RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury. - Wikidata - wikimedia - TriplyDB

RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

http://www.wikidata.org/entity/Q33848879

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Accessory proteins for heterotrimeric G-proteins in the kidney CD47 blockade reduces ischemia-reperfusion injury and improves outcomes in a rat kidney transplant model.Mouse model of ischemic acute kidney injury: technical notes and tricks miRNA profiling discriminates types of rejection and injury in human renal allografts Asymmetric dimethylarginine accumulates in the kidney during ischemia/reperfusion injury.The nephroprotective properties of recombinant human erythropoietin in kidney transplantation: experimental facts and clinical proofs.RGS4 Overexpression in Lung Attenuates Airway Hyper-responsiveness in Mice.Modification and functional inhibition of regulator of G-protein signaling 4 (RGS4) by 4-hydroxy-2-nonenal.RGS4 inhibits angiotensin II signaling and macrophage localization during renal reperfusion injury independent of vasospasm.Netrin-1 pretreatment protects rat kidney against ischemia/reperfusion injury via suppression of oxidative stress and neuropeptide Y expression.Suppression of renal TRPM7 may alleviate kidney injury in the renal transplantation.Regulating the regulators: Epigenetic, transcriptional, and post-translational regulation of RGS proteins.Human vascular progenitor cells derived from renal arteries are endothelial-like and assist in the repair of injured renal capillary networks.

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RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

http://www.wikidata.org/entity/Q33848879

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2011 nî lūn-bûn

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2011 թուականի Մարտին հրատարակուած գիտական յօդուած

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2011 թվականի մարտին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

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RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

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RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

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RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

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RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

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RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

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RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.

@en

P2093

Andrew M Siedlecki

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Anthony J Muslin

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Keith A Hruska

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Winston Thomas

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Xiaohua Jin

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P2860

Inhibition of G-protein-mediated MAP kinase activation by a new mammalian gene family

Upregulation of RGS4 expression by IL-1beta in colonic smooth muscle is enhanced by ERK1/2 and p38 MAPK and inhibited by the PI3K/Akt/GSK3beta pathway

Structure of RGS4 bound to AlF4--activated G(i alpha1): stabilization of the transition state for GTP hydrolysis

N-terminal residues control proteasomal degradation of RGS2, RGS4, and RGS5 in human embryonic kidney 293 cells

GAIP and RGS4 are GTPase-activating proteins for the Gi subfamily of G protein alpha subunits

RGS family members: GTPase-activating proteins for heterotrimeric G-protein alpha-subunits

Regulator of G protein signaling 2 mediates cardiac compensation to pressure overload and antihypertrophic effects of PDE5 inhibition in mice

Support of renal blood flow after ischaemic-reperfusion injury by endogenous formation of nitric oxide and of cyclo-oxygenase vasodilator metabolites.

Real-time monitoring of nitric oxide in ischemia-reperfusion rat kidney.

The use of the endothelin receptor antagonist, tezosentan, before or after renal ischemia protects renal function.

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10.1038/KI.2011.63

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reperfusion injury

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