Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation.
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Modulation of inflammasome activity by Porphyromonas gingivalis in periodontitis and associated systemic diseasesDangerous Liaisons: Caspase-11 and Reactive Oxygen Species Crosstalk in Pathogen EliminationPeriodontitis: from microbial immune subversion to systemic inflammationThe enduring importance of animal models in understanding periodontal diseaseImmune dysregulation mediated by the oral microbiome: potential link to chronic inflammation and atherosclerosis.Polymicrobial synergy and dysbiosis in inflammatory disease.Distinct gene signatures in aortic tissue from ApoE-/- mice exposed to pathogens or Western diet.Mechanisms of Bone Resorption in Periodontitis.On the translocation of bacteria and their lipopolysaccharides between blood and peripheral locations in chronic, inflammatory diseases: the central roles of LPS and LPS-induced cell death.A Dual Role for P2X7 Receptor during Porphyromonas gingivalis InfectionUnprimed, M1 and M2 Macrophages Differentially Interact with Porphyromonas gingivalis.Porphyromonas gingivalis virulence factors involved in subversion of leukocytes and microbial dysbiosisIL-1R/TLR2 through MyD88 Divergently Modulates Osteoclastogenesis through Regulation of Nuclear Factor of Activated T Cells c1 (NFATc1) and B Lymphocyte-induced Maturation Protein-1 (Blimp1).Associations between Periodontal Microbiota and Death Rates.Signaling events in pathogen-induced macrophage foam cell formation.Immune modulation by bacterial outer membrane vesicles.Metabolic Remodeling, Inflammasome Activation, and Pyroptosis in Macrophages Stimulated by Porphyromonas gingivalis and Its Outer Membrane Vesicles.Differential Regulation of Mas-Related G Protein-Coupled Receptor X2-Mediated Mast Cell Degranulation by Antimicrobial Host Defense Peptides and Porphyromonas gingivalis Lipopolysaccharide.Variants of Porphyromonas gingivalis lipopolysaccharide alter lipidation of autophagic protein, microtubule-associated protein 1 light chain 3, LC3.The role of Toll-like receptors in periodontitis.Clinical Isolates of Pseudomonas aeruginosa from Chronically Infected Cystic Fibrosis Patients Fail To Activate the Inflammasome during Both Stable Infection and Pulmonary Exacerbation.Endotoxemia-menace, marker, or mistake?Microbiota, Immune Subversion, and Chronic Inflammation.Porphyromonas gingivalis Outer Membrane Vesicles Induce Selective Tumor Necrosis Factor Tolerance in a Toll-Like Receptor 4- and mTOR-Dependent MannerContradictory roles of Porphyromonas gingivalis gingipains in caspase-1 activation.Maternal modifiers of the infant gut microbiota: metabolic consequences.Importance of heterogeneity in Porhyromonas gingivalis lipopolysaccharide lipid A in tissue specific inflammatory signalling.
P2860
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P2860
Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Distinct lipid a moieties cont ...... pecific vascular inflammation.
@ast
Distinct lipid a moieties cont ...... pecific vascular inflammation.
@en
type
label
Distinct lipid a moieties cont ...... pecific vascular inflammation.
@ast
Distinct lipid a moieties cont ...... pecific vascular inflammation.
@en
prefLabel
Distinct lipid a moieties cont ...... pecific vascular inflammation.
@ast
Distinct lipid a moieties cont ...... pecific vascular inflammation.
@en
P2093
P2860
P1433
P1476
Distinct lipid a moieties cont ...... pecific vascular inflammation.
@en
P2093
Caroline A Genco
Carolyn Kramer
Connie Slocum
Cynthia V Gudino
Ellen O Weinberg
George Papadopoulos
Richard P Darveau
Stephen R Coats
P2860
P304
P356
10.1371/JOURNAL.PPAT.1004215
P577
2014-07-10T00:00:00Z