Phenotypes of rotavirus reassortants depend upon the recipient genetic background.
about
Viruses and cells with mutations affecting viral entry are selected during persistent rotavirus infections of MA104 cellsSubunit rotavirus vaccine administered parenterally to rabbits induces active protective immunity.Structures of rotavirus reassortants demonstrate correlation of altered conformation of the VP4 spike and expression of unexpected VP4-associated phenotypesNew insights into rotavirus entry machinery: stabilization of rotavirus spike conformation is independent of trypsin cleavage.Diversity in Indian equine rotaviruses: identification of genotype G10,P6[1] and G1 strains and a new VP7 genotype (G16) strain in specimens from diarrheic foals in India.Genetic mapping of reovirus virulence and organ tropism in severe combined immunodeficient mice: organ-specific virulence genes.Genetic mapping indicates that VP4 is the rotavirus cell attachment protein in vitro and in vivo.Interactions between the two surface proteins of rotavirus may alter the receptor-binding specificity of the virusIntegrin-using rotaviruses bind alpha2beta1 integrin alpha2 I domain via VP4 DGE sequence and recognize alphaXbeta2 and alphaVbeta3 by using VP7 during cell entryThe VP7 genes of two G9 rotaviruses isolated in 1980 from diarrheal stool samples collected in Washington, DC, are unique molecularly and serotypicallyCharacterization of virus-like particles produced by the expression of rotavirus capsid proteins in insect cellsTemplate-dependent, in vitro replication of rotavirus RNA.Murine rotavirus genes encoding outer capsid proteins VP4 and VP7 are not major determinants of host range restriction and virulenceNucleotide sequence of VP4 and VP7 genes of human rotaviruses with subgroup I specificity and long RNA pattern: implication for new G serotype specificityRotavirus genome segment 4 determines viral replication phenotype in cultured liver cells (HepG2).Passive immunity modulates genetic reassortment between rotaviruses in mixedly infected mice.The VP8* domain of neonatal rotavirus strain G10P[11] binds to type II precursor glycansRotavirus gene structure and function.Neutralizing antibodies to heterologous animal rotavirus serotypes 5, 6, 7, and 10 in sera from Ecuadorian children.Trypsin cleavage stabilizes the rotavirus VP4 spike.Rotavirus-induced fusion from without in tissue culture cells.Viral determinants of rotavirus pathogenicity in pigs: production of reassortants by asynchronous coinfection.Virus-like particle-induced fusion from without in tissue culture cells: role of outer-layer proteins VP4 and VP7.Immunodominance of the VP4 neutralization protein of rotavirus in protective natural infections of young children.Specific interactions between rotavirus outer capsid proteins VP4 and VP7 determine expression of a cross-reactive, neutralizing VP4-specific epitope.Simian rotavirus SA11 strains.
P2860
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P2860
Phenotypes of rotavirus reassortants depend upon the recipient genetic background.
description
1989 nî lūn-bûn
@nan
1989 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Phenotypes of rotavirus reassortants depend upon the recipient genetic background.
@ast
Phenotypes of rotavirus reassortants depend upon the recipient genetic background.
@en
type
label
Phenotypes of rotavirus reassortants depend upon the recipient genetic background.
@ast
Phenotypes of rotavirus reassortants depend upon the recipient genetic background.
@en
prefLabel
Phenotypes of rotavirus reassortants depend upon the recipient genetic background.
@ast
Phenotypes of rotavirus reassortants depend upon the recipient genetic background.
@en
P2093
P2860
P356
P1476
Phenotypes of rotavirus reassortants depend upon the recipient genetic background.
@en
P2093
P2860
P304
P356
10.1073/PNAS.86.10.3743
P407
P577
1989-05-01T00:00:00Z