Activity-dependent release of endogenous BDNF from mossy fibers evokes a TRPC3 current and Ca2+ elevations in CA3 pyramidal neurons. - Wikidata - wikimedia - TriplyDB

Activity-dependent release of endogenous BDNF from mossy fibers evokes a TRPC3 current and Ca2+ elevations in CA3 pyramidal neurons.

Activity-dependent release of endogenous BDNF from mossy fibers evokes a TRPC3 current and Ca2+ elevations in CA3 pyramidal neurons.

http://www.wikidata.org/entity/Q33841367

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Hyperforin modulates dendritic spine morphology in hippocampal pyramidal neurons by activating Ca(2+) -permeable TRPC6 channels BDNF deregulation in Rett syndrome Brain-derived neurotrophic factor activation of CaM-kinase kinase via transient receptor potential canonical channels induces the translation and synaptic incorporation of GluA1-containing calcium-permeable AMPA receptors Disruption of Src Is Associated with Phenotypes Related to Williams-Beuren Syndrome and Altered Cellular Localization of TFII-I(1,2).Neurobiology of local and intercellular BDNF signaling.Leptin-induced spine formation requires TrpC channels and the CaM kinase cascade in the hippocampus.Vesicular zinc promotes presynaptic and inhibits postsynaptic long-term potentiation of mossy fiber-CA3 synapse.Neurotrophin-mediated dendrite-to-nucleus signaling revealed by microfluidic compartmentalization of dendrites Postsynaptic TRPC1 function contributes to BDNF-induced synaptic potentiation at the developing neuromuscular junction The neurophysiology and pathology of brain zinc.Aging-induced Seizure-related Changes to the Hippocampal Mossy Fiber Pathway in Forebrain Specific BDNF Overexpressing Mice BDNF and its pro-peptide are stored in presynaptic dense core vesicles in brain neurons.Distinct subsets of Syt-IV/BDNF vesicles are sorted to axons versus dendrites and recruited to synapses by activity Intracellular Ca2+ stores and Ca2+ influx are both required for BDNF to rapidly increase quantal vesicular transmitter release.Extracellular proteolysis in structural and functional plasticity of mossy fiber synapses in hippocampus Activity-dependent BDNF release and TRPC signaling is impaired in hippocampal neurons of Mecp2 mutant mice Calcium-Dependent and Synapsin-Dependent Pathways for the Presynaptic Actions of BDNF.The role of transient receptor potential cation channels in Ca2+ signaling Role for Endogenous BDNF in Endocannabinoid-Mediated Long-Term Depression at Neocortical Inhibitory Synapses(,.)Mechanisms of BDNF regulation in asthmatic airway smooth muscle.Contribution of TRPC3 to store-operated calcium entry and inflammatory transductions in primary nociceptors.TRPC6 channel-mediated neurite outgrowth in PC12 cells and hippocampal neurons involves activation of RAS/MEK/ERK, PI3K, and CAMKIV signaling.TRPC3 channels play a critical role in the theta component of pilocarpine-induced status epilepticus in mice.

P2860

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P2860

Activity-dependent release of endogenous BDNF from mossy fibers evokes a TRPC3 current and Ca2+ elevations in CA3 pyramidal neurons.

http://www.wikidata.org/entity/Q33841367

description

2010 nî lūn-bûn

@nan

2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

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Activity-dependent release of ...... ions in CA3 pyramidal neurons.

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P1433

Journal of Neurophysiology

P1476

Activity-dependent release of ...... ions in CA3 pyramidal neurons.

@en

P2093

Gaston Calfa

http://www.w3.org/2001/XMLSchema#string

Lucas Pozzo-Miller

http://www.w3.org/2001/XMLSchema#string

Michelle D Amaral

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Takafumi Inoue

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Yong Li

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P2860

Involvement of BDNF receptor TrkB in spatial memory formation

RIM1alpha phosphorylation at serine-413 by protein kinase A is not required for presynaptic long-term plasticity or learning

A new generation of Ca2+ indicators with greatly improved fluorescence properties

The TRP channels, a remarkably functional family

Neurotrophin-evoked depolarization requires the sodium channel Na(V)1.9

Human trks: molecular cloning, tissue distribution, and expression of extracellular domain immunoadhesins

Synaptic secretion of BDNF after high-frequency stimulation of glutamatergic synapses.

BDNF enhances quantal neurotransmitter release and increases the number of docked vesicles at the active zones of hippocampal excitatory synapses

RIM1alpha is required for presynaptic long-term potentiation

BDNF-mediated neurotransmission relies upon a myosin VI motor complex

P304

2846-2856

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scholarly article

P356

10.1152/JN.01140.2009

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P433

5

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P478

103

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2010-03-10T00:00:00Z

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41894793

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20220070

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2867575

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