Interferon gamma-dependent intestinal pathology contributes to the lethality in bacterial superantigen-induced toxic shock syndrome
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The staphylococcal enterotoxin (SE) family: SEB and siblingsStaphylococcal superantigens in colonization and diseaseThe impact of Staphylococcus aureus-associated molecular patterns on staphylococcal superantigen-induced toxic shock syndrome and pneumonia.Staphylococcal enterotoxin B-induced microRNA-155 targets SOCS1 to promote acute inflammatory lung injury.Chimeric anti-staphylococcal enterotoxin B antibodies and lovastatin act synergistically to provide in vivo protection against lethal doses of SEB.Late multiple organ surge in interferon-regulated target genes characterizes staphylococcal enterotoxin B lethality.3,39-Diindolylmethane Ameliorates Staphylococcal Enterotoxin B–Induced Acute Lung Injury through Alterations in the Expression of MicroRNA that Target Apoptosis and Cell-Cycle Arrest in Activated T Cells.Exacerbated type II interferon response drives hypervirulence and toxic shock by an emergent epidemic strain of Streptococcus suisMesenchymal stromal (stem) cells suppress pro-inflammatory cytokine production but fail to improve survival in experimental staphylococcal toxic shock syndrome.Soluble T cell receptor Vβ domains engineered for high-affinity binding to staphylococcal or streptococcal superantigensUnexpected heterogeneity of multifunctional T cells in response to superantigen stimulation in humans.Rapid and Rigorous IL-17A Production by a Distinct Subpopulation of Effector Memory T Lymphocytes Constitutes a Novel Mechanism of Toxic Shock Syndrome Immunopathology.Passive therapy with humanized anti-staphylococcal enterotoxin B antibodies attenuates systemic inflammatory response and protects from lethal pneumonia caused by staphylococcal enterotoxin B-producing Staphylococcus aureus.Superantigen-Producing Staphylococcus aureus Elicits Systemic Immune Activation in a Murine Wound Colonization Model.Induction of intestinal pro-inflammatory immune responses by lipoteichoic acid.Systemic inflammatory response elicited by superantigen destabilizes T regulatory cells, rendering them ineffective during toxic shock syndromeChronic exposure to staphylococcal superantigen elicits a systemic inflammatory disease mimicking lupus.Cytokine profile of the blood in mice with normal and abnormal heart rhythm.Concomitant Disruption of CD4 and CD8 Genes Facilitates the Development of Double Negative αβ TCR+ Peripheral T Cells That Respond Robustly to Staphylococcal Superantigen.
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P2860
Interferon gamma-dependent intestinal pathology contributes to the lethality in bacterial superantigen-induced toxic shock syndrome
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Interferon gamma-dependent int ...... n-induced toxic shock syndrome
@ast
Interferon gamma-dependent int ...... n-induced toxic shock syndrome
@en
type
label
Interferon gamma-dependent int ...... n-induced toxic shock syndrome
@ast
Interferon gamma-dependent int ...... n-induced toxic shock syndrome
@en
prefLabel
Interferon gamma-dependent int ...... n-induced toxic shock syndrome
@ast
Interferon gamma-dependent int ...... n-induced toxic shock syndrome
@en
P2093
P2860
P1433
P1476
Interferon gamma-dependent int ...... n-induced toxic shock syndrome
@en
P2093
Ashenafi Y Tilahun
Chella S David
Govindarajan Rajagopalan
Marah Holz
Tsung-Teh Wu
P2860
P304
P356
10.1371/JOURNAL.PONE.0016764
P407
P577
2011-02-03T00:00:00Z