Estradiol regulates susceptibility following primary exposure to genital herpes simplex virus type 2, while progesterone induces inflammation.
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Liposomal gD ectodomain (gD1-306) vaccine protects against HSV2 genital or rectal infection of female and male mice.Progesterone and 17beta-estradiol enhance regulatory responses to human papillomavirus type 16 virus-like particles in peripheral blood mononuclear cells from healthy womenHerpes simplex virus and the chemokines that mediate the inflammation.Protection against genital herpes infection in mice immunized under different hormonal conditions correlates with induction of vagina-associated lymphoid tissue.Sex influences immune responses to viruses, and efficacy of prophylaxis and treatments for viral diseasesNanoparticle-based drug delivery to the vagina: a review.The cotton rat provides a novel model to study genital herpes infection and to evaluate preventive strategies.Illumination of murine gammaherpesvirus-68 cycle reveals a sexual transmission route from females to males in laboratory mice.Association of increased prenatal estrogen with risk factors for schizophrenia.Accelerated immune senescence and reduced response to vaccination in ovariectomized female rhesus macaques.A novel pre-clinical murine model to study the life cycle and progression of cervical and anal papillomavirus infections.Single and combination herpes simplex virus type 2 glycoprotein vaccines adjuvanted with CpG oligodeoxynucleotides or monophosphoryl lipid A exhibit differential immunity that is not correlated to protection in animal modelsHigh Physiological Concentrations of Progesterone Reverse Estradiol-Mediated Changes in Differentiation and Functions of Bone Marrow Derived Dendritic Cells.Estradiol Enhances CD4+ T-Cell Anti-Viral Immunity by Priming Vaginal DCs to Induce Th17 Responses via an IL-1-Dependent PathwayVaginal epithelial dendritic cells renew from bone marrow precursorsApolipoprotein E alleles can contribute to the pathogenesis of numerous clinical conditions including HSV-1 corneal disease.Psychological stress exacerbates primary vaginal herpes simplex virus type 1 (HSV-1) infection by impairing both innate and adaptive immune responses.Effects of Female Sex Hormones on Susceptibility to HSV-2 in Vaginal Cells Grown in Air-Liquid Interface.Estradiol improves genital herpes vaccine efficacy in mice.HSV-2 vaccine: current status and insight into factors for developing an efficient vaccine.Gender specific differences in the immune response to infection.Influence of sex steroids on inflammation and bone metabolism.Influence of common mucosal co-factors on HIV infection in the female genital tract.Sex Drives Dimorphic Immune Responses to Viral Infections.Progesterone-based compounds affect immune responses and susceptibility to infections at diverse mucosal sites.Characterization of a Hormone-Responsive Organotypic Human Vaginal Tissue Model: Morphologic and Immunologic Effects.A novel role for IL-17 in enhancing type 1 helper T cell immunity in the female genital tract following mucosal HSV-2 vaccination.Estrogen induced changes in uterine brain-derived neurotrophic factor and its receptors.Proinflammatory cytokines and chemokines - but not interferon-β - produced in response to HSV-2 in primary human genital epithelial cells are associated with viral replication and the presence of the virion host shutoff protein.Susceptibility of human female primary genital epithelial cells to herpes simplex virus, type-2 and the effect of TLR3 ligand and sex hormones on infection.Differential responses of murine vaginal and uterine epithelial cells prior to and following herpes simplex virus type 2 (HSV-2) infection.Sperm exposure during menses is a risk factor for developing antisperm antibody (ASA) in female.Primary replication and invasion of the bovine gammaherpesvirus BoHV-4 in the genital mucosae.Hormonal Contraception and HIV-1 Acquisition: Biological Mechanisms.Delayed but effective induction of mucosal memory immune responses against genital HSV-2 in the absence of secondary lymphoid organs.Estradiol impairs the Th17 immune response against Candida albicans
P2860
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P2860
Estradiol regulates susceptibility following primary exposure to genital herpes simplex virus type 2, while progesterone induces inflammation.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մարտին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Estradiol regulates susceptibi ...... esterone induces inflammation.
@ast
Estradiol regulates susceptibi ...... esterone induces inflammation.
@en
type
label
Estradiol regulates susceptibi ...... esterone induces inflammation.
@ast
Estradiol regulates susceptibi ...... esterone induces inflammation.
@en
prefLabel
Estradiol regulates susceptibi ...... esterone induces inflammation.
@ast
Estradiol regulates susceptibi ...... esterone induces inflammation.
@en
P2093
P2860
P1433
P1476
Estradiol regulates susceptibi ...... esterone induces inflammation.
@en
P2093
Amy E Gillgrass
Charu Kaushic
Kenneth L Rosenthal
Sherie A Fernandez
P2860
P304
P356
10.1128/JVI.79.5.3107-3116.2005
P577
2005-03-01T00:00:00Z