The varicella-zoster virus ORF47 kinase interferes with host innate immune response by inhibiting the activation of IRF3 - Wikidata - wikimedia - TriplyDB

The varicella-zoster virus ORF47 kinase interferes with host innate immune response by inhibiting the activation of IRF3

The varicella-zoster virus ORF47 kinase interferes with host innate immune response by inhibiting the activation of IRF3

http://www.wikidata.org/entity/Q33828713

about

Varicella-zoster virus immediate-early protein ORF61 abrogates the IRF3-mediated innate immune response through degradation of activated IRF3 Molecular mechanisms of varicella zoster virus pathogenesis.Pathogenesis of varicelloviruses in primates Microbial strategies for antagonizing Toll-like-receptor signal transduction.Varicella Viruses Inhibit Interferon-Stimulated JAK-STAT Signaling through Multiple Mechanisms The ORF61 Protein Encoded by Simian Varicella Virus and Varicella-Zoster Virus Inhibits NF-κB Signaling by Interfering with IκBα Degradation.Insights into the role of immunosenescence during varicella zoster virus infection (shingles) in the aging cell model ORF9p phosphorylation by ORF47p is crucial for the formation and egress of varicella-zoster virus viral particles.Molecular and genetic inflammation networks in major human diseases.Herpes simplex virus 1 serine/threonine kinase US3 hyperphosphorylates IRF3 and inhibits beta interferon production.Toll-like receptor sensing of human herpesvirus infection.Recent advances in understanding viral evasion of type I interferon.Potential of protein kinase inhibitors for treating herpesvirus-associated disease.Attack, parry and riposte: molecular fencing between the innate immune system and human herpesviruses.Insights into the function of tegument proteins from the varicella zoster virus.Suppressor of Cytokine Signaling 3 Expression Induced by Varicella-Zoster Virus Infection Results in the Modulation of Virus Replication.Involvement of herpes simplex virus type 1 UL13 protein kinase in induction of SOCS genes, the negative regulators of cytokine signaling.Dendritic cells as Achilles' heel and Trojan horse during varicella zoster virus infection.Innate and adaptive immunity gene expression of human keratinocytes cultured of severe burn injury.Cellular Stress Response to Varicella-Zoster Virus Infection of Human Skin Includes Highly Elevated Interleukin-6 Expression

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The varicella-zoster virus ORF47 kinase interferes with host innate immune response by inhibiting the activation of IRF3

http://www.wikidata.org/entity/Q33828713

description

2011 nî lūn-bûn

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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

The varicella-zoster virus ORF ...... ibiting the activation of IRF3

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The varicella-zoster virus ORF ...... ibiting the activation of IRF3

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The varicella-zoster virus ORF ...... ibiting the activation of IRF3

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The varicella-zoster virus ORF ...... ibiting the activation of IRF3

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The varicella-zoster virus ORF ...... ibiting the activation of IRF3

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The varicella-zoster virus ORF ...... ibiting the activation of IRF3

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The varicella-zoster virus ORF ...... ibiting the activation of IRF3

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Catherine Sadzot-Delvaux

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Emmanuel Di Valentin

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Estelle Dortu

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Isabelle Ote

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Jacques Piette

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Marielle Lebrun

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Nadia El Mjiyad

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Patricia Vandevenne

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Yvette Habraken

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P2860

Positive and negative regulation of the innate antiviral response and beta interferon gene expression by deacetylation.

DAI (DLM-1/ZBP1) is a cytosolic DNA sensor and an activator of innate immune response

AIM2 recognizes cytosolic dsDNA and forms a caspase-1-activating inflammasome with ASC

An orthogonal proteomic-genomic screen identifies AIM2 as a cytoplasmic DNA sensor for the inflammasome

Virus-dependent phosphorylation of the IRF-3 transcription factor regulates nuclear translocation, transactivation potential, and proteasome-mediated degradation

Direct triggering of the type I interferon system by virus infection: activation of a transcription factor complex containing IRF-3 and CBP/p300.

Innate cellular response to virus particle entry requires IRF3 but not virus replication

The herpes simplex virus ICP0 RING finger domain inhibits IRF3- and IRF7-mediated activation of interferon-stimulated genes

Varicella-zoster virus

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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e16870

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scholarly article

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10.1371/JOURNAL.PONE.0016870

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