Activation of MyD88 signaling upon staphylococcal enterotoxin binding to MHC class II molecules
about
Staphylococcal superantigens in colonization and diseaseTherapeutic inhibition of pro-inflammatory signaling and toxicity to staphylococcal enterotoxin B by a synthetic dimeric BB-loop mimetic of MyD88Antigen-specific CD4(+) T cells regulate function of myeloid-derived suppressor cells in cancer via retrograde MHC class II signaling.Different transcriptional profiles of RAW264.7 infected with Mycobacterium tuberculosis H37Rv and BCG identified via deep sequencingA small molecule that mimics the BB-loop in the Toll interleukin-1 (IL-1) receptor domain of MyD88 attenuates staphylococcal enterotoxin B-induced pro-inflammatory cytokine production and toxicity in miceRole of miRNA in the regulation of inflammatory genes in staphylococcal enterotoxin B-induced acute inflammatory lung injury and mortalityΔ(9) Tetrahydrocannabinol attenuates Staphylococcal enterotoxin B-induced inflammatory lung injury and prevents mortality in mice by modulation of miR-17-92 cluster and induction of T-regulatory cells.Discovery of small molecule inhibitors of MyD88-dependent signaling pathways using a computational screenHLA-DRB1*11 and variants of the MHC class II locus are strong risk factors for systemic juvenile idiopathic arthritis.Positive Feedback Cycle of TNFα Promotes Staphylococcal Enterotoxin B-Induced THP-1 Cell ApoptosisInterleukin-10 is produced by a specific subset of taste receptor cells and critical for maintaining structural integrity of mouse taste buds.Regulation of suppressive function of myeloid-derived suppressor cells by CD4+ T cellsTec family kinases in inflammation and disease.TLR-induced activation of Btk -- role for endosomal MHC class II molecules revealedPro-IL-16 is associated with MHC class II-mediated negative regulation of mouse resting B cell activation through MAP kinases, NF-κB and Skp2-dependent p27kip regulation.Structure-Based Design and Synthesis of a Small Molecule that Exhibits Anti-inflammatory Activity by Inhibition of MyD88-mediated Signaling to Bacterial Toxin Exposure.Chronic exposure to staphylococcal superantigen elicits a systemic inflammatory disease mimicking lupus.Comparison of the innate immune responses of porcine monocyte-derived dendritic cells and splenic dendritic cells stimulated with LPS.Small Molecule Analogues of the parasitic worm product ES-62 interact with the TIR domain of MyD88 to inhibit pro-inflammatory signalling.Superantigen influence in conjunction with cytokine polymorphism potentiates autoimmunity in systemic lupus erythematosus patients.An unexpected role for MHC class II.Manipulation of Innate and Adaptive Immunity by Staphylococcal Superantigens.
P2860
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P2860
Activation of MyD88 signaling upon staphylococcal enterotoxin binding to MHC class II molecules
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Activation of MyD88 signaling ...... ding to MHC class II molecules
@ast
Activation of MyD88 signaling ...... ding to MHC class II molecules
@en
type
label
Activation of MyD88 signaling ...... ding to MHC class II molecules
@ast
Activation of MyD88 signaling ...... ding to MHC class II molecules
@en
prefLabel
Activation of MyD88 signaling ...... ding to MHC class II molecules
@ast
Activation of MyD88 signaling ...... ding to MHC class II molecules
@en
P2093
P2860
P1433
P1476
Activation of MyD88 signaling ...... ding to MHC class II molecules
@en
P2093
Gordon Ruthel
Kamal U Saikh
Robert G Ulrich
Shahabuddin Alam
Stefan Fernandez
Teri L Kissner
P2860
P304
P356
10.1371/JOURNAL.PONE.0015985
P407
P577
2011-01-20T00:00:00Z