FRUCTOSE 1,6-DIPHOSPHATASE OF RAT LIVER AND THEIR RELATION TO THE CONTROL OF GLUCONEOGENESIS.

THE CROONIAN LECTURE, 1963. GLUCONEOGENESIS.

The effect of palmityl coenzyme A on glucose 6-phosphate dehydrogenase and other enzymes.

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247-254

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scholarly article

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