Silencing of the JNK pathway maintains progesterone receptor activity in decidualizing human endometrial stromal cells exposed to oxidative stress signals.
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Induction of 11β-HSD 1 and activation of distinct mineralocorticoid receptor- and glucocorticoid receptor-dependent gene networks in decidualizing human endometrial stromal cells.NADPH oxidase-derived reactive oxygen species mediate decidualization of human endometrial stromal cells in response to cyclic AMP signaling.The SUMO E3-ligase PIAS1 couples reactive oxygen species-dependent JNK activation to oxidative cell deathThe clock protein period 2 synchronizes mitotic expansion and decidual transformation of human endometrial stromal cellsProgesterone and the Repression of Myometrial Inflammation: The Roles of MKP-1 and the AP-1 SystemInflammatory mediators increase SUMOylation of retinoid X receptor α in a c-Jun N-terminal kinase-dependent manner in human hepatocellular carcinoma cells.Progesterone-Dependent Induction of Phospholipase C-Related Catalytically Inactive Protein 1 (PRIP-1) in Decidualizing Human Endometrial Stromal Cells.Prostaglandin E2 (PGE2) promotes proliferation and invasion by enhancing SUMO-1 activity via EP4 receptor in endometrial cancer.Possible involvement of CD10 in the development of endometriosis due to its inhibitory effects on CD44-dependent cell adhesion.Loss of miR-542-3p enhances IGFBP-1 expression in decidualizing human endometrial stromal cellsProgesterone action in the myometrium and decidua in preterm birthThe molecular basis of recurrent pregnancy loss: impaired natural embryo selection.The role of FOXO1 in the decidual transformation of the endometrium and early pregnancy.The motile and invasive capacity of human endometrial stromal cells: implications for normal and impaired reproductive function.Retinoic acid stimulation of VEGF secretion from human endometrial stromal cells is mediated by production of reactive oxygen species.NLRP7 contributes to in vitro decidualization of endometrial stromal cells.Androgens modulate the morphological characteristics of human endometrial stromal cells decidualized in vitro.Overexpression of microRNA-542-3p attenuates the differentiating capacity of endometriotic stromal cells.Crosstalk between human endometrial stromal cells and decidual NK cells promotes decidualization in vitro by upregulating IL‑25.Analysis of chromatin accessibility in decidualizing human endometrial stromal cells.Loss of high-mobility group N5 contributes to the promotion of human endometrial stromal cell decidualizationA novel and compact review on the role of oxidative stress in female reproduction
P2860
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P2860
Silencing of the JNK pathway maintains progesterone receptor activity in decidualizing human endometrial stromal cells exposed to oxidative stress signals.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Silencing of the JNK pathway m ...... d to oxidative stress signals.
@ast
Silencing of the JNK pathway m ...... d to oxidative stress signals.
@en
type
label
Silencing of the JNK pathway m ...... d to oxidative stress signals.
@ast
Silencing of the JNK pathway m ...... d to oxidative stress signals.
@en
prefLabel
Silencing of the JNK pathway m ...... d to oxidative stress signals.
@ast
Silencing of the JNK pathway m ...... d to oxidative stress signals.
@en
P2093
P2860
P50
P356
P1433
P1476
Silencing of the JNK pathway m ...... d to oxidative stress signals.
@en
P2093
Beatriz Leitao
Jenny Higham
Marius C Jones
Masashi Takano
Tomoko Goto
P2860
P304
P356
10.1096/FJ.09-149153
P407
P577
2009-12-21T00:00:00Z