Glomerular heparan sulfate alterations: mechanisms and relevance for proteinuria.
about
Histological changes of kidney in diabetic nephropathyA model for lupus brain diseaseDiabetic nephropathy and extracellular matrixAgrin isoforms with distinct amino termini: differential expression, localization, and functionLack of collagen XVIII/endostatin exacerbates immune-mediated glomerulonephritis.Elevated urine heparanase levels are associated with proteinuria and decreased renal allograft functionGenetic kidney diseases disclose the pathogenesis of proteinuria.Heparan sulfate proteoglycans.Effects of restoring normoglycemia in type 1 diabetes on inflammatory profile and renal extracellular matrix structure after simultaneous pancreas and kidney transplantationMicrovascular basement membranes in diabetes mellitus.Antibody response against perlecan and collagen types IV and VI in chronic renal allograft rejection in the rat.Disruption of glomerular basement membrane charge through podocyte-specific mutation of agrin does not alter glomerular permselectivityContribution of proteoglycans towards the integrated functions of renal glomerular capillaries: a historical perspective.The glomerular basement membrane as a model system to study the bioactivity of heparan sulfate glycosaminoglycans.Extracellular superoxide dismutase protects against matrix degradation of heparan sulfate in the lungExtracellular superoxide dismutase in pulmonary fibrosisUnchanged distribution density of anionic sites on the glomerular wall in rats with active heymann nephritis.Xylosyltransferase II is a significant contributor of circulating xylosyltransferase levels and platelets constitute an important source of xylosyltransferase in serum.Association of serum nitrate and nitrite levels with longitudinal assessments of disease activity and damage in systemic lupus erythematosus and lupus nephritis.Effect of castration on renal glycosaminoglycans and their urinary excretion in male and female rats with chronic renal failure.Blood vessels pattern heparan sulfate gradients between their apical and basolateral aspects.Losartan chemistry and its effects via AT1 mechanisms in the kidneyDiabetes-Induced Reactive Oxygen Species: Mechanism of Their Generation and Role in Renal InjuryPathophysiology of the diabetic kidney.The endothelium in diabetic nephropathy.Nephritogenic lupus antibodies recognize glomerular basement membrane-associated chromatin fragments released from apoptotic intraglomerular cells.Hyperoside pre-treatment prevents glomerular basement membrane damage in diabetic nephropathy by inhibiting podocyte heparanase expression.Glomerular basement membrane heparan sulfate in health and disease: A regulator of local complement activation.Reduced sulfation of chondroitin sulfate but not heparan sulfate in kidneys of diabetic db/db mice.Reactive oxygen species mediate high glucose-induced heparanase-1 production and heparan sulphate proteoglycan degradation in human and rat endothelial cells: a potential role in the pathogenesis of atherosclerosis.Heparanase is essential for the development of diabetic nephropathy in mice.Tubulointerstitial heparan sulfate proteoglycan changes in human renal diseases correlate with leukocyte influx and proteinuria.Heparan sulfate depletion amplifies TNF-alpha-induced protein leakage in an in vitro model of protein-losing enteropathy.Proteases in Plasma and Kidney of db/db Mice as Markers of Diabetes-Induced Nephropathy.Nucleosomes possess a high affinity for glomerular laminin and collagen IV and bind nephritogenic antibodies in murine lupus-like nephritis.Age-dependent alteration of intraocular soluble heparan sulfate levels and its implications for proliferative diabetic retinopathyOxidation of heparan sulphate by hypochlorite: role of N-chloro derivatives and dichloramine-dependent fragmentation.Human immunodeficiency virus-1 tat induces hyperproliferation and dysregulation of renal glomerular epithelial cells.Hypochlorite and superoxide radicals can act synergistically to induce fragmentation of hyaluronan and chondroitin sulphates.Role of reactive oxygen species in pathogenesis of nephrotic syndrome.
P2860
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P2860
Glomerular heparan sulfate alterations: mechanisms and relevance for proteinuria.
description
2000 nî lūn-bûn
@nan
2000 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Glomerular heparan sulfate alterations: mechanisms and relevance for proteinuria.
@ast
Glomerular heparan sulfate alterations: mechanisms and relevance for proteinuria.
@en
type
label
Glomerular heparan sulfate alterations: mechanisms and relevance for proteinuria.
@ast
Glomerular heparan sulfate alterations: mechanisms and relevance for proteinuria.
@en
prefLabel
Glomerular heparan sulfate alterations: mechanisms and relevance for proteinuria.
@ast
Glomerular heparan sulfate alterations: mechanisms and relevance for proteinuria.
@en
P2093
P1433
P1476
Glomerular heparan sulfate alterations: mechanisms and relevance for proteinuria.
@en
P2093
Van Den Born J
P304
P356
10.1046/J.1523-1755.2000.00858.X
P407
P577
2000-02-01T00:00:00Z
P5875
P6179
1035638795