Basal and reovirus-induced beta interferon (IFN-beta) and IFN-beta-stimulated gene expression are cell type specific in the cardiac protective response.
about
Epstein-Barr virus latent membrane protein 1 (LMP1) C-terminal-activating region 3 contributes to LMP1-mediated cellular migration via its interaction with Ubc9Early Production of Type I Interferon during West Nile Virus Infection: Role for Lymphoid Tissues in IRF3-Independent Interferon ProductionRoles of PAR1 and PAR2 in viral myocarditis.A single-amino-acid polymorphism in reovirus protein μ2 determines repression of interferon signaling and modulates myocarditisLMP1-Induced Sumoylation Influences the Maintenance of Epstein-Barr Virus Latency through KAP1Oligodendroglia are limited in type I interferon induction and responsiveness in vivoType I interferons produced by hematopoietic cells protect mice against lethal infection by mammalian reovirus.Basal expression levels of IFNAR and Jak-STAT components are determinants of cell-type-specific differences in cardiac antiviral responsesPAR-1 contributes to the innate immune response during viral infection.Cardiac cell-specific apoptotic and cytokine responses to reovirus infection: determinants of myocarditic phenotype.IFN-alpha expression and antiviral effects are subtype and cell type specific in the cardiac response to viral infectionAn ITAM in a nonenveloped virus regulates activation of NF-κB, induction of beta interferon, and viral spread.Interferon regulatory factor 7 functions as a novel negative regulator of pathological cardiac hypertrophyIn vivo ablation of type I interferon receptor from cardiomyocytes delays coxsackieviral clearance and accelerates myocardial disease.Multiple roles of the coagulation protease cascade during virus infection.Systems biology unravels interferon responses to respiratory virus infections.NF-κB activation is cell type-specific in the heart.Spontaneous activation of a MAVS-dependent antiviral signaling pathway determines high basal interferon-β expression in cardiac myocytes.Retinoic acid-inducible gene-I and interferon-beta promoter stimulator-1 augment proapoptotic responses following mammalian reovirus infection via interferon regulatory factor-3.Acquired resistance to reoviral oncolysis in Ras-transformed fibrosarcoma cells.A Cytoplasmic RNA Virus Alters the Function of the Cell Splicing Protein SRSF2.Genes Induced by Reovirus Infection Have a Distinct Modular Cis-Regulatory ArchitectureAntiviral effects of interferon-β are enhanced in the absence of the translational suppressor 4E-BP1 in myocarditis induced by Coxsackievirus B3.Reovirus mu2 protein inhibits interferon signaling through a novel mechanism involving nuclear accumulation of interferon regulatory factor 9.
P2860
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P2860
Basal and reovirus-induced beta interferon (IFN-beta) and IFN-beta-stimulated gene expression are cell type specific in the cardiac protective response.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մարտին հրատարակված գիտական հոդված
@hy
2005年の論文
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2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Basal and reovirus-induced bet ...... e cardiac protective response.
@ast
Basal and reovirus-induced bet ...... e cardiac protective response.
@en
type
label
Basal and reovirus-induced bet ...... e cardiac protective response.
@ast
Basal and reovirus-induced bet ...... e cardiac protective response.
@en
prefLabel
Basal and reovirus-induced bet ...... e cardiac protective response.
@ast
Basal and reovirus-induced bet ...... e cardiac protective response.
@en
P2093
P2860
P1433
P1476
Basal and reovirus-induced bet ...... e cardiac protective response.
@en
P2093
Barbara Sherry
Kathleen Smoak
Mary Ann Blum
Michael J Stewart
P2860
P304
P356
10.1128/JVI.79.5.2979-2987.2005
P577
2005-03-01T00:00:00Z