Functional characterization of aquaporin-4 specific T cells: towards a model for neuromyelitis optica
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Change in autoantibody and cytokine responses during the evolution of neuromyelitis optica in patients with systemic lupus erythematosus: A preliminary studyImmunology of neuromyelitis optica: a T cell-B cell collaborationNeuromyelitis optica: clinical features, immunopathogenesis and treatment.Experimental models of neuromyelitis optica.Early loss of oligodendrocytes in human and experimental neuromyelitis optica lesions.Update on the diagnosis and treatment of neuromyelitis optica: recommendations of the Neuromyelitis Optica Study Group (NEMOS).New insights into neuromyelitis opticaPathogenic aquaporin-4 reactive T cells are sufficient to induce mouse model of neuromyelitis optica.Variants of Interleukin-7/Interleukin-7 Receptor Alpha are Associated with Both Neuromyelitis Optica and Multiple Sclerosis Among Chinese Han Population in Southeastern ChinaA Single Amino Acid Substitution Prevents Recognition of a Dominant Human Aquaporin-4 Determinant in the Context of HLA-DRB1*03:01 by a Murine TCR.Aquaporin 4-specific T cells in neuromyelitis optica exhibit a Th17 bias and recognize Clostridium ABC transporterHighly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNS.Epigallocatechin-3-gallate: a useful, effective and safe clinical approach for targeted prevention and individualised treatment of neurological diseases?Aquaporin 4 molecular mimicry and implications for neuromyelitis optica.Human aquaporin 4281-300 is the immunodominant linear determinant in the context of HLA-DRB1*03:01: relevance for diagnosing and monitoring patients with neuromyelitis optica.Tolerance checkpoint bypass permits emergence of pathogenic T cells to neuromyelitis optica autoantigen aquaporin-4.Review of Animal Models of Neuromyelitis Optica.Deletional tolerance prevents AQP4-directed autoimmunity in mice.Pathogenic and physiological autoantibodies in the central nervous system.Experimental models of neuromyelitis optica: current status, challenges and future directions.Müller cells and retinal axons can be primary targets in experimental neuromyelitis optica spectrum disorder.Pathogenic T cell responses against aquaporin 4.T cells targeting neuromyelitis optica autoantigen aquaporin-4 cause paralysis and visual system injury.Induction of Paralysis and Visual System Injury in Mice by T Cells Specific for Neuromyelitis Optica Autoantigen Aquaporin-4.Where Do AQP4 Antibodies Fit in the Pathogenesis of NMO?Neuromyelitis optica
P2860
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P2860
Functional characterization of aquaporin-4 specific T cells: towards a model for neuromyelitis optica
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
name
Functional characterization of ...... model for neuromyelitis optica
@ast
Functional characterization of ...... model for neuromyelitis optica
@en
type
label
Functional characterization of ...... model for neuromyelitis optica
@ast
Functional characterization of ...... model for neuromyelitis optica
@en
prefLabel
Functional characterization of ...... model for neuromyelitis optica
@ast
Functional characterization of ...... model for neuromyelitis optica
@en
P2093
P2860
P1433
P1476
Functional characterization of ...... model for neuromyelitis optica
@en
P2093
Bernhard Hemmer
Franziska Petermann
Marco Prinz
Rajneesh Srivastava
Sudhakar Reddy Kalluri
Veit Rothhammer
P2860
P304
P356
10.1371/JOURNAL.PONE.0016083
P407
P577
2011-01-14T00:00:00Z