Persistent virus infection inhibits type I interferon production by plasmacytoid dendritic cells to facilitate opportunistic infections.
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The virome in host health and diseaseInnate immune responses and rapid control of inflammation in African green monkeys treated or not with interferon-alpha during primary SIVagm infectionc-FLIP-Short reduces type I interferon production and increases viremia with coxsackievirus B3Inhibition of Innate Immune Responses Is Key to Pathogenesis by ArenavirusesInnate and Adaptive Immune Regulation During Chronic Viral InfectionsNegative regulation of interferon-β gene expression during acute and persistent virus infectionsImmune responses during spontaneous control of HIV and AIDS: what is the hope for a cure?Anatomy of viral persistence.Treatment with a sphingosine analog does not alter the outcome of a persistent virus infection.Innate immune recognition and activation during HIV infection.Modulation of host innate and adaptive immune defenses by cytomegalovirus: timing is everythingExhausted CD8 T cells downregulate the IL-18 receptor and become unresponsive to inflammatory cytokines and bacterial co-infections.Increased antigen presentation but impaired T cells priming after upregulation of interferon-beta induced by lipopolysaccharides is mediated by upregulation of B7H1 and GITRLPlasmacytoid dendritic cells: recent progress and open questionsOut-of-sequence signal 3 as a mechanism for virus-induced immune suppression of CD8 T cell responses.Resident viruses and their interactions with the immune systemCell-intrinsic transforming growth factor-beta signaling mediates virus-specific CD8+ T cell deletion and viral persistence in vivoThe role of dendritic cells in viral persistenceNegative regulation of type I IFN expression by OASL1 permits chronic viral infection and CD8⁺ T-cell exhaustion.Induction and function of virus-specific CD4+ T cell responses.Constitutive but not inducible attenuation of transforming growth factor β signaling increases natural killer cell responses without directly affecting dendritic cells early after persistent viral infection.Direct infection of dendritic cells during chronic viral infection suppresses antiviral T cell proliferation and induces IL-10 expression in CD4 T cells.Plasmacytoid dendritic cells and the control of herpesvirus infections.Inflammatory monocytes recruited to the liver within 24 hours after virus-induced inflammation resemble Kupffer cells but are functionally distinct.New insights into type I interferon and the immunopathogenesis of persistent viral infections.Type I interferon negatively controls plasmacytoid dendritic cell numbers in vivo.Plasmacytoid dendritic cells control T-cell response to chronic viral infection.Differential roles for RIG-I-like receptors and nucleic acid-sensing TLR pathways in controlling a chronic viral infection.Wild-type coxsackievirus infection dramatically alters the abundance, heterogeneity, and immunostimulatory capacity of conventional dendritic cells in vivoPlasmacytoid dendritic cells are productively infected and activated through TLR-7 early after arenavirus infectionLate interleukin-6 escalates T follicular helper cell responses and controls a chronic viral infection.The role of IL-10 in regulating immunity to persistent viral infectionsCD8+ T cell-derived IFN-γ prevents infection by a second heterologous virus.CD28 Deficiency Enhances Type I IFN Production by Murine Plasmacytoid Dendritic Cells.Type I interferons: diversity of sources, production pathways and effects on immune responses.Timing and magnitude of type I interferon responses by distinct sensors impact CD8 T cell exhaustion and chronic viral infection.Human immunodeficiency virus type 1 mediates global disruption of innate antiviral signaling and immune defenses within infected cells.Viral infection induces de novo lesions of coronary allograft vasculopathy through a natural killer cell-dependent pathway.Chronic but not acute virus infection induces sustained expansion of myeloid suppressor cell numbers that inhibit viral-specific T cell immunity.Human plasmacytoid dendritic cells sense lymphocytic choriomeningitis virus-infected cells in vitro
P2860
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P2860
Persistent virus infection inhibits type I interferon production by plasmacytoid dendritic cells to facilitate opportunistic infections.
description
2008 nî lūn-bûn
@nan
2008 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Persistent virus infection inh ...... tate opportunistic infections.
@ast
Persistent virus infection inh ...... tate opportunistic infections.
@en
type
label
Persistent virus infection inh ...... tate opportunistic infections.
@ast
Persistent virus infection inh ...... tate opportunistic infections.
@en
prefLabel
Persistent virus infection inh ...... tate opportunistic infections.
@ast
Persistent virus infection inh ...... tate opportunistic infections.
@en
P2093
P2860
P1433
P1476
Persistent virus infection inh ...... tate opportunistic infections.
@en
P2093
Elina I Zuniga
Lauren Mack
Li-Ying Liou
Marilyn Mendoza
Michael B A Oldstone
P2860
P304
P356
10.1016/J.CHOM.2008.08.016
P577
2008-10-01T00:00:00Z