Quantitative fitness analysis shows that NMD proteins and many other protein complexes suppress or enhance distinct telomere cap defects. - Wikidata - wikimedia - TriplyDB

Quantitative fitness analysis shows that NMD proteins and many other protein complexes suppress or enhance distinct telomere cap defects.

Quantitative fitness analysis shows that NMD proteins and many other protein complexes suppress or enhance distinct telomere cap defects.

http://www.wikidata.org/entity/Q33872233

about

PRECOG: a tool for automated extraction and visualization of fitness components in microbial growth phenomics.Costs, benefits and redundant mechanisms of adaption to chronic low-dose stress in yeast Analysis of time-resolved gene expression measurements across individuals Colony-live--a high-throughput method for measuring microbial colony growth kinetics--reveals diverse growth effects of gene knockouts in Escherichia coli.A novel checkpoint and RPA inhibitory pathway regulated by Rif1 The 9-1-1 checkpoint clamp stimulates DNA resection by Dna2-Sgs1 and Exo1.Rif2 promotes a telomere fold-back structure through Rpd3L recruitment in budding yeast Pentose phosphate pathway function affects tolerance to the G-quadruplex binder TMPyP4 Environmental stresses disrupt telomere length homeostasis.Quantitative Fitness Analysis Identifies exo1∆ and Other Suppressors or Enhancers of Telomere Defects in Schizosaccharomyces pombe.High-Resolution SNP/CGH Microarrays Reveal the Accumulation of Loss of Heterozygosity in Commonly Used Candida albicans Strains Tryptophan-Dependent Control of Colony Formation After DNA Damage via Sea3-Regulated TORC1 Signaling in Saccharomyces cerevisiae.A naturally thermolabile activity compromises genetic analysis of telomere function in Saccharomyces cerevisiae.Fission Yeast SCYL1/2 Homologue Ppk32: A Novel Regulator of TOR Signalling That Governs Survival during Brefeldin A Induced Stress to Protein Trafficking.Everything you ever wanted to know about Saccharomyces cerevisiae telomeres: beginning to end.Systematic Analysis of the DNA Damage Response Network in Telomere Defective Budding Yeast Multiple genetic pathways regulate replicative senescence in telomerase-deficient yeast.Similarities and differences between "uncapped" telomeres and DNA double-strand breaks.Biology of telomeres: lessons from budding yeast.A Functional Link Between Bir1 and the Saccharomyces cerevisiae Ctf19 Kinetochore Complex Revealed Through Quantitative Fitness Analysis.Automated inference procedure for the determination of cell growth parameters.Genome-wide studies of telomere biology in budding yeast Bayesian hierarchical modelling for inferring genetic interactions in yeast.Structural basis for Mep2 ammonium transceptor activation by phosphorylation.Genetic Networks Required to Coordinate Chromosome Replication by DNA Polymerases α, δ, and ε in Saccharomyces cerevisiae.How cells kill a "killer" messenger.Sphingolipids regulate telomere clustering by affecting the transcription of genes involved in telomere homeostasis.Genome-wide analysis to identify pathways affecting telomere-initiated senescence in budding yeast.Interrogation of γ-tubulin alleles using high-resolution fitness measurements reveals a distinct cytoplasmic function in spindle alignment.A quantitative fitness analysis workflow.Fast Bayesian parameter estimation for stochastic logistic growth models.Regulation of telomere metabolism by the RNA processing protein Xrn1.Interplay between nonsense-mediated mRNA decay and DNA damage response pathways reveals that Stn1 and Ten1 are the key CST telomere-cap components.RNA decay, evolution, and the testis.The evolutionarily conserved factor Sus1/ENY2 plays a role in telomere length maintenance.Nuclear import of Cdc13 limits chromosomal capping.Genome-Wide Quantitative Fitness Analysis (QFA) of Yeast Cultures.Vps74 Connects the Golgi Apparatus and Telomeres in Saccharomyces cerevisiae.Cis and trans interactions between genes encoding PAF1 complex and ESCRT machinery components in yeast.The contribution of non-essential Schizosaccharomyces pombe genes to fitness in response to altered nutrient supply and target of rapamycin activity.

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P2860

Quantitative fitness analysis shows that NMD proteins and many other protein complexes suppress or enhance distinct telomere cap defects.

http://www.wikidata.org/entity/Q33872233

description

2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Quantitative fitness analysis ...... distinct telomere cap defects.

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Quantitative fitness analysis ...... distinct telomere cap defects.

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Quantitative fitness analysis ...... distinct telomere cap defects.

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Quantitative fitness analysis ...... distinct telomere cap defects.

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Quantitative fitness analysis ...... distinct telomere cap defects.

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Quantitative fitness analysis ...... distinct telomere cap defects.

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Quantitative fitness analysis ...... distinct telomere cap defects

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A Peter Banks

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Adam Ciesiolka

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Alexander Young

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Darren James Wilkinson

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Laura Maringele

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Min Yu

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Stephen Gregory Addinall

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P2860

RPA-like mammalian Ctc1-Stn1-Ten1 complex binds to single-stranded DNA and protects telomeres independently of the Pot1 pathway

Conserved telomere maintenance component 1 interacts with STN1 and maintains chromosome ends in higher eukaryotes

Components of the Ku-dependent non-homologous end-joining pathway are involved in telomeric length maintenance and telomeric silencing.

High-resolution yeast phenomics resolves different physiological features in the saline response

Telomere length as a quantitative trait: genome-wide survey and genetic mapping of telomere length-control genes in yeast.

Saccharomyces cerevisiae Ebs1p is a putative ortholog of human Smg7 and promotes nonsense-mediated mRNA decay.

Cdc13p: a single-strand telomeric DNA-binding protein with a dual role in yeast telomere maintenance.

Ku interacts with telomerase RNA to promote telomere addition at native and broken chromosome ends.

Cdc13 cooperates with the yeast Ku proteins and Stn1 to regulate telomerase recruitment

The role of Stn1p in Saccharomyces cerevisiae telomere capping can be separated from its interaction with Cdc13p.

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