Impaired vasodilation by red blood cells in sickle cell disease. - Wikidata - wikimedia - TriplyDB

Impaired vasodilation by red blood cells in sickle cell disease.

Impaired vasodilation by red blood cells in sickle cell disease.

http://www.wikidata.org/entity/Q33850500

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Hypoxic Vasodilation by Red Blood Cells: Evidence for an S-Nitrosothiol-Based Signal Angiotensin-converting enzyme (ACE) inhibitors for proteinuria and microalbuminuria in people with sickle cell disease Angiotensin-converting enzyme (ACE) inhibitors for proteinuria and microalbuminuria in people with sickle cell disease Angiotensin-converting enzyme (ACE) inhibitors for proteinuria and microalbuminuria in people with sickle cell disease Regulation of cardiovascular cellular processes by S-nitrosylation S-nitrosylation in cardiovascular signaling S-nitrosothiol signaling in respiratory biology.Red cell physiology and signaling relevant to the critical care setting Application of a nitric oxide sensor in biomedicine 2015 Clinical trials update in sickle cell anemia Mechanism-based triarylphosphine-ester probes for capture of endogenous RSNOs Extrapulmonary effects of inhaled nitric oxide: role of reversible S-nitrosylation of erythrocytic hemoglobin.S-nitrosothiol depletion in amyotrophic lateral sclerosis.Hypoxic adaptation during development: relation to pattern of neurological presentation and cognitive disability.Avicinylation (thioesterification): a protein modification that can regulate the response to oxidative and nitrosative stress.L-arginine/NO pathway is altered in children with haemolytic-uraemic syndrome (HUS).Inclusion of a nitric oxide congener in the insufflation gas repletes S-nitrosohemoglobin and stabilizes physiologic status during prolonged carbon dioxide pneumoperitoneum.Evolution of adverse changes in stored RBCs S-nitrosohemoglobin deficiency: a mechanism for loss of physiological activity in banked blood.A nitric oxide processing defect of red blood cells created by hypoxia: deficiency of S-nitrosohemoglobin in pulmonary hypertension.Sodium-dependent bile salt transporters of the SLC10A transporter family: more than solute transporters.Identification of S-nitrosylation motifs by site-specific mapping of the S-nitrosocysteine proteome in human vascular smooth muscle cells An S-nitrosothiol (SNO) synthase function of hemoglobin that utilizes nitrite as a substrate.Protein S-nitrosylation in health and disease: a current perspective.Pharmacotherapy in sickle cell disease--state of the art and future prospects Computation of plasma hemoglobin nitric oxide scavenging in hemolytic anemias.Modulation of outer medullary NaCl transport and oxygenation by nitric oxide and superoxide Nitric oxide synthase generates nitric oxide locally to regulate compartmentalized protein S-nitrosylation and protein trafficking.Hemoglobin βCys93 is essential for cardiovascular function and integrated response to hypoxia.Dynamic denitrosylation via S-nitrosoglutathione reductase regulates cardiovascular function.Assessment of nitric oxide signals by triiodide chemiluminescence.Lactate dehydrogenase as a biomarker of hemolysis-associated nitric oxide resistance, priapism, leg ulceration, pulmonary hypertension, and death in patients with sickle cell disease.Enzymatic mechanisms regulating protein S-nitrosylation: implications in health and disease.Effect of processing and storage on red blood cell function in vivo HLA class II haplotypes distinctly associated with vaso-occlusion in children with sickle cell disease.Beyond hydroxyurea: new and old drugs in the pipeline for sickle cell disease.Anemia and splenomegaly in cGKI-deficient mice.Sickle hemoglobin disturbs normal coupling among erythrocyte O2 content, glycolysis, and antioxidant capacity.Transport and peripheral bioactivities of nitrogen oxides carried by red blood cell hemoglobin: role in oxygen delivery.Repletion of S-nitrosohemoglobin improves organ function and physiological status in swine after brain death.

P2860

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P2860

Impaired vasodilation by red blood cells in sickle cell disease.

http://www.wikidata.org/entity/Q33850500

description

2005 nî lūn-bûn

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2005 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2005 թվականի փետրվարին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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Impaired vasodilation by red blood cells in sickle cell disease.

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Impaired vasodilation by red blood cells in sickle cell disease.

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Impaired vasodilation by red blood cells in sickle cell disease.

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Impaired vasodilation by red blood cells in sickle cell disease.

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Impaired vasodilation by red blood cells in sickle cell disease.

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Impaired vasodilation by red blood cells in sickle cell disease.

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Impaired vasodilation by red blood cells in sickle cell disease.

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P2093

Douglas T Hess

http://www.w3.org/2001/XMLSchema#string

John R Pawloski

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Jonathan S Stamler

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P2860

Crystallographic analysis of the interaction of nitric oxide with quaternary-T human hemoglobin

Essential roles of S-nitrosothiols in vascular homeostasis and endotoxic shock

Export by red blood cells of nitric oxide bioactivity.

Endothelial dysfunction in patients with sickle cell disease is related to selective impairment of shear stress-mediated vasodilation.

Sickle cell anemia is associated with reduced nitric oxide bioactivity in peripheral conduit and resistance vessels.

S-nitrosothiols signal the ventilatory response to hypoxia.

Cell-free hemoglobin limits nitric oxide bioavailability in sickle-cell disease.

Routes to S-nitroso-hemoglobin formation with heme redox and preferential reactivity in the beta subunits.

Abnormal redox status of membrane-protein thiols in sickle erythrocytes.

Interaction of sickle cell hemoglobin with erythrocyte membranes

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