MicroRNAs are potential therapeutic targets in fibrosing kidney disease: lessons from animal models. - Wikidata - wikimedia - TriplyDB

MicroRNAs are potential therapeutic targets in fibrosing kidney disease: lessons from animal models.

MicroRNAs are potential therapeutic targets in fibrosing kidney disease: lessons from animal models.

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Recent advances in understanding of chronic kidney disease Peroxisomes and Kidney Injury Increased circulating miR-21 levels are associated with kidney fibrosis.MicroRNA-205 inhibits renal cells apoptosis via targeting CMTM4.What is damaging the kidney in lupus nephritis?MiR-21-5p Links Epithelial-Mesenchymal Transition Phenotype with Stem-Like Cell Signatures via AKT Signaling in Keloid Keratinocytes.MicroRNA expression signature and the therapeutic effect of the microRNA‑21 antagomir in hypertrophic scarring.Dysregulated microRNAs involved in contrast-induced acute kidney injury in rat and human.Plasma miR-221/222 Family as Novel Descriptive and Prognostic Biomarkers for Glioma.

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MicroRNAs are potential therapeutic targets in fibrosing kidney disease: lessons from animal models.

http://www.wikidata.org/entity/Q33877294

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2013 nî lūn-bûn

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2013 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2013 թվականի հունվարին հրատարակված գիտական հոդված

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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MicroRNAs are potential therap ...... e: lessons from animal models.

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MicroRNAs are potential therap ...... e: lessons from animal models.

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MicroRNAs are potential therap ...... e: lessons from animal models.

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MicroRNAs are potential therap ...... e: lessons from animal models.

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MicroRNAs are potential therap ...... e: lessons from animal models.

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Drug discovery today. Disease models

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MicroRNAs are potential therap ...... e: lessons from animal models.

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Didier Portilla

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Jeremy S Duffield

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Monica Grafals

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P2860

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

Circulating microRNAs as stable blood-based markers for cancer detection.

MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts

c-Myc suppression of miR-23a/b enhances mitochondrial glutaminase expression and glutamine metabolism

Effects of long-term fenofibrate therapy on cardiovascular events in 9795 people with type 2 diabetes mellitus (the FIELD study): randomised controlled trial

Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis

Serum amyloid P inhibits fibrosis through Fc gamma R-dependent monocyte-macrophage regulation in vivo.

Identification of a microRNA signature of renal ischemia reperfusion injury.

OncomiR addiction in an in vivo model of microRNA-21-induced pre-B-cell lymphoma.

Functional rescue of the glomerulosclerosis phenotype in Mpv17 mice by transgenesis with the human Mpv17 homologue.

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