Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolin
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Effect of the p53-tristetraprolin-stathmin-1 pathway on trophoblasts at maternal-fetal interface.RNase L attenuates mitogen-stimulated gene expression via transcriptional and post-transcriptional mechanisms to limit the proliferative responseTristetraprolin inhibits the growth of human glioma cells through downregulation of urokinase plasminogen activator/urokinase plasminogen activator receptor mRNAsTristetraprolin mediates anti-inflammatory effects of nicotine in lipopolysaccharide-stimulated macrophages.Ectopic over-expression of tristetraprolin in human cancer cells promotes biogenesis of let-7 by down-regulation of Lin28Tristetraprolin impairs myc-induced lymphoma and abolishes the malignant stateThe role of tristetraprolin in cancer and inflammation.Resveratrol Induces Glioma Cell Apoptosis through Activation of Tristetraprolin.Tristetraprolin induces cell cycle arrest in breast tumor cells through targeting AP-1/c-Jun and NF-κB pathwayTumor suppressor p53 plays a key role in induction of both tristetraprolin and let-7 in human cancer cells.Tristetraprolin (TTP): interactions with mRNA and proteins, and current thoughts on mechanisms of action.Tristetraprolin down-regulates IL-23 expression in colon cancer cells.Tristetraprolin: roles in cancer and senescence.Regulation and functions of mammalian LATS/NDR kinases: looking beyond canonical Hippo signalling.Expression of proviral integration site for Moloney murine leukemia virus 1 (Pim-1) is post-transcriptionally regulated by tristetraprolin in cancer cells.Tristetraprolin inhibits mitochondrial function through suppression of α-Synuclein expression in cancer cells.Dysregulation of TTP and HuR plays an important role in cancers.The LATS1 and LATS2 tumor suppressors: beyond the Hippo pathway.Tristetraprolin down-regulates IL-17 through mRNA destabilization.A synonymous polymorphism of the Tristetraprolin (TTP) gene, an AU-rich mRNA-binding protein, affects translation efficiency and response to Herceptin treatment in breast cancer patients.Casein kinase 2 regulates the mRNA-destabilizing activity of tristetraprolin.Tristetraprolin suppresses AHRR expression through mRNA destabilization.The role of RNA-binding protein tristetraprolin in cancer and immunity.ZFP36L1 and ZFP36L2 inhibit cell proliferation in a cyclin D-dependent and p53-independent manner.
P2860
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P2860
Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolin
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2010 nî lūn-bûn
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2010 թուականի Մարտին հրատարակուած գիտական յօդուած
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2010 թվականի մարտին հրատարակված գիտական հոդված
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年學術文章
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Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolin
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Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolin
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label
Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolin
@ast
Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolin
@en
prefLabel
Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolin
@ast
Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolin
@en
P2093
P2860
P356
P1476
Stability of the LATS2 tumor suppressor gene is regulated by tristetraprolin
@en
P2093
Byung Ju Lee
Chae Won Kim
Dae Hwa Choi
Ho Seok Suh
Hong Kyeung Kim
Hyo Jeong Kim
Hyun Hee Lee
Jeong Woo Park
Mai-Tram Vo
Myoung Seok Ko
P2860
P304
17329-17337
P356
10.1074/JBC.M109.094235
P407
P577
2010-03-24T00:00:00Z