Human coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry.
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Targeting membrane-bound viral RNA synthesis reveals potent inhibition of diverse coronaviruses including the middle East respiratory syndrome virusA transmembrane serine protease is linked to the severe acute respiratory syndrome coronavirus receptor and activates virus entryTMPRSS2 and ADAM17 cleave ACE2 differentially and only proteolysis by TMPRSS2 augments entry driven by the severe acute respiratory syndrome coronavirus spike proteinACE2 receptor expression and severe acute respiratory syndrome coronavirus infection depend on differentiation of human airway epithelia.Genetic characterization of Betacoronavirus lineage C viruses in bats reveals marked sequence divergence in the spike protein of pipistrellus bat coronavirus HKU5 in Japanese pipistrelle: implications for the origin of the novel Middle East respiratDesign of wide-spectrum inhibitors targeting coronavirus main proteasesMolecular pathology of emerging coronavirus infectionsCurrent advancements and potential strategies in the development of MERS-CoV vaccinesCrystal structure of NL63 respiratory coronavirus receptor-binding domain complexed with its human receptorCrystal structure of mouse coronavirus receptor-binding domain complexed with its murine receptorCrystal Structure of the Receptor-Binding Domain from Newly Emerged Middle East Respiratory Syndrome CoronavirusGlycan shield and epitope masking of a coronavirus spike protein observed by cryo-electron microscopyFactors determining human-to-human transmissibility of zoonotic pathogens via contactCoronaviruses and the human airway: a universal system for virus-host interaction studiesPhysiology of kidney reninThe nucleocapsid protein of human coronavirus NL63A decade after SARS: strategies for controlling emerging coronavirusesUnderstanding Human Coronavirus HCoV-NL63~!2009-11-13~!2010-04-09~!2010-05-25~!Interaction of severe acute respiratory syndrome-coronavirus and NL63 coronavirus spike proteins with angiotensin converting enzyme-2.Coronavirus diversity, phylogeny and interspecies jumping.Acquisition of cell-cell fusion activity by amino acid substitutions in spike protein determines the infectivity of a coronavirus in cultured cellsDifferential downregulation of ACE2 by the spike proteins of severe acute respiratory syndrome coronavirus and human coronavirus NL63.Coronaviruses post-SARS: update on replication and pathogenesis.Structure, Function, and Evolution of Coronavirus Spike Proteins.Coronavirus antibodies in African bat speciesMechanisms of severe acute respiratory syndrome pathogenesis and innate immunomodulation.Binding of the 5'-untranslated region of coronavirus RNA to zinc finger CCHC-type and RNA-binding motif 1 enhances viral replication and transcriptionCoronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus.Severe acute respiratory syndrome coronavirus infection of human ciliated airway epithelia: role of ciliated cells in viral spread in the conducting airways of the lungs.Viral diversity in asthmaCoronaviruses: an overview of their replication and pathogenesis.Human coronavirus NL63 utilizes heparan sulfate proteoglycans for attachment to target cellsFirst infection by all four non-severe acute respiratory syndrome human coronaviruses takes place during childhood.Role of the spike glycoprotein of human Middle East respiratory syndrome coronavirus (MERS-CoV) in virus entry and syncytia formation.Modulation of the unfolded protein response by the severe acute respiratory syndrome coronavirus spike protein.Receptor recognition mechanisms of coronaviruses: a decade of structural studies.Infection with human coronavirus NL63 enhances streptococcal adherence to epithelial cells.Human Coronavirus HKU1 Spike Protein Uses O-Acetylated Sialic Acid as an Attachment Receptor Determinant and Employs Hemagglutinin-Esterase Protein as a Receptor-Destroying EnzymeThe structure and functions of coronavirus genomic 3' and 5' ends.Evidence for ACE2-utilizing coronaviruses (CoVs) related to severe acute respiratory syndrome CoV in bats.
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P2860
Human coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Human coronavirus NL63 employs ...... s receptor for cellular entry.
@ast
Human coronavirus NL63 employs ...... s receptor for cellular entry.
@en
type
label
Human coronavirus NL63 employs ...... s receptor for cellular entry.
@ast
Human coronavirus NL63 employs ...... s receptor for cellular entry.
@en
prefLabel
Human coronavirus NL63 employs ...... s receptor for cellular entry.
@ast
Human coronavirus NL63 employs ...... s receptor for cellular entry.
@en
P2860
P50
P356
P1476
Human coronavirus NL63 employs ...... s receptor for cellular entry.
@en
P2093
Heike Hofmann
Martina Geier
P2860
P304
P356
10.1073/PNAS.0409465102
P407
P577
2005-05-16T00:00:00Z