Anaphylatoxins Activate Ca2+, Akt/PI3-Kinase, and FOXO1/FoxP3 in the Retinal Pigment Epithelium - Wikidata - wikimedia - TriplyDB

Anaphylatoxins Activate Ca2+, Akt/PI3-Kinase, and FOXO1/FoxP3 in the Retinal Pigment Epithelium

Anaphylatoxins Activate Ca2+, Akt/PI3-Kinase, and FOXO1/FoxP3 in the Retinal Pigment Epithelium

http://www.wikidata.org/entity/Q33802700

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C3a triggers formation of sub-retinal pigment epithelium deposits via the ubiquitin proteasome pathway.

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Anaphylatoxins Activate Ca2+, Akt/PI3-Kinase, and FOXO1/FoxP3 in the Retinal Pigment Epithelium

http://www.wikidata.org/entity/Q33802700

description

2017 nî lūn-bûn

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2017 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի հունիսին հրատարակված գիտական հոդված

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

@zh-my

2017年学术文章

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2017年學術文章

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name

Anaphylatoxins Activate Ca2+, ...... the Retinal Pigment Epithelium

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Anaphylatoxins Activate Ca2+, ...... the Retinal Pigment Epithelium

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type

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Anaphylatoxins Activate Ca2+, ...... the Retinal Pigment Epithelium

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Anaphylatoxins Activate Ca2+, ...... the Retinal Pigment Epithelium

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Anaphylatoxins Activate Ca2+, ...... the Retinal Pigment Epithelium

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Anaphylatoxins Activate Ca2+, ...... the Retinal Pigment Epithelium

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FIMMU.2017.00703

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Frontiers in Immunology

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Anaphylatoxins Activate Ca2+, ...... the Retinal Pigment Epithelium

@en

P2093

Balasubramaniam Annamalai

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Bärbel Rohrer

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Catharina Busch

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Christian Huber

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Christine Skerka

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Emeraldo A H Jo

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Gerhild Wildner

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Khava Abdusalamova

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Maria Diedrichs-Möhring

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Nadine Reichhart

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P2860

The CD46-Jagged1 interaction is critical for human TH1 immunity

Sublytic membrane-attack-complex (MAC) activation alters regulated rather than constitutive vascular endothelial growth factor (VEGF) secretion in retinal pigment epithelium monolayers

An integrated hypothesis that considers drusen as biomarkers of immune-mediated processes at the RPE-Bruch's membrane interface in aging and age-related macular degeneration

Regulation of L-type inward calcium channel activity by captopril and angiotensin II via the phosphatidyl inositol 3-kinase pathway in cardiomyocytes from volume-overload hypertrophied rat hearts

Estimation of systemic complement C3 activity in age-related macular degeneration.

Paracrine modulation of CXCR4 by IGF-1 and VEGF: implications for choroidal neovascularization.

Molecular composition of drusen as related to substructural phenotype.

Regional immunity and ocular immune privilege.

The alternative pathway is required, but not alone sufficient, for retinal pathology in mouse laser-induced choroidal neovascularization.

Absence of signaling into CD4⁺ cells via C3aR and C5aR enables autoinductive TGF-β1 signaling and induction of Foxp3⁺ regulatory T cells.

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703

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scholarly article

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10.3389/FIMMU.2017.00703

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8

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2017-06-15T00:00:00Z

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28663750

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5472091

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