Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.
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Treatment of allergic asthma: modulation of Th2 cells and their responsesInterleukin 17 contributes to the chronicity of inflammatory diseases such as rheumatoid arthritisInherent low Erk and p38 activity reduce Fas Ligand expression and degranulation in T helper 17 cells leading to activation induced cell death resistanceEctopic expression of Fas Ligand on cardiomyocytes renders cardiac allografts resistant to CD4(+) T-cell mediated rejection.Th17 cell-mediated neuroinflammation is involved in neurodegeneration of aβ1-42-induced Alzheimer's disease model rats.Toxoplasma gondii prevalent in China induce weaker apoptosis of neural stem cells C17.2 via endoplasmic reticulum stress (ERS) signaling pathways.Apoptosis modulation as a promising target for treatment of systemic sclerosis.CD8+ T cells induce thyroid epithelial cell hyperplasia and fibrosis.TGF-β promotes proliferation of thyroid epithelial cells in IFN-γ(-/-) mice by down-regulation of p21 and p27 via AKT pathwayReactive oxygen species-triggered trophoblast apoptosis is initiated by endoplasmic reticulum stress via activation of caspase-12, CHOP, and the JNK pathway in Toxoplasma gondii infection in mice.Mechanisms underlying antigen-specific tolerance of stable and convertible Th17 cells during suppression of autoimmune diabetes.FAS-ligand regulates differential activation-induced cell death of human T-helper 1 and 17 cells in healthy donors and multiple sclerosis patientsDistinct different sensitivity of Treg and Th17 cells to Fas-mediated apoptosis signaling in patients with acute coronary syndrome.NLRP12 modulates host defense through IL-17A-CXCL1 axis.FLIP: molecular switch between apoptosis and necroptosis.The role of interleukin-6 signalling and its therapeutic blockage in skewing the T cell balance in rheumatoid arthritis.Organ-specific protection mediated by cooperation between vascular and epithelial barriers.Tissue-based class control: the other side of tolerance.Murine Th17 cells utilize IL-2 receptor gamma chain cytokines but are resistant to cytokine withdrawal-induced apoptosis.Interleukin-17 and interleukin-23: importance in the pathogenesis of lung impairment in patients with systemic sclerosisNon-apoptotic Fas (CD95) Signaling on T Cells Regulates the Resolution of Th2-Mediated Inflammation
P2860
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P2860
Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.
@ast
Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.
@en
type
label
Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.
@ast
Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.
@en
prefLabel
Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.
@ast
Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.
@en
P2093
P2860
P356
P1476
Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.
@en
P2093
Helen Braley-Mullen
Jason S Ellis
Shiguang Yu
Tumenjargal Sharav
Yujiang Fang
P2860
P304
P356
10.1189/JLB.0509352
P577
2010-06-01T00:00:00Z