Widespread hypomethylation occurs early and synergizes with gene amplification during esophageal carcinogenesis. - Wikidata - wikimedia - TriplyDB

Widespread hypomethylation occurs early and synergizes with gene amplification during esophageal carcinogenesis.

Widespread hypomethylation occurs early and synergizes with gene amplification during esophageal carcinogenesis.

http://www.wikidata.org/entity/Q33869648

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Signaling pathways in the molecular pathogenesis of adenocarcinomas of the esophagus and gastroesophageal junction Epigenetic subgroups of esophageal and gastric adenocarcinoma with differential GATA5 DNA methylation associated with clinical and lifestyle factors An integrative analysis reveals functional targets of GATA6 transcriptional regulation in gastric cancer.Methylome profiling reveals distinct alterations in phenotypic and mutational subgroups of myeloproliferative neoplasms Relationship between methylation and colonic inflammation in inflammatory bowel disease.Base-pair resolution DNA methylation sequencing reveals profoundly divergent epigenetic landscapes in acute myeloid leukemia.Next-generation sequencing of endoscopic biopsies identifies ARID1A as a tumor-suppressor gene in Barrett's esophagus Aberrant epigenetic and genetic marks are seen in myelodysplastic leukocytes and reveal Dock4 as a candidate pathogenic gene on chromosome 7q Kidney cancer is characterized by aberrant methylation of tissue-specific enhancers that are prognostic for overall survival.Pancreatic cancer patient survival correlates with DNA methylation of pancreas development genes.Genetic and Epigenetic Alterations in Barrett's Esophagus and Esophageal Adenocarcinoma.Heritable transmission of diabetic metabolic memory in zebrafish correlates with DNA hypomethylation and aberrant gene expression The Role of Gastroesophageal Reflux and Other Factors during Progression to Esophageal Adenocarcinoma Methylation of Werner syndrome protein is associated with the occurrence and development of invasive meningioma via the regulation of Myc and p53 expression A comprehensive survey of genomic alterations in gastric cancer reveals systematic patterns of molecular exclusivity and co-occurrence among distinct therapeutic targets.Dissecting characteristics and dynamics of differentially expressed proteins during multistage carcinogenesis of human colorectal cancer.Myeloma is characterized by stage-specific alterations in DNA methylation that occur early during myelomagenesis Targeting chemokine pathways in esophageal adenocarcinoma.Gastrointestinal adenocarcinomas of the esophagus, stomach, and colon exhibit distinct patterns of genome instability and oncogenesis.Loss of fragile histidine triad and amplification of 1p36.22 and 11p15.5 in primary gastric adenocarcinomas.Cancer as a dysregulated epigenome allowing cellular growth advantage at the expense of the host Supportive evidence for FOXP1, BARX1, and FOXF1 as genetic risk loci for the development of esophageal adenocarcinoma.High resolution methylome analysis reveals widespread functional hypomethylation during adult human erythropoiesis.Identification of the CIMP-like subtype and aberrant methylation of members of the chromosomal segregation and spindle assembly pathways in esophageal adenocarcinoma.Hypomethylation of noncoding DNA regions and overexpression of the long noncoding RNA, AFAP1-AS1, in Barrett's esophagus and esophageal adenocarcinoma.DNA methylation as an adjunct to histopathology to detect prevalent, inconspicuous dysplasia and early-stage neoplasia in Barrett's esophagus Genome-wide methylation analysis shows similar patterns in Barrett's esophagus and esophageal adenocarcinoma.Spatial intratumoral heterogeneity and temporal clonal evolution in esophageal squamous cell carcinoma Real-time methylomic aberrations during initiation and progression of induced human mammary epithelial cell tumorigenesis Molecular mechanisms of Barrett's esophagus.Epigenetic biomarkers in esophageal cancer.The role of CXC chemokines and their receptors in the progression and treatment of tumors.The '-omics' revolution and oesophageal adenocarcinoma.Cellular origins and molecular mechanisms of Barrett's esophagus and esophageal adenocarcinoma.Epigenetics in esophageal cancers.The use of imaging and biomarkers in diagnosing Barrett's esophagus and predicting the risk of neoplastic progression.High resolution integrative analysis reveals widespread genetic and epigenetic changes after chronic in-vitro acid and bile exposure in Barrett's epithelium cells.Selective inhibition of esophageal cancer cells by combination of HDAC inhibitors and Azacytidine Genomics of Esophageal Cancer and Biomarkers for Early Detection.Genome-wide hydroxymethylation tested using the HELP-GT assay shows redistribution in cancer.

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P2860

Widespread hypomethylation occurs early and synergizes with gene amplification during esophageal carcinogenesis.

http://www.wikidata.org/entity/Q33869648

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2011 nî lūn-bûn

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2011 թուականի Մարտին հրատարակուած գիտական յօդուած

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2011 թվականի մարտին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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Amit Verma

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Anirban Maitra

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Ari Melnick

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Christina Montagna

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Davendra Sohal

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Elizabeth A Montgomery

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Hector Alvarez

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J Mollenhauer

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Jean Wang

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Joanna Opalinska

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P2860

Differential methylation of tissue- and cancer-specific CpG island shores distinguishes human induced pluripotent stem cells, embryonic stem cells and fibroblasts

Integrated analysis of homozygous deletions, focal amplifications, and sequence alterations in breast and colorectal cancers

Inflammation and cancer

Comparative isoschizomer profiling of cytosine methylation: the HELP assay

Inactivation of p16, RUNX3, and HPP1 occurs early in Barrett's-associated neoplastic progression and predicts progression risk

Radiofrequency ablation in Barrett's esophagus with dysplasia

Chemokine signaling via the CXCR2 receptor reinforces senescence

Hypomethylation distinguishes genes of some human cancers from their normal counterparts

Transcriptional oncogenomic hot spots in Barrett's adenocarcinomas: serial analysis of gene expression.

An integrative genomic and epigenomic approach for the study of transcriptional regulation

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