A unique heparin-binding domain in the envelope protein of the neuropathogenic PVC-211 murine leukemia virus may contribute to its brain capillary endothelial cell tropism.
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Heparan Sulfate Proteoglycans Mediate Attachment and Entry of Human T-Cell Leukemia Virus Type 1 Virions into CD4+ T CellsIdentification of the Receptor Binding Domain of the Mouse Mammary Tumor Virus Envelope ProteinA Mouse-Passaged Dengue Virus Strain with Reduced Affinity for Heparan Sulfate Causes Severe Disease in Mice by Establishing Increased Systemic Viral LoadsMurine endogenous retrovirusesHost range and receptor binding properties of vectors bearing feline leukemia virus subgroup B envelopes can be modulated by envelope sequences outside of the receptor binding domain.Importance of macrophage inflammatory protein-1α and splenic macrophages in neurodegeneration induced by PVC-211 murine leukemia virus.Primate gammaretroviruses require an ancillary factor not required for murine gammaretroviruses to infect BHK cellsNaturally Occurring Polymorphisms of the Mouse Gammaretrovirus Receptors CAT-1 and XPR1 Alter Virus Tropism and Pathogenicity.Disparate regions of envelope protein regulate syncytium formation versus spongiform encephalopathy in neurological disease induced by murine leukemia virus TR.Changes in viral protein function that accompany retroviral endogenization.The other transmissible spongiform encephalopathies.Symmetry-related clustering of positive charges is a common mechanism for heparan sulfate binding in enteroviruses.Adeno-associated viral vectors based on serotype 3b use components of the fibroblast growth factor receptor signaling complex for efficient transductionNeurodegeneration induced by PVC-211 murine leukemia virus is associated with increased levels of vascular endothelial growth factor and macrophage inflammatory protein 1 alpha and is inhibited by blocking activation of microgliaHeparin binds to murine leukemia virus and inhibits Env-independent attachment and infectionG100R mutation within 4070A murine leukemia virus Env increases virus receptor binding, kinetics of entry, and viral transduction efficiency.Heparan sulfate-independent infection attenuates high-neurovirulence GDVII virus-induced encephalitis.Human rhinovirus type 89 variants use heparan sulfate proteoglycan for cell attachment.
P2860
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P2860
A unique heparin-binding domain in the envelope protein of the neuropathogenic PVC-211 murine leukemia virus may contribute to its brain capillary endothelial cell tropism.
description
2001 nî lūn-bûn
@nan
2001 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
A unique heparin-binding domai ...... lary endothelial cell tropism.
@ast
A unique heparin-binding domai ...... lary endothelial cell tropism.
@en
type
label
A unique heparin-binding domai ...... lary endothelial cell tropism.
@ast
A unique heparin-binding domai ...... lary endothelial cell tropism.
@en
prefLabel
A unique heparin-binding domai ...... lary endothelial cell tropism.
@ast
A unique heparin-binding domai ...... lary endothelial cell tropism.
@en
P2093
P2860
P1433
P1476
A unique heparin-binding domai ...... lary endothelial cell tropism.
@en
P2093
P2860
P304
12439-12445
P356
10.1128/JVI.75.24.12439-12445.2001
P577
2001-12-01T00:00:00Z