Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo
about
Angptl4 protects against severe proinflammatory effects of saturated fat by inhibiting fatty acid uptake into mesenteric lymph node macrophagesA critical role for the Sp1-binding sites in the transforming growth factor-beta-mediated inhibition of lipoprotein lipase gene expression in macrophagesLack of macrophage fatty-acid-binding protein aP2 protects mice deficient in apolipoprotein E against atherosclerosisTissue-specific expression of human lipoprotein lipase in the vascular system affects vascular reactivity in transgenic miceIncreased atherosclerosis in LDL receptor-null mice lacking ACAT1 in macrophagesIncreased atherosclerosis in myeloperoxidase-deficient micePPARalpha deficiency reduces insulin resistance and atherosclerosis in apoE-null miceLipid-laden partially-activated plasmacytoid and CD4(-)CD8α(+) dendritic cells accumulate in tissues in elderly miceMacrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in low density lipoprotein receptor-deficient miceLipoprotein lipase, a key role in atherosclerosis?A novel role of Sp1 and Sp3 in the interferon-gamma -mediated suppression of macrophage lipoprotein lipase gene transcription.Combined adipocyte-macrophage fatty acid-binding protein deficiency improves metabolism, atherosclerosis, and survival in apolipoprotein E-deficient mice.Adipocyte fatty acid-binding protein, aP2, alters late atherosclerotic lesion formation in severe hypercholesterolemiaRescue of cardiomyopathy in peroxisome proliferator-activated receptor-alpha transgenic mice by deletion of lipoprotein lipase identifies sources of cardiac lipids and peroxisome proliferator-activated receptor-alpha activatorsFunction and Therapeutic Potential of Mesenchymal Stem Cells in Atherosclerosis.Inhibition of cardiac lipoprotein utilization by transgenic overexpression of Angptl4 in the heart.N-3 vs. saturated fatty acids: effects on the arterial wallMechanisms of lipase maturation.Efficient phagocytosis requires triacylglycerol hydrolysis by adipose triglyceride lipaseAmelioration of hypertriglyceridemia with hypo-alpha-cholesterolemia in LPL deficient mice by hematopoietic cell-derived LPL.Endothelial-derived tissue factor pathway inhibitor regulates arterial thrombosis but is not required for development or hemostasis.Conditional disruption of the peroxisome proliferator-activated receptor gamma gene in mice results in lowered expression of ABCA1, ABCG1, and apoE in macrophages and reduced cholesterol efflux.Selective macrophage ascorbate deficiency suppresses early atherosclerosis.Macrophage adipose triglyceride lipase deficiency attenuates atherosclerotic lesion development in low-density lipoprotein receptor knockout miceEndothelial lipase is a major determinant of HDL level.The macrophage foam cell as a target for therapeutic intervention.Triglycerides and heart disease: still a hypothesis?Monocyte to macrophage differentiation goes along with modulation of the plasmalogen pattern through transcriptional regulation.Hepatic lipase expression in macrophages contributes to atherosclerosis in apoE-deficient and LCAT-transgenic mice.Deficiency of receptor-associated protein attenuates angiotensin II-induced atherosclerosis in hypercholesterolemic mice without influencing abdominal aortic aneurysmsMicroRNA-590 Inhibits Lipoprotein Lipase Expression and Prevents Atherosclerosis in apoE Knockout Mice.Active autophagy but not lipophagy in macrophages with defective lipolysis.MicroRNA-27 Prevents Atherosclerosis by Suppressing Lipoprotein Lipase-Induced Lipid Accumulation and Inflammatory Response in Apolipoprotein E Knockout MiceThematic review series: The immune system and atherogenesis. Cytokine regulation of macrophage functions in atherogenesis.Macrophage deficiency of Akt2 reduces atherosclerosis in Ldlr null miceHydrolysis products generated by lipoprotein lipase and endothelial lipase differentially impact THP-1 macrophage cell signalling pathways.Macrophage lipoprotein lipase modulates the development of atherosclerosis but not adiposity.Macrophage IKKα Deficiency Suppresses Akt Phosphorylation, Reduces Cell Survival, and Decreases Early AtherosclerosisMacrophage EP4 deficiency increases apoptosis and suppresses early atherosclerosis.Cyclooxygenase products and atherosclerosis
P2860
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P2860
Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo
@ast
Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo
@en
type
label
Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo
@ast
Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo
@en
prefLabel
Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo
@ast
Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo
@en
P2093
P2860
P921
P356
P1476
Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo
@en
P2093
C F Semenkovich
K J Carter
L A Gleaves
M F Linton
V R Babaev
P2860
P304
P356
10.1172/JCI6117
P407
P577
1999-06-01T00:00:00Z