Persistent transactivation of EGFR and ErbB2/HER2 by protease-activated receptor-1 promotes breast carcinoma cell invasion.
about
Unravelling the molecular complexity of GPCR-mediated EGFR transactivation using functional genomics approachesEthanol enhances the interaction of breast cancer cells over-expressing ErbB2 with fibronectinEtk/Bmx regulates proteinase-activated-receptor1 (PAR1) in breast cancer invasion: signaling partners, hierarchy and physiological significanceComparative expression study of the endo-G protein coupled receptor (GPCR) repertoire in human glioblastoma cancer stem-like cells, U87-MG cells and non malignant cells of neural origin unveils new potential therapeutic targetsThe Transmodulation of HER2 and EGFR by Substance P in Breast Cancer Cells Requires c-Src and Metalloproteinase ActivationTransactivation of EGFR/PI3K/Akt involved in ATP-induced inflammatory protein expression and cell motilityLgr5 over-expression is positively related to the tumor progression and HER2 expression in stage pTNM IV colorectal cancerPRSS3 is a prognostic marker in invasive ductal carcinoma of the breast.Tissue factor and PAR2 signaling in the tumor microenvironmentInvestigating AP-2 and YY1 protein expression as a cause of high HER2 gene transcription in breast cancers with discordant HER2 gene amplification.Promotion of human trophoblasts invasion by gonadotropin-releasing hormone (GnRH) I and GnRH II via distinct signaling pathways.Her2 activates NF-kappaB and induces invasion through the canonical pathway involving IKKalpha.Evidence for tissue factor phosphorylation and its correlation with protease-activated receptor expression and the prognosis of primary breast cancer.GPCRs and cancerN-linked glycosylation of protease-activated receptor-1 second extracellular loop: a critical determinant for ligand-induced receptor activation and internalizationIdentification of a metalloprotease-chemokine signaling system in the ovarian cancer microenvironment: implications for antiangiogenic therapy.Mesotrypsin promotes malignant growth of breast cancer cells through shedding of CD109Withacnistin inhibits recruitment of STAT3 and STAT5 to growth factor and cytokine receptors and induces regression of breast tumoursAntitumor activity of phenethyl isothiocyanate in HER2-positive breast cancer models.Constructing the HBV-human protein interaction network to understand the relationship between HBV and hepatocellular carcinoma.Par genes: molecular probes to pathological assessment in breast cancer progressionMigration of growth factor-stimulated epithelial and endothelial cells depends on EGFR transactivation by ADAM17.Cell surface heparan sulfate proteoglycans control adhesion and invasion of breast carcinoma cells.Suppression of Heregulin-β1/HER2-Modulated Invasive and Aggressive Phenotype of Breast Carcinoma by Pterostilbene via Inhibition of Matrix Metalloproteinase-9, p38 Kinase Cascade and Akt Activation.Proteinase-Activated Receptor 1 (PAR1) regulates leukemic stem cell functionsMicroRNA-31 controls G protein alpha-13 (GNA13) expression and cell invasion in breast cancer cellsG Protein-Coupled Receptor and RhoA-Stimulated Transcriptional Responses: Links to Inflammation, Differentiation, and Cell Proliferation.A Her2-let-7-β2-AR circuit affects prognosis in patients with Her2-positive breast cancer.Expression of MMP-1/PAR-1 and patterns of invasion in oral squamous cell carcinoma as potential prognostic markersRecycling and Endosomal Sorting of Protease-activated Receptor-1 Is Distinctly Regulated by Rab11A and Rab11B Proteins.Kallikrein 6 is a novel molecular trigger of reactive astrogliosis.Matrix metalloprotease-1a promotes tumorigenesis and metastasis.Protease-activated receptors (PARs)--biology and role in cancer invasion and metastasis.Clinical significance and novel mechanism of action of kallikrein 6 in glioblastoma.STAT3-mediated coincidence detection regulates noncanonical immediate early gene induction.Thrombin stimulation of inflammatory breast cancer cells leads to aggressiveness via the EGFR-PAR1-Pak1 pathwayCaveolae are required for protease-selective signaling by protease-activated receptor-1.PAR1 is selectively over expressed in high grade breast cancer patients: a cohort studySignal integration: a framework for understanding the efficacy of therapeutics targeting the human EGFR familybeta-Arrestin mediates beta1-adrenergic receptor-epidermal growth factor receptor interaction and downstream signaling.
P2860
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P2860
Persistent transactivation of EGFR and ErbB2/HER2 by protease-activated receptor-1 promotes breast carcinoma cell invasion.
description
2008 nî lūn-bûn
@nan
2008 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի մարտին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Persistent transactivation of ...... reast carcinoma cell invasion.
@ast
Persistent transactivation of ...... reast carcinoma cell invasion.
@en
type
label
Persistent transactivation of ...... reast carcinoma cell invasion.
@ast
Persistent transactivation of ...... reast carcinoma cell invasion.
@en
prefLabel
Persistent transactivation of ...... reast carcinoma cell invasion.
@ast
Persistent transactivation of ...... reast carcinoma cell invasion.
@en
P2093
P2860
P356
P1433
P1476
Persistent transactivation of ...... reast carcinoma cell invasion.
@en
P2093
P2860
P2888
P304
P356
10.1038/ONC.2008.84
P407
P577
2008-03-31T00:00:00Z
P5875
P6179
1023678086