Inactivation of p21 by E1A leads to the induction of apoptosis in DNA-damaged cells.
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Cell cycle regulation and p53 activation by protein phosphatase 2C alphaAdenovirus-mediated sensitization to the cytotoxic drugs docetaxel and mitoxantrone is dependent on regulatory domains in the E1ACR1 gene-regionThe dynamics of E1A in regulating networks and canonical pathways in quiescent cellsp400 is required for E1A to promote apoptosis.Recent lessons in gene expression, cell cycle control, and cell biology from adenovirus.Armoring CRAds with p21/Waf-1 shRNAs: the next generation of oncolytic adenoviruses.The adenovirus e3 promoter is sensitive to activation signals in human T cells.Comparative sequence analysis of the largest E1A proteins of human and simian adenoviruses.Growth arrest in ovarian cancer cells by hTERT inhibition short-hairpin RNA targeting human telomerase reverse transcriptase induces immediate growth inhibition but not necessarily induces apoptosis in ovarian cancer cells.Oncolytic adenoviral mutants with E1B19K gene deletions enhance gemcitabine-induced apoptosis in pancreatic carcinoma cells and anti-tumor efficacy in vivo.Identification of p53-46F as a super p53 with an enhanced ability to induce p53-dependent apoptosis.Rosmarinic acid potentiates carnosic acid induced apoptosis in lung fibroblasts.Coordinate inhibition of cytokine-mediated induction of ferritin H, manganese superoxide dismutase, and interleukin-6 by the adenovirus E1A oncogene.Epstein-Barr virus can inhibit genotoxin-induced G1 arrest downstream of p53 by preventing the inactivation of CDK2.miRNAs as Potential Treatment Targets and Treatment Options in Cancer.Paclitaxel and vincristine potentiate adenoviral oncolysis that is associated with cell cycle and apoptosis modulation, whereas they differentially affect the viral life cycle in non-small-cell lung cancer cells.Camptothecin exhibits topoisomerase1-independent KMT1A suppression and myogenic differentiation in alveolar rhabdomyosarcoma cells.Loss of Cyclin-Dependent Kinase Inhibitor Alters Oncolytic Adenovirus Replication and Promotes More Efficient Virus Production.
P2860
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P2860
Inactivation of p21 by E1A leads to the induction of apoptosis in DNA-damaged cells.
description
2001 nî lūn-bûn
@nan
2001 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Inactivation of p21 by E1A leads to the induction of apoptosis in DNA-damaged cells.
@ast
Inactivation of p21 by E1A leads to the induction of apoptosis in DNA-damaged cells.
@en
type
label
Inactivation of p21 by E1A leads to the induction of apoptosis in DNA-damaged cells.
@ast
Inactivation of p21 by E1A leads to the induction of apoptosis in DNA-damaged cells.
@en
prefLabel
Inactivation of p21 by E1A leads to the induction of apoptosis in DNA-damaged cells.
@ast
Inactivation of p21 by E1A leads to the induction of apoptosis in DNA-damaged cells.
@en
P2093
P2860
P1433
P1476
Inactivation of p21 by E1A leads to the induction of apoptosis in DNA-damaged cells.
@en
P2093
D Chattopadhyay
M L Harter
P2860
P304
P356
10.1128/JVI.75.20.9844-9856.2001
P577
2001-10-01T00:00:00Z