Semaphorin 3A signaling through neuropilin-1 is an early trigger for distal axonopathy in the SOD1G93A mouse model of amyotrophic lateral sclerosis
about
ALS as a distal axonopathy: molecular mechanisms affecting neuromuscular junction stability in the presymptomatic stages of the diseaseGene expression profiling for human iPS-derived motor neurons from sporadic ALS patients reveals a strong association between mitochondrial functions and neurodegeneration.Dysregulated expression of death, stress and mitochondrion related genes in the sciatic nerve of presymptomatic SOD1(G93A) mouse model of Amyotrophic Lateral Sclerosis.Expression of a Mutant SEMA3A Protein with Diminished Signalling Capacity Does Not Alter ALS-Related Motor Decline, or Confer Changes in NMJ Plasticity after BotoxA-Induced Paralysis of Male Gastrocnemic MuscleCollapsin Response Mediator Protein-2 (CRMP2) is a Plausible Etiological Factor and Potential Therapeutic Target in Alzheimer's Disease: Comparison and Contrast with Microtubule-Associated Protein TauGenome-wide analysis reveals NRP1 as a direct HIF1α-E2F7 target in the regulation of motorneuron guidance in vivo.The Axon Guidance Protein Semaphorin 3A Is Increased in the Motor Cortex of Patients With Amyotrophic Lateral Sclerosis.Adaptive testing for multiple traits in a proportional odds model with applications to detect SNP-brain network associations.Label-Free LC-MS/MS Proteomic Analysis of Cerebrospinal Fluid Identifies Protein/Pathway Alterations and Candidate Biomarkers for Amyotrophic Lateral SclerosisNeuropilin 1 sequestration by neuropathogenic mutant glycyl-tRNA synthetase is permissive to vascular homeostasisImplications of white matter damage in amyotrophic lateral sclerosis (Review).Degeneration of proprioceptive sensory nerve endings in mice harboring amyotrophic lateral sclerosis-causing mutations.Slow-Myofiber Commitment by Semaphorin 3A Secreted from Myogenic Stem Cells.Multiple-step, one-pot synthesis of 2-substituted-3-phosphono-1-thia-4-aza-2-cyclohexene-5-carboxylates and their corresponding ethyl esters.ALS-related human cortical and motor neurons survival is differentially affected by Sema3A.Semaphorin3A Signaling Is Dispensable for Motor Axon Reinnervation of the Adult Neuromuscular Junction.Repulsive Environment Attenuation during Adult Mouse Optic Nerve RegenerationBMP- and neuropilin 1-mediated motor axon navigation relies on spastin alternative translation
P2860
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P2860
Semaphorin 3A signaling through neuropilin-1 is an early trigger for distal axonopathy in the SOD1G93A mouse model of amyotrophic lateral sclerosis
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Semaphorin 3A signaling throug ...... amyotrophic lateral sclerosis
@ast
Semaphorin 3A signaling throug ...... amyotrophic lateral sclerosis
@en
type
label
Semaphorin 3A signaling throug ...... amyotrophic lateral sclerosis
@ast
Semaphorin 3A signaling throug ...... amyotrophic lateral sclerosis
@en
prefLabel
Semaphorin 3A signaling throug ...... amyotrophic lateral sclerosis
@ast
Semaphorin 3A signaling throug ...... amyotrophic lateral sclerosis
@en
P2093
P2860
P1476
Semaphorin 3A signaling throug ...... amyotrophic lateral sclerosis
@en
P2093
Alexander Christov
Kenneth Hensley
Peter Kobalka
Saaid Siddiqui
Zarine Kamaluddin
P2860
P304
P356
10.1097/NEN.0000000000000086
P577
2014-07-01T00:00:00Z