Protective effect of resin adsorption on septic plasma-induced tubular injury. - Wikidata - wikimedia - TriplyDB

Protective effect of resin adsorption on septic plasma-induced tubular injury.

Protective effect of resin adsorption on septic plasma-induced tubular injury.

http://www.wikidata.org/entity/Q33873370

about

Septic AKI in ICU patients. diagnosis, pathophysiology, and treatment type, dosing, and timing: a comprehensive review of recent and future developments Epithelial transport during septic acute kidney injury.Effect of therapeutic plasma exchange on plasma levels and total removal of adipokines and inflammatory markers.Septic acute kidney injury: molecular mechanisms and the importance of stratification and targeting therapy.The implications and management of septic acute kidney injury.IMMUNE CELL PHENOTYPE AND FUNCTION IN SEPSIS.Cellular apoptosis in the cardiorenal axis.Monocytes, peripheral blood mononuclear cells, and THP-1 cells exhibit different cytokine expression patterns following stimulation with lipopolysaccharide Extracellular vesicles in renal disease.Adsorptive modulation of inflammatory mediators dampens endothelial cell activation.Sensors and hybrid therapies: a new approach with automated citrate anticoagulation.Hemoadsorption corrects hyperresistinemia and restores anti-bacterial neutrophil function.Heart-kidney crosstalk and role of humoral signaling in critical illness Effects of coupled plasma filtration adsorption on septic patients with multiple organ dysfunction syndrome.Resveratrol-poor red wines modulate SIRT1 in human renal cells.Effects of electroacupuncture pretreatment on inflammatory response and acute kidney injury in endotoxaemic rats.Exosomes and microvesicles in normal physiology, pathophysiology, and renal diseases.Protective effects of propofol on endotoxemia-induced acute kidney injury in rats.The Role of Endotoxin in the Setting of Cardiorenal Syndrome Type 5.Endotoxin Effects on Cardiac and Renal Functions and Cardiorenal Syndromes.Lipopolysaccharide Mediates the Destruction of Intercellular Tight Junction among Renal Tubular Epithelial Cells via RhoT1/SMAD-4/JAM-3 Pathway.Endothelial progenitor cell-derived extracellular vesicles protect from complement-mediated mesangial injury in experimental anti-Thy1.1 glomerulonephritis

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P2860

Protective effect of resin adsorption on septic plasma-induced tubular injury.

http://www.wikidata.org/entity/Q33873370

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Protective effect of resin adsorption on septic plasma-induced tubular injury.

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Protective effect of resin adsorption on septic plasma-induced tubular injury.

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Protective effect of resin adsorption on septic plasma-induced tubular injury.

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Protective effect of resin adsorption on septic plasma-induced tubular injury.

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Protective effect of resin adsorption on septic plasma-induced tubular injury.

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Protective effect of resin adsorption on septic plasma-induced tubular injury.

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Protective effect of resin adsorption on septic plasma-induced tubular injury

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Carola Lauritano

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Ciro Tetta

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Dinna Cruz

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Federico Figliolini

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Giuseppe Paolo Segoloni

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Luigi Biancone

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Massimo De Cal

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Silvia Beltramo

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P2860

Acute renal failure - definition, outcome measures, animal models, fluid therapy and information technology needs: the Second International Consensus Conference of the Acute Dialysis Quality Initiative (ADQI) Group

Platelet and soluble CD40L in meningococcal sepsis.

Inflammatory cytokines induce apoptotic and necrotic cell shedding from human proximal tubular epithelial cell monolayers

Coexpressed nitric oxide synthase and apical beta(1) integrins influence tubule cell adhesion after cytokine-induced injury.

Megalin and cubilin: synergistic endocytic receptors in renal proximal tubule.

Endothelial progenitor cell derived microvesicles activate an angiogenic program in endothelial cells by a horizontal transfer of mRNA.

CD40 contributes to lethality in acute sepsis: in vivo role for CD40 in innate immunity.

Prevention of neutrophil extravasation by hepatocyte growth factor leads to attenuations of tubular apoptosis and renal dysfunction in mouse ischemic kidneys

Epithelial polarity and tubulogenesis in vitro.

Recent advances in the pathophysiology of ischemic acute renal failure.

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10.1186/CC8835

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1

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Giovanni Camussi

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2010-01-11T00:00:00Z

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