Mitochondrial damage in muscle occurs after marked depletion of glutathione and is prevented by giving glutathione monoester
about
Primary role of functional ischemia, quantitative evidence for the two-hit mechanism, and phosphodiesterase-5 inhibitor therapy in mouse muscular dystrophyGlutathione deficiency leads to mitochondrial damage in brainIn situ detection and measurement of intracellular reactive oxygen species in single isolated mature skeletal muscle fibers by real time fluorescence microscopy.Cystathionine gamma-Lyase-deficient mice require dietary cysteine to protect against acute lethal myopathy and oxidative injury.Alterations in the glutathione metabolism could be implicated in the ischemia-induced small intestinal cell damage in horsesHigh-affinity transport of glutathione is part of a multicomponent system essential for mitochondrial functionOxidative stress accumulates in adipose tissue during aging and inhibits adipogenesis.Hydrogen peroxide efflux from muscle mitochondria underestimates matrix superoxide production--a correction using glutathione depletion.Myopathy in vitamin E deficient rats: muscle fibre necrosis associated with disturbances of mitochondrial function.gamma-Glutamyl transpeptidase overexpression increases metastatic growth of B16 melanoma cells in the mouse liver.Impaired uptake of glutathione by hepatic mitochondria from chronic ethanol-fed rats. Tracer kinetic studies in vitro and in vivo and susceptibility to oxidant stressEffect of chronic ethanol feeding on glutathione and functional integrity of mitochondria in periportal and perivenous rat hepatocytes.Oral N-acetylcysteine rescues lethality of hepatocyte-specific Gclc-knockout mice, providing a model for hepatic cirrhosis.Glutathione metabolism in the lung: inhibition of its synthesis leads to lamellar body and mitochondrial defectsGlutathione is required for intestinal function.Glutathione ester prevents buthionine sulfoximine-induced cataracts and lens epithelial cell damage.Mitochondrial peroxiredoxins are critical for the maintenance of redox state and the survival of adult DrosophilaMuscle choline kinase beta defect causes mitochondrial dysfunction and increased mitophagy.Dissecting the role of glutathione biosynthesis in Plasmodium falciparum.The mechanism of cardioprotection by S-nitrosoglutathione monoethyl ester in rat isolated heart during cardioplegic ischaemic arrestGlutathione ester delays the onset of scurvy in ascorbate-deficient guinea pigs.Time line of redox events in aging postmitotic cells.Effector T Cells Abrogate Stroma-Mediated Chemoresistance in Ovarian CancerAscorbic acid prevents oxidative stress in glutathione-deficient mice: effects on lung type 2 cell lamellar bodies, lung surfactant, and skeletal muscle.Defective homocysteine metabolism: potential implications for skeletal muscle malfunctionButhionine sulfoximine sensitizes antihormone-resistant human breast cancer cells to estrogen-induced apoptosis.CFTR activity and mitochondrial function.Glutathione deficiency increases hepatic ascorbic acid synthesis in adult mice.Glutathione deficiency decreases tissue ascorbate levels in newborn rats: ascorbate spares glutathione and protects.Inhibition of glutathione synthesis in the newborn rat: a model for endogenously produced oxidative stress.Disruption of GRM1-mediated signalling using riluzole results in DNA damage in melanoma cells.Effects of N-acetylcysteine and glutathione ethyl ester drops on streptozotocin-induced diabetic cataract in rats.Characterization of mitochondrial function in cells with impaired cystic fibrosis transmembrane conductance regulator (CFTR) function.Redox regulation of homocysteine-dependent glutathione synthesis.Glutathione in health and disease: pharmacotherapeutic issues.Nitric oxide-generating system as an autocrine mechanism in human polymorphonuclear leucocytes.Elevated skeletal muscle apoptotic signaling following glutathione depletion.Depletion of tumour glutathione in vivo by buthionine sulphoximine: modulation by the rate of cellular proliferation and inhibition of cancer growth.Inhibition of cancer growth and selective glutathione depletion in Ehrlich tumour cells in vivo by extracellular ATP.Glutathione and the rate of cellular proliferation determine tumour cell sensitivity to tumour necrosis factor in vivo.
P2860
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P2860
Mitochondrial damage in muscle occurs after marked depletion of glutathione and is prevented by giving glutathione monoester
description
1989 nî lūn-bûn
@nan
1989 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Mitochondrial damage in muscle ...... y giving glutathione monoester
@ast
Mitochondrial damage in muscle ...... y giving glutathione monoester
@en
type
label
Mitochondrial damage in muscle ...... y giving glutathione monoester
@ast
Mitochondrial damage in muscle ...... y giving glutathione monoester
@en
prefLabel
Mitochondrial damage in muscle ...... y giving glutathione monoester
@ast
Mitochondrial damage in muscle ...... y giving glutathione monoester
@en
P2860
P356
P1476
Mitochondrial damage in muscle ...... y giving glutathione monoester
@en
P2093
J Mårtensson
P2860
P304
P356
10.1073/PNAS.86.2.471
P407
P577
1989-01-01T00:00:00Z