The length of and nonhydrophobic residues in the transmembrane domain of dengue virus envelope protein are critical for its retention and assembly in the endoplasmic reticulum
about
Human antibody responses after dengue virus infection are highly cross-reactive to Zika virusGetting membrane proteins on and off the shuttle bus between the endoplasmic reticulum and the Golgi complexSulfated polysaccharide, curdlan sulfate, efficiently prevents entry/fusion and restricts antibody-dependent enhancement of dengue virus infection in vitro: a possible candidate for clinical applicationInduction of endoplasmic reticulum-derived replication-competent membrane structures by West Nile virus non-structural protein 4BInduction of a peptide with activity against a broad spectrum of pathogens in the Aedes aegypti salivary gland, following Infection with Dengue Virus.C-terminal helical domains of dengue virus type 4 E protein affect the expression/stability of prM protein and conformation of prM and E proteins.The transmembrane domain of the molecular chaperone Cosmc directs its localization to the endoplasmic reticulum.Bioluminescence assay for detecting cell surface membrane protein expression.Vaccination with dengue virus-like particles induces humoral and cellular immune responses in mice.Static retention of the lumenal monotopic membrane protein torsinA in the endoplasmic reticulum.Anchors aweigh: protein localization and transport mediated by transmembrane domainsImproved genetic stability of recombinant yellow fever 17D virus expressing a lentiviral Gag gene fragment.The major determinant of attenuation in mice of the Candid1 vaccine for Argentine hemorrhagic fever is located in the G2 glycoprotein transmembrane domain.Evaluation of chimeric DNA vaccines consisting of premembrane and envelope genes of Japanese encephalitis and dengue viruses as a strategy for reducing induction of dengue virus infection-enhancing antibody response.Phenotypic and genotypic characterization of dengue virus isolates differentiates dengue fever and dengue hemorrhagic fever from dengue shock syndrome.Retention of a recombinant GFP protein expressed by the yellow fever 17D virus in the E/NS1 intergenic region in the endoplasmic reticulum.The unique transmembrane hairpin of flavivirus fusion protein E is essential for membrane fusion.Mosquito-borne flaviviruses: overview of viral life-cycle and host–virus interactions
P2860
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P2860
The length of and nonhydrophobic residues in the transmembrane domain of dengue virus envelope protein are critical for its retention and assembly in the endoplasmic reticulum
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The length of and nonhydrophob ...... y in the endoplasmic reticulum
@ast
The length of and nonhydrophob ...... y in the endoplasmic reticulum
@en
type
label
The length of and nonhydrophob ...... y in the endoplasmic reticulum
@ast
The length of and nonhydrophob ...... y in the endoplasmic reticulum
@en
prefLabel
The length of and nonhydrophob ...... y in the endoplasmic reticulum
@ast
The length of and nonhydrophob ...... y in the endoplasmic reticulum
@en
P2860
P50
P921
P356
P1433
P1476
The length of and nonhydrophob ...... y in the endoplasmic reticulum
@en
P2860
P304
P356
10.1128/JVI.01963-09
P577
2010-02-24T00:00:00Z