A mechanism of palindromic gene amplification in Saccharomyces cerevisiae. - Wikidata - wikimedia - TriplyDB

A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

http://www.wikidata.org/entity/Q33841787

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Mechanisms of gene duplication and amplification Telomere dysfunction and chromosome instability DNA end resection: many nucleases make light work Replication stalling at unstable inverted repeats: interplay between DNA hairpins and fork stabilizing proteins New approaches to the analysis of palindromic sequences from the human genome: evolution and polymorphism of an intronic site at the NF1 locus Inverted repeat-stimulated sister-chromatid exchange events are RAD1-independent but reduced in a msh2 mutant.Replication protein A prevents promiscuous annealing between short sequence homologies: Implications for genome integrity Sae2 is an endonuclease that processes hairpin DNA cooperatively with the Mre11/Rad50/Xrs2 complex.Phosphorylation-regulated transitions in an oligomeric state control the activity of the Sae2 DNA repair enzyme.Minding the gap: the underground functions of BRCA1 and BRCA2 at stalled replication forks DNA repair pathway selection caused by defects in TEL1, SAE2, and de novo telomere addition generates specific chromosomal rearrangement signatures.Fusion of nearby inverted repeats by a replication-based mechanism leads to formation of dicentric and acentric chromosomes that cause genome instability in budding yeast.GAP-Seq: a method for identification of DNA palindromes.CtIP maintains stability at common fragile sites and inverted repeats by end resection-independent endonuclease activity.End resection at double-strand breaks: mechanism and regulation.The distribution of inverted repeat sequences in the Saccharomyces cerevisiae genome.The role of replication bypass pathways in dicentric chromosome formation in budding yeast RPA coordinates DNA end resection and prevents formation of DNA hairpins.The mitochondrial genome of the glomeromycete Rhizophagus sp. DAOM 213198 reveals an unusual organization consisting of two circular chromosomes.RPA antagonizes microhomology-mediated repair of DNA double-strand breaks.Replication fork integrity and intra-S phase checkpoint suppress gene amplification.Intrastrand annealing leads to the formation of a large DNA palindrome and determines the boundaries of genomic amplification in human cancer Mre11-Sae2 and RPA Collaborate to Prevent Palindromic Gene Amplification E. coli SbcCD and RecA control chromosomal rearrangement induced by an interrupted palindrome.DNA palindromes with a modest arm length of greater, similar 20 base pairs are a significant target for recombinant adeno-associated virus vector integration in the liver, muscles, and heart in mice.Multiple pathways of duplication formation with and without recombination (RecA) in Salmonella enterica.Palindromic amplification of the ERBB2 oncogene in primary HER2-positive breast tumors Gene copy-number variation in haploid and diploid strains of the yeast Saccharomyces cerevisiae Glioblastoma multiforme: the role of DSB repair between genotype and phenotype.De novo-generated small palindromes are characteristic of amplicon boundary junction of double minutes.Mechanism and regulation of DNA end resection in eukaryotes XRCC2 and XRCC3 regulate the balance between short- and long-tract gene conversions between sister chromatids.Homology-mediated end-capping as a primary step of sister chromatid fusion in the breakage-fusion-bridge cycles.Chromosome aberrations resulting from double-strand DNA breaks at a naturally occurring yeast fragile site composed of inverted ty elements are independent of Mre11p and Sae2p.Mechanisms and regulation of DNA end resection.Making the best of the loose ends: Mre11/Rad50 complexes and Sae2 promote DNA double-strand break resection.Unprecedented large inverted repeats at the replication terminus of circular bacterial chromosomes suggest a novel mode of chromosome rescue.Differential regulation of short- and long-tract gene conversion between sister chromatids by Rad51C.Functions and regulation of the MRX complex at DNA double-strand breaks.Large inverted duplications in the human genome form via a fold-back mechanism.

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P2860

A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

http://www.wikidata.org/entity/Q33841787

description

2005 nî lūn-bûn

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2005 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2005 թվականի հունիսին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

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A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

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type

label

A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

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A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

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prefLabel

A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

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A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

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A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.

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Alison J Rattray

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Beena Neelam

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Brenda K Shafer

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Jeffrey N Strathern

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P2860

Mre11 and Ku70 interact in somatic cells, but are differentially expressed in early meiosis

The Rad50 zinc-hook is a structure joining Mre11 complexes in DNA recombination and repair

The Stability of Broken Ends of Chromosomes in Zea Mays.

Inverted repeats as genetic elements for promoting DNA inverted duplication: implications in gene amplification

Saccharomyces cerevisiae Ku70 potentiates illegitimate DNA double-strand break repair and serves as a barrier to error-prone DNA repair pathways

Base-calling of automated sequencer traces using phred. I. Accuracy assessment

A general method for identifying recessive diploid-specific mutations in Saccharomyces cerevisiae, its application to the isolation of mutants blocked at intermediate stages of meiotic prophase and characterization of a new gene SAE2.

The nuclease activity of Mre11 is required for meiosis but not for mating type switching, end joining, or telomere maintenance.

5-Fluoroorotic acid as a selective agent in yeast molecular genetics

A method for gene disruption that allows repeated use of URA3 selection in the construction of multiply disrupted yeast strains

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10.1101/GAD.1315805

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11

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Saccharomyces cerevisiae

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1142561

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