Protease-activated receptor-2 regulates the innate immune response to viral infection in a coxsackievirus B3-induced myocarditis - Wikidata - wikimedia - TriplyDB

Protease-activated receptor-2 regulates the innate immune response to viral infection in a coxsackievirus B3-induced myocarditis

Protease-activated receptor-2 regulates the innate immune response to viral infection in a coxsackievirus B3-induced myocarditis

http://www.wikidata.org/entity/Q33830395

about

Tissue factor, protease activated receptors and pathologic heart remodelling Platelets and coagulation in infection Roles of PAR1 and PAR2 in viral myocarditis.Protease activated receptor-2 contributes to heart failure.Coagulation, protease-activated receptors, and viral myocarditis.Cardiac Function Remains Impaired Despite Reversible Cardiac Remodeling after Acute Experimental Viral Myocarditis Multiple roles of the coagulation protease cascade during virus infection.Protease-Activated Receptor 1 Enhances Poly I:C Induction of the Antiviral Response in Macrophages and Mice.MicroRNA-20b suppresses the expression of ZFP-148 in viral myocarditis.Cardiac fibroblasts aggravate viral myocarditis: cell specific coxsackievirus B3 replication.Ectopic trypsin in the myocardium promotes dilated cardiomyopathy after influenza A virus infection.Inhibition of coagulation factor Xa improves myocardial function during CVB3-induced myocarditis.Infectious myocarditis: the role of the cardiac vasculature.Proteinase-activated receptor 2 promotes tumor cell proliferation and metastasis by inducing epithelial-mesenchymal transition and predicts poor prognosis in hepatocellular carcinoma.Advances in Clinical and Basic Science of Coagulation: Illustrated abstracts of the 9th Chapel Hill Symposium on Hemostasis

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P2860

Protease-activated receptor-2 regulates the innate immune response to viral infection in a coxsackievirus B3-induced myocarditis

http://www.wikidata.org/entity/Q33830395

description

2013 nî lūn-bûn

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2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2013 թվականի հուլիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Protease-activated receptor-2 ...... ievirus B3-induced myocarditis

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Protease-activated receptor-2 ...... ievirus B3-induced myocarditis

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Protease-activated receptor-2 ...... ievirus B3-induced myocarditis

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Protease-activated receptor-2 ...... ievirus B3-induced myocarditis

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Protease-activated receptor-2 ...... ievirus B3-induced myocarditis

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Protease-activated receptor-2 ...... ievirus B3-induced myocarditis

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J.JACC.2013.05.076

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Journal of the American College of Cardiology

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Protease-activated receptor-2 ...... ievirus B3-induced myocarditis

@en

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Alice Weithauser

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Andrea Stroux

http://www.w3.org/2001/XMLSchema#string

Andreas Böhm

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Bernhard H Rauch

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Carsten Tschope

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Heinz Zeichhardt

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Heinz-Peter Schultheiss

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Heyo K Kroemer

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Konstantinos Savvatis

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Nigel Mackman

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P2860

Novel signaling interactions between proteinase-activated receptor 2 and Toll-like receptors in vitro and in vivo

Analysis of proteinase-activated receptor 2 and TLR4 signal transduction: a novel paradigm for receptor cooperativity

Protective role for protease-activated receptor-2 against influenza virus pathogenesis via an IFN-gamma-dependent pathway

Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

Cardiovascular responses mediated by protease-activated receptor-2 (PAR-2) and thrombin receptor (PAR-1) are distinguished in mice deficient in PAR-2 or PAR-1

Thrombin signalling and protease-activated receptors

Protease-activated receptor 2 deficiency reduces cardiac ischemia/reperfusion injury.

Ongoing enterovirus-induced myocarditis is associated with persistent heart muscle infection: quantitative analysis of virus replication, tissue damage, and inflammation

Molecular biology and pathogenesis of viral myocarditis.

A critical link between Toll-like receptor 3 and type II interferon signaling pathways in antiviral innate immunity.

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1737-1745

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scholarly article

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10.1016/J.JACC.2013.05.076

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