Transcription factor Smad3 is required for the inhibition of adipogenesis by retinoic acid. - Wikidata - wikimedia - TriplyDB

Transcription factor Smad3 is required for the inhibition of adipogenesis by retinoic acid.

Transcription factor Smad3 is required for the inhibition of adipogenesis by retinoic acid.

http://www.wikidata.org/entity/Q33800199

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Getting 'Smad' about obesity and diabetes The Role of Vitamins in the Pathogenesis of Non-alcoholic Fatty Liver Disease TGF-β Negatively Regulates CXCL1 Chemokine Expression in Mammary Fibroblasts through Enhancement of Smad2/3 and Suppression of HGF/c-Met Signaling Mechanisms Retinoids Regulate Adipogenesis Involving the TGFβ/SMAD and Wnt/β-Catenin Pathways in Human Bone Marrow Mesenchymal Stem Cells Maternal obesity downregulates microRNA let-7g expression, a possible mechanism for enhanced adipogenesis during ovine fetal skeletal muscle development Transcriptional analysis of abdominal fat in genetically fat and lean chickens reveals adipokines, lipogenic genes and a link between hemostasis and leanness.Deficiency of retinaldehyde dehydrogenase 1 induces BMP2 and increases bone mass in vivo.Retinoic acid has different effects on UCP1 expression in mouse and human adipocytes.Current methods of adipogenic differentiation of mesenchymal stem cells The contribution of vitamin A to autocrine regulation of fat depots.Retinoic acid promotes myogenesis in myoblasts by antagonizing transforming growth factor-beta signaling via C/EBPβ Noggin is novel inducer of mesenchymal stem cell adipogenesis: implications for bone health and obesity.Retinoic acid upregulates preadipocyte genes to block adipogenesis and suppress diet-induced obesity.Diabetic mice exhibited a peculiar alteration in body composition with exaggerated ectopic fat deposition after muscle injury due to anomalous cell differentiation.Retinol-binding protein 4 and its membrane receptor STRA6 control adipogenesis by regulating cellular retinoid homeostasis and retinoic acid receptor α activity Retinoids regulate stem cell differentiation Carotenoids in Adipose Tissue Biology and Obesity.Wnt/β-Catenin Mediates AICAR Effect to Increase GATA3 Expression and Inhibit Adipogenesis.Inhibition of ASCT2 is essential in all-trans retinoic acid-induced reduction of adipogenesis in 3T3-L1 cells.The one-two punch: Retinoic acid suppresses obesity both by promoting energy expenditure and by inhibiting adipogenesis.Retinoic acid modulates lipid accumulation glucose concentration dependently through inverse regulation of SREBP-1 expression in 3T3L1 adipocytes.Retinoid X Receptor Activation Alters the Chromatin Landscape To Commit Mesenchymal Stem Cells to the Adipose Lineage.Non-classical Transcriptional Activity of Retinoic Acid.Combined effect of retinoic acid and calcium on the in vitro differentiation of human adipose-derived stem cells to adipocytes.Tissue Expression Analysis and Characterization of Smad3 Promoter in Bovine Myoblasts and Preadipocytes.

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Transcription factor Smad3 is required for the inhibition of adipogenesis by retinoic acid.

http://www.wikidata.org/entity/Q33800199

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Transcription factor Smad3 is ...... adipogenesis by retinoic acid.

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Transcription factor Smad3 is ...... adipogenesis by retinoic acid.

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type

label

Transcription factor Smad3 is ...... adipogenesis by retinoic acid.

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Transcription factor Smad3 is ...... adipogenesis by retinoic acid.

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prefLabel

Transcription factor Smad3 is ...... adipogenesis by retinoic acid.

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Transcription factor Smad3 is ...... adipogenesis by retinoic acid.

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JBC.M109.054536

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Journal of Biological Chemistry

P1476

Transcription factor Smad3 is ...... adipogenesis by retinoic acid.

@en

P2093

Catherine St-Louis

http://www.w3.org/2001/XMLSchema#string

François Marchildon

http://www.w3.org/2001/XMLSchema#string

Nadine L Wiper-Bergeron

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Rahima Akter

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Victoria Roodman

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P2860

Glucocorticoid-stimulated preadipocyte differentiation is mediated through acetylation of C/EBPbeta by GCN5

Receptor-associated Mad homologues synergize as effectors of the TGF-beta response

Defective adipocyte differentiation in mice lacking the C/EBPbeta and/or C/EBPdelta gene.

CCAAT/enhancer-binding proteins: structure, function and regulation

Transforming growth factor beta-induced phosphorylation of Smad3 is required for growth inhibition and transcriptional induction in epithelial cells

Regulated expression of three C/EBP isoforms during adipose conversion of 3T3-L1 cells

Retinoic acid regulates C/EBP homologous protein expression (CHOP), which negatively regulates myeloid target genes

An essential role for C/EBPbeta in female reproduction

Retinoic acid repressed the expression of c-fos and c-jun and induced apoptosis in regenerating rat liver after partial hepatectomy

CCAAT enhancer- binding protein beta is required for normal hepatocyte proliferation in mice after partial hepatectomy

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13274-13284

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scholarly article

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10.1074/JBC.M109.054536

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17

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285

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2010-02-23T00:00:00Z

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20179325

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2857127

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