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Spreading depolarizations and late secondary insults after traumatic brain injury

Spreading depolarizations and late secondary insults after traumatic brain injury

http://www.wikidata.org/entity/Q33835140

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Real-time optical diagnosis of the rat brain exposed to a laser-induced shock wave: observation of spreading depolarization, vasoconstriction and hypoxemia-oligemia Spreading Depression, Spreading Depolarizations, and the Cerebral Vasculature Spreading depolarizations cycle around and enlarge focal ischaemic brain lesions Dynamic metabolic response to multiple spreading depolarizations in patients with acute brain injury: an online microdialysis study.Real-time monitoring of changes in brain extracellular sodium and potassium concentrations and intracranial pressure after selective vasopressin-1a receptor inhibition following focal traumatic brain injury in rats.Characterization of the relationship between intracranial pressure and electroencephalographic monitoring in burst-suppressed patients.Supply-demand mismatch transients in susceptible peri-infarct hot zones explain the origins of spreading injury depolarizations.Traumatic brain injury in adult rats causes progressive nigrostriatal dopaminergic cell loss and enhanced vulnerability to the pesticide paraquat.The utility of EEG, SSEP, and other neurophysiologic tools to guide neurocritical care.The new neurometabolic cascade of concussion.Correlates of spreading depolarization in human scalp electroencephalography Pannexin protein expression in the rat middle cerebral artery.Advancing age and ischemia elevate the electric threshold to elicit spreading depolarization in the cerebral cortex of young adult rats.Cortical excitation and inhibition following focal traumatic brain injury.Long-Term Consequences of Traumatic Brain Injury: Current Status of Potential Mechanisms of Injury and Neurological Outcomes Peri-infarct blood-brain barrier dysfunction facilitates induction of spreading depolarization associated with epileptiform discharges.Evolution of neuronal and astroglial disruption in the peri-contusional cortex of mice revealed by in vivo two-photon imaging.Electroencephalographic inverse localization of brain activity in acute traumatic brain injury as a guide to surgery, monitoring and treatment.Clinical relevance of cortical spreading depression in neurological disorders: migraine, malignant stroke, subarachnoid and intracranial hemorrhage, and traumatic brain injury.The changing landscape of voltage-gated calcium channels in neurovascular disorders and in neurodegenerative diseases Temporal fate specification and neural progenitor competence during development.Spreading depolarization in the ischemic brain: does aging have an impact?Electrophysiologic monitoring in acute brain injury.Age or ischemia uncouples the blood flow response, tissue acidosis, and direct current potential signature of spreading depolarization in the rat brain.Brain temperature but not core temperature increases during spreading depolarizations in patients with spontaneous intracerebral hemorrhage.Spreading Depression in Primary and Secondary Headache Disorders.Recording, analysis, and interpretation of spreading depolarizations in neurointensive care: Review and recommendations of the COSBID research group.Spreading Depolarizations: A Therapeutic Target Against Delayed Cerebral Ischemia After Subarachnoid Hemorrhage.Novel Treatments in Neuroprotection for Aneurysmal Subarachnoid Hemorrhage.Traumatic brain injury-induced autoregulatory dysfunction and spreading depression-related neurovascular uncoupling: Pathomechanisms, perspectives, and therapeutic implications.Cortical spreading depolarizations in the postresuscitation period in a cardiac arrest male rat model.Excitotoxicity and Metabolic Crisis Are Associated with Spreading Depolarizations in Severe Traumatic Brain Injury Patients.Enhanced neuronal excitability in adult rat brainstem causes widespread repetitive brainstem depolarizations with cardiovascular consequences Vascular, electrophysiological, and metabolic consequences of cortical spreading depression in a mouse model of simulated neurosurgical conditions Experimental and preliminary clinical evidence of an ischemic zone with prolonged negative DC shifts surrounded by a normally perfused tissue belt with persistent electrocorticographic depression.The continuum of spreading depolarizations in acute cortical lesion development: Examining Leão's legacy.Mild Blast Injury Produces Acute Changes in Basal Intracellular Calcium Levels and Activity Patterns in Mouse Hippocampal Neurons.Spreading depolarizations in patients with spontaneous intracerebral hemorrhage: Association with perihematomal edema progression.Mechanisms of Neuronal Silencing After Cortical Spreading Depression.Inverse neurovascular coupling to cortical spreading depolarizations in severe brain trauma.

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Spreading depolarizations and late secondary insults after traumatic brain injury

http://www.wikidata.org/entity/Q33835140

description

2009 nî lūn-bûn

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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Spreading depolarizations and late secondary insults after traumatic brain injury

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Spreading depolarizations and late secondary insults after traumatic brain injury

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Spreading depolarizations and late secondary insults after traumatic brain injury

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Spreading depolarizations and late secondary insults after traumatic brain injury

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Spreading depolarizations and late secondary insults after traumatic brain injury

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Spreading depolarizations and late secondary insults after traumatic brain injury

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Journal of Neurotrauma

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Spreading depolarizations and late secondary insults after traumatic brain injury

@en

P2093

Andrew Manning

http://www.w3.org/2001/XMLSchema#string

Anthony J Strong

http://www.w3.org/2001/XMLSchema#string

Co-Operative Study of Brain Injury Depolarizations

http://www.w3.org/2001/XMLSchema#string

Frank C Tortella

http://www.w3.org/2001/XMLSchema#string

M Ross Bullock

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Martin Fabricius

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Robin Bhatia

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P2860

Peri-infarct depolarizations reveal penumbra-like conditions in striatum.

Arterial hypotension triggers perifocal depolarizations and aggravates secondary damage in focal brain injury.

Peri-infarct depolarizations lead to loss of perfusion in ischaemic gyrencephalic cerebral cortex.

Persisting depletion of brain glucose following cortical spreading depression, despite apparent hyperaemia: evidence for risk of an adverse effect of Leão's spreading depression.

Jugular venous desaturation and outcome after head injury.

Cortical spreading depression activates and upregulates MMP-9.

Mechanisms of spreading depression and hypoxic spreading depression-like depolarization.

Prevention of secondary ischemic insults after severe head injury.

Increased incidence and impact of nonconvulsive and convulsive seizures after traumatic brain injury as detected by continuous electroencephalographic monitoring.

Continuous EEG monitoring in the intensive care unit: an overview.

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1857-1866

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scholarly article

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10.1089/NEU.2009.0961

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11

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26

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Jens Peter Dreier

Anna Teresa Mazzeo

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2009-11-01T00:00:00Z

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19508156

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traumatic brain injury

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2865988

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