Hormonal modulation of endothelial NO production. - Wikidata - wikimedia - TriplyDB

Hormonal modulation of endothelial NO production.

Hormonal modulation of endothelial NO production.

http://www.wikidata.org/entity/Q33833608

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Constrictor prostanoids and uridine adenosine tetraphosphate: vascular mediators and therapeutic targets in hypertension and diabetes Mechanistic Pathways of Sex Differences in Cardiovascular Disease.Assessment of endothelial dysfunction in childhood obesity and clinical use.Should there be sex-specific criteria for the diagnosis and treatment of heart failure?Stress-related programming of autonomic imbalance: role in allergy and asthma.Endothelial Dysfunction Markers in Low Cardiovascular Risk Individuals: Comparison of Males and Females.Progesterone supplementation attenuates hypertension and the autoantibody to the angiotensin II type I receptor in response to elevated interleukin-6 during pregnancy.Female resistance to pneumonia identifies lung macrophage nitric oxide synthase-3 as a therapeutic target R- and S-equol have equivalent cytoprotective effects in Friedreich's ataxia.Sex, hormones and neuroeffector mechanisms.Mechanisms of cerebrovascular protection: oestrogen, inflammation and mitochondria Congress on women's health Trudy Bush lecture 2014: new insights into sex Hormones and Cardiovascular disease Potential role of intermedin/adrenomedullin 2 in early embryonic development in rats Implications for reproductive medicine: Sex differences in cardiovascular disease Women-specific factors to consider in risk, diagnosis and treatment of cardiovascular disease.The prolactin family hormones regulate vascular tone through NO and prostacyclin production in isolated rat aortic rings Strategies and methods to study sex differences in cardiovascular structure and function: a guide for basic scientists.Plasma gelsolin improves lung host defense against pneumonia by enhancing macrophage NOS3 function.Therapeutic Efficacy of an ω-3-Fatty Acid-Containing 17-β Estradiol Nano-Delivery System against Experimental Atherosclerosis.Local versus systemic influences on uterine vascular reactivity during pregnancy in the single-horn gravid rat.Dynamics of enhanced mitochondrial respiration in female compared with male rat cerebral arteries Association of insulin resistance with overactive bladder in female patients Sex-specific risk of cardiovascular disease and cognitive decline: pregnancy and menopause.Age and gender differences in the influences of eNOS T-786C polymorphism on arteriosclerotic parameters in general population in Japan.Prolactin protects retinal pigment epithelium by inhibiting sirtuin 2-dependent cell death Increased bladder wall thickness in diabetic and nondiabetic women with overactive bladder.Low-dose testosterone alleviates vascular damage caused by castration in male rats in puberty via modulation of the PI3K/AKT signaling pathway.Association of endothelial nitric oxide synthase gene polymorphisms with endometrial carcinoma: a preliminary study.Sex hormone therapy and progression of cardiovascular disease in menopausal women Genotype-related effect of crowding stress on blood pressure and vascular function in young female rats.Differential effects of oral and transdermal menopausal hormone therapy on prostacyclin and thromboxane in platelets.Hormones, heart disease, and health: individualized medicine versus throwing the baby out with the bathwater.How mental stress affects endothelial function.Estrogen and the cardiovascular system.Regulation of endothelial nitric oxide synthase and high-density lipoprotein quality by estradiol in cardiovascular pathology.A relative L-arginine deficiency contributes to endothelial dysfunction across the stages of the menopausal transition.Correlation between polymorphism of endothelial nitric oxide synthase and avascular necrosis of femoral head.Peroxynitrite decomposition with FeTMPyP improves plasma-induced vascular dysfunction and infarction during mild but not severe hyperglycemic stroke.Risk factors for cardiovascular disease in women with diabetes.Testosterone and β-oestradiol prevent inward remodelling of rat small mesenteric arteries: role of NO and transglutaminase.

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P2860

Hormonal modulation of endothelial NO production.

http://www.wikidata.org/entity/Q33833608

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Hormonal modulation of endothelial NO production.

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Hormonal modulation of endothelial NO production.

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Hormonal modulation of endothelial NO production.

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Hormonal modulation of endothelial NO production.

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Sue P Duckles

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Virginia M Miller

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P2860

Short-term administration of growth hormone (GH) lowers blood pressure by activating eNOS/nitric oxide (NO)-pathway in male hypophysectomized (Hx) rats.

Estrogen and progesterone receptors: from molecular structures to clinical targets

Testosterone increases thromboxane A2 receptor density and responsiveness in rat aortas and platelets

Insulin at physiological concentrations selectively activates insulin but not insulin-like growth factor I (IGF-I) or insulin/IGF-I hybrid receptors in endothelial cells.

Growth hormone exerts acute vascular effects independent of systemic or muscle insulin-like growth factor I

Abnormal vascular reactivity in growth hormone deficiency.

Modulation of antioxidant enzyme expression and function by estrogen.

Insulin signalling: metabolic pathways and mechanisms for specificity.

Dual effects of insulin-like growth factor-I on the constitutive and inducible nitric oxide (NO) synthase-dependent formation of NO in vascular cells.

Clinical review 110: Cardiovascular actions of estrogens in men.

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