NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome. - Wikidata - wikimedia - TriplyDB

NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

http://www.wikidata.org/entity/Q33840366

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Pathophysiology of Pediatric Multiple Organ Dysfunction Syndrome.Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps.Galactomannan and Zymosan Block the Epinephrine-Induced Particle Transport in Tracheal Epithelium.A Common Genetic Variant in TLR1 Enhances Human Neutrophil Priming and Impacts Length of Intensive Care Stay in Pediatric Sepsis.Endothelial cell signaling and ventilator-induced lung injury: molecular mechanisms, genomic analyses, and therapeutic targets.Genetic Phagocyte NADPH Oxidase Deficiency Enhances Nonviable Candida albicans-Induced Inflammation in Mouse Lungs.Neutrophil azurophilic granule exocytosis is primed by TNF-α and partially regulated by NADPH oxidase.

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NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

http://www.wikidata.org/entity/Q33840366

description

2014 nî lūn-bûn

@nan

2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

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2014年論文

@zh-tw

2014年论文

@wuu

name

NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

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NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

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type

label

NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

@ast

NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

@en

prefLabel

NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

@ast

NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

@en

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AJPLUNG.00054.2014

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American Journal of Physiology - Lung Cellular and Molecular Physiology

P1476

NOX2 protects against progressive lung injury and multiple organ dysfunction syndrome.

@en

P2093

Brieanna M Hilkin

http://www.w3.org/2001/XMLSchema#string

Jessica G Moreland

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Jessica S Hook

http://www.w3.org/2001/XMLSchema#string

Kelli L Goss

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Laura C Whitmore

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P2860

Mouse model of X-linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production

The repertoire for pattern recognition of pathogens by the innate immune system is defined by cooperation between toll-like receptors

Dectin-1 is a major beta-glucan receptor on macrophages

Macrophage plasticity and polarization: in vivo veritas

NADPH oxidase limits innate immune responses in the lungs in mice

Acute inflammatory response to endotoxin in mice and humans

Chronic granulomatous disease. Report on a national registry of 368 patients.

Systemic inflammatory response syndrome (SIRS): molecular pathophysiology and gene therapy.

Genetic ablation of phagocytic NADPH oxidase in mice limits TNFα-induced inflammation in the lungs but not other tissues.

Female X-chromosome mosaicism for NOX2 deficiency presents unique inflammatory phenotype and improves outcome in polymicrobial sepsis.

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L71-82

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scholarly article

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10.1152/AJPLUNG.00054.2014

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1

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307

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Elizabeth A Newell

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2014-05-02T00:00:00Z

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4080282

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