Parallel increases in striatal glutamic acid decarboxylase activity and mRNA levels in rats with lesions of the nigrostriatal pathway.
about
Colocalization of tyrosine hydroxylase and GAD65 mRNA in mesostriatal neurons.Increased GABAergic tone in the ventromedial hypothalamus contributes to suppression of counterregulatory responses after antecedent hypoglycemiaIncreased GAD expression in the striatum after transient cerebral ischemiaEstradiol alters only GAD67 mRNA levels in ischemic rat brain with no consequent effects on GABA.Dopaminergic development of prenatal ventral mesencephalon and striatum in organotypic co-cultures.Unilateral 6-hydroxydopamine lesion of dopamine neurons and subchronic L-DOPA administration in the adult rat alters the expression of the vesicular GABA transporter in different subsets of striatal neurons and in the substantia nigra, pars reticulaGlutamic acid decarboxylase 65 and 67 expression in the lateral septum is up-regulated in association with the postpartum period in miceGABA-opioid interactions in the globus pallidus: [D-Ala2]-Met-enkephalinamide attenuates potassium-evoked GABA release after nigrostriatal lesion.Increased intracellular gamma-aminobutyric acid selectively lowers the level of the larger of two glutamate decarboxylase proteins in cultured GABAergic neurons from rat cerebral cortex.A transcriptional regulatory element of the gene encoding the 67,000-M(r) form of human glutamate decarboxylase is similar to a Drosophila regulatory element.Glutamate decarboxylases in nonneural cells of rat testis and oviduct: differential expression of GAD65 and GAD67.GABA signalling: therapeutic targets for epilepsy, Parkinson's disease and Huntington's disease.Neuroprotection of cultured foetal rat hippocampal cells against glucose deprivation: are GABAergic neurons less vulnerable or more sensitive to TCP protection?Effects of increased gamma-aminobutyric acid levels on GAD67 protein and mRNA levels in rat cerebral cortex.An in vivo ultrahigh field 14.1 T (1) H-MRS study on 6-OHDA and α-synuclein-based rat models of Parkinson's disease: GABA as an early disease marker.Glutamate decarboxylase (GAD67 and GAD65) gene expression is increased in a subpopulation of neurons in the putamen of Parkinsonian monkeys.Modulation of Corpus Striatal Neurochemistry by Astrocytes and Vasoactive Intestinal Peptide (VIP) in Parkinsonian Rats.Transcriptional organization of a Drosophila glutamic acid decarboxylase gene.Inhibition of mitochondrial oxidative phosphorylation induces hyper-expression of glutamic acid decarboxylase in pancreatic islet cells.
P2860
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P2860
Parallel increases in striatal glutamic acid decarboxylase activity and mRNA levels in rats with lesions of the nigrostriatal pathway.
description
1990 nî lūn-bûn
@nan
1990 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Parallel increases in striatal ...... of the nigrostriatal pathway.
@ast
Parallel increases in striatal ...... of the nigrostriatal pathway.
@en
type
label
Parallel increases in striatal ...... of the nigrostriatal pathway.
@ast
Parallel increases in striatal ...... of the nigrostriatal pathway.
@en
prefLabel
Parallel increases in striatal ...... of the nigrostriatal pathway.
@ast
Parallel increases in striatal ...... of the nigrostriatal pathway.
@en
P2093
P1433
P1476
Parallel increases in striatal ...... of the nigrostriatal pathway.
@en
P2093
P304
P356
10.1016/0006-8993(90)90849-7
P407
P577
1990-10-01T00:00:00Z