Mitochondrial damage in the trabecular meshwork occurs only in primary open-angle glaucoma and in pseudoexfoliative glaucoma
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The Role of the Reactive Oxygen Species and Oxidative Stress in the Pathomechanism of the Age-Related Ocular Diseases and Other Pathologies of the Anterior and Posterior Eye Segments in AdultsThe Eye Drop Preservative Benzalkonium Chloride Potently Induces Mitochondrial Dysfunction and Preferentially Affects LHON Mutant CellsMitochondrial disorders and the eye.Association of a polymorphism in the BIRC6 gene with pseudoexfoliative glaucoma.Association of known common genetic variants with primary open angle, primary angle closure, and pseudoexfoliation glaucoma in Pakistani cohorts.sCD44 internalization in human trabecular meshwork cells.Focus on molecular events in the anterior chamber leading to glaucoma.Mitochondrial DNA lineages of African origin confer susceptibility to primary open-angle glaucoma in Saudi patientsIncreased mitochondrial fission and volume density by blocking glutamate excitotoxicity protect glaucomatous optic nerve head astrocytes.Quantitative proteomics: TGFβ₂ signaling in trabecular meshwork cells.Correlation between Systemic Oxidative Stress and Intraocular Pressure LevelCumulative mtDNA damage and mutations contribute to the progressive loss of RGCs in a rat model of glaucomaBuilding a glaucoma interaction network using a text mining approach.Association between systemic oxidative stress and visual field damage in open-angle glaucomaIncreased malondialdehyde concentration and reduced total antioxidant capacity in aqueous humor and blood samples from patients with glaucomaDelineation of a de novo 7q21.3q31.1 Deletion by CGH-SNP Arrays in a Girl with Multiple Congenital Anomalies Including Severe Glaucoma.Molecular diagnostics of ocular diseases: the application of antibody microarray.Bioactive lysophospholipids: role in regulation of aqueous humor outflow and intraocular pressure in the context of pathobiology and therapy of glaucoma.The dysfunction of the trabecular meshwork during glaucoma course.Ethyl pyruvate treatment mitigates oxidative stress damage in cultured trabecular meshwork cells.Rescue of cell death and inflammation of a mouse model of complex 1-mediated vision loss by repurposed drug molecules.Homocysteine induces oxidative stress to damage trabecular meshwork cells.The Eye, Oxidative Damage and Polyunsaturated Fatty Acids.
P2860
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P2860
Mitochondrial damage in the trabecular meshwork occurs only in primary open-angle glaucoma and in pseudoexfoliative glaucoma
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Mitochondrial damage in the tr ...... in pseudoexfoliative glaucoma
@ast
Mitochondrial damage in the tr ...... in pseudoexfoliative glaucoma
@en
type
label
Mitochondrial damage in the tr ...... in pseudoexfoliative glaucoma
@ast
Mitochondrial damage in the tr ...... in pseudoexfoliative glaucoma
@en
prefLabel
Mitochondrial damage in the tr ...... in pseudoexfoliative glaucoma
@ast
Mitochondrial damage in the tr ...... in pseudoexfoliative glaucoma
@en
P2093
P2860
P1433
P1476
Mitochondrial damage in the tr ...... in pseudoexfoliative glaucoma
@en
P2093
Cristina Cartiglia
Mariagrazia Longobardi
Sergio Claudio Saccà
P2860
P304
P356
10.1371/JOURNAL.PONE.0014567
P407
P577
2011-01-20T00:00:00Z