Rapid onset of cardiomyopathy in STZ-induced female diabetic mice involves the downregulation of pro-survival Pim-1 - Wikidata - wikimedia - TriplyDB

Rapid onset of cardiomyopathy in STZ-induced female diabetic mice involves the downregulation of pro-survival Pim-1

Rapid onset of cardiomyopathy in STZ-induced female diabetic mice involves the downregulation of pro-survival Pim-1

http://www.wikidata.org/entity/Q33816171

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Targeting the Pim kinases in multiple myeloma The Role of MicroRNAs in Cardiac Stem Cells Data supporting the activation of autophagy genes in the diabetic heart Metabolic Remodeling in Diabetic Cardiomyopathy.MicroRNA-30d regulates cardiomyocyte pyroptosis by directly targeting foxo3a in diabetic cardiomyopathy Targeting metabolic disturbance in the diabetic heart.Vascular endothelial microparticles-incorporated microRNAs are altered in patients with diabetes mellitus.Pim1 kinase promotes angiogenesis through phosphorylation of endothelial nitric oxide synthase at Ser-633.Enhanced A2A adenosine receptor-mediated increase in coronary flow in type I diabetic mice.Role of microRNA in diabetic cardiomyopathy: From mechanism to intervention.Concise Review: Challenges in Regenerating the Diabetic Heart: A Comprehensive Review.High glucose promotes vascular smooth muscle cell proliferation by upregulating proto-oncogene serine/threonine-protein kinase Pim-1 expression.Diabetes induces the activation of pro-ageing miR-34a in the heart, but has differential effects on cardiomyocytes and cardiac progenitor cells.Diastolic dysfunction is more apparent in STZ-induced diabetic female mice, despite less pronounced hyperglycemia.Down-regulation of proangiogenic microRNA-126 and microRNA-132 are early modulators of diabetic cardiac microangiopathy.Molecular mechanism of diabetic cardiomyopathy and modulation of microRNA function by synthetic oligonucleotides.

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Rapid onset of cardiomyopathy in STZ-induced female diabetic mice involves the downregulation of pro-survival Pim-1

http://www.wikidata.org/entity/Q33816171

description

2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2014 թվականի ապրիլին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Rapid onset of cardiomyopathy ...... gulation of pro-survival Pim-1

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Rapid onset of cardiomyopathy ...... gulation of pro-survival Pim-1

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Rapid onset of cardiomyopathy ...... gulation of pro-survival Pim-1

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Rapid onset of cardiomyopathy ...... gulation of pro-survival Pim-1

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Rapid onset of cardiomyopathy ...... gulation of pro-survival Pim-1

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Cardiovascular Diabetology

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Rapid onset of cardiomyopathy ...... gulation of pro-survival Pim-1

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Amol Shindikar

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Andrew Moore

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Costanza Emanueli

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Federica Riu

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Ingrid Fomison-Nurse

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Ivor F Galvin

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Pankaj Saxena

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Paolo Madeddu

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Pujika E Munasinghe

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Richard W Bunton

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P2860

Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

Down-regulation of micro-RNA-1 (miR-1) in lung cancer. Suppression of tumorigenic property of lung cancer cells and their sensitization to doxorubicin-induced apoptosis by miR-1

Rat's age versus human's age: what is the relationship?

Intravenous gene therapy with PIM-1 via a cardiotropic viral vector halts the progression of diabetic cardiomyopathy through promotion of prosurvival signaling

Pim-1 kinase protects mitochondrial integrity in cardiomyocytes.

Vitamin B1 analog benfotiamine prevents diabetes-induced diastolic dysfunction and heart failure through Akt/Pim-1-mediated survival pathway

Comparative analysis of human and mouse expression data illuminates tissue-specific evolutionary patterns of miRNAs

Diabetic cardiomyopathy: from the pathophysiology of the cardiac myocytes to current diagnosis and management strategies

Protein kinase RNA-like endoplasmic reticulum kinase (PERK) signaling pathway plays a major role in reactive oxygen species (ROS)-mediated endoplasmic reticulum stress-induced apoptosis in diabetic cardiomyopathy.

MicroRNA therapeutics for cardiovascular disease: opportunities and obstacles.

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Michael Williams

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