D-mef2 is a target for Tinman activation during Drosophila heart development.
about
Translational machinery of the chaetognath Spadella cephaloptera: a transcriptomic approach to the analysis of cytosolic ribosomal protein genes and their expressionMyocyte enhancer factor 2C and Nkx2-5 up-regulate each other's expression and initiate cardiomyogenesis in P19 cellsMutations in the cardiac transcription factor NKX2.5 affect diverse cardiac developmental pathwaysFunctional analyses of three Csx/Nkx-2.5 mutations that cause human congenital heart diseaseSmad proteins function as co-modulators for MEF2 transcriptional regulatory proteinsMEF2C is activated by multiple mechanisms in a subset of T-acute lymphoblastic leukemia cell linesThe cardiac transcription factors Nkx2-5 and GATA-4 are mutual cofactors.Bone morphogenetic proteins induce cardiomyocyte differentiation through the mitogen-activated protein kinase kinase kinase TAK1 and cardiac transcription factors Csx/Nkx-2.5 and GATA-4Specification of the somatic musculature in DrosophilaCharacterization of homo- and heterodimerization of cardiac Csx/Nkx2.5 homeoproteinTranscriptional integration of competence modulated by mutual repression generates cell-type specificity within the cardiogenic mesodermExpression, regulation, and requirement of the toll transmembrane protein during dorsal vessel formation in Drosophila melanogasterGATA-4 and Nkx-2.5 coactivate Nkx-2 DNA binding targets: role for regulating early cardiac gene expression.Identification and analysis of vnd/NK-2 homeodomain binding sites in genomic DNA.The transcription factor Mef2 is required for normal circadian behavior in DrosophilaSequence-specific DNA binding by the vnd/NK-2 homeodomain of Drosophila.Genome-wide screens for in vivo Tinman binding sites identify cardiac enhancers with diverse functional architectures.The ATP-sensitive potassium (KATP) channel-encoded dSUR gene is required for Drosophila heart function and is regulated by tinman.A functional genomic screen for cardiogenic genes using RNA interference in developing Drosophila embryosThe multitype zinc-finger protein U-shaped functions in heart cell specification in the Drosophila embryoMolecular mechanisms of chamber-specific myocardial gene expression: transgenic analysis of the ANF promoter.The myogenic regulatory gene Mef2 is a direct target for transcriptional activation by Twist during Drosophila myogenesisSmad proteins act in combination with synergistic and antagonistic regulators to target Dpp responses to the Drosophila mesodermOogenic function of the myogenic factor D-MEF2: negative regulation of the decapentaplegic receptor gene thick veinsThe Drosophila Transcription Factors Tinman and Pannier Activate and Collaborate with Myocyte Enhancer Factor-2 to Promote Heart Cell Fate.Differential regulation of mesodermal gene expression by Drosophila cell type-specific Forkhead transcription factors.Cellular Mechanisms of Drosophila Heart Morphogenesis.Cardiac expression of the Drosophila Sulphonylurea receptor gene is regulated by an intron enhancer dependent upon the NK homeodomain factor Tinman.The GATA factor Serpent cross-regulates lozenge and u-shaped expression during Drosophila blood cell developmentThe heterotrimeric protein Go is required for the formation of heart epithelium in Drosophila.Transcription of Drosophila troponin I gene is regulated by two conserved, functionally identical, synergistic elementsGenome-Wide Approaches to Drosophila Heart DevelopmentAlternative requirements for Vestigial, Scalloped, and Dmef2 during muscle differentiation in Drosophila melanogaster.A genomewide survey of basic helix-loop-helix factors in Drosophila.Upregulation of the Drosophila Friend of GATA gene U-shaped by JAK/STAT signaling maintains lymph gland prohemocyte potency.From molecular mechanisms of cardiac development to genetic substrate of congenital heart diseases.Drosophila models of cardiac disease.The myogenic repressor gene Holes in muscles is a direct transcriptional target of Twist and Tinman in the Drosophila embryonic mesoderm.Two functionally identical modular enhancers in Drosophila troponin T gene establish the correct protein levels in different muscle types.Transactivation in Drosophila of human enhancers by human transcription factors involved in congenital heart diseases.
P2860
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P2860
D-mef2 is a target for Tinman activation during Drosophila heart development.
description
1997 nî lūn-bûn
@nan
1997 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
name
D-mef2 is a target for Tinman activation during Drosophila heart development.
@ast
D-mef2 is a target for Tinman activation during Drosophila heart development.
@en
type
label
D-mef2 is a target for Tinman activation during Drosophila heart development.
@ast
D-mef2 is a target for Tinman activation during Drosophila heart development.
@en
prefLabel
D-mef2 is a target for Tinman activation during Drosophila heart development.
@ast
D-mef2 is a target for Tinman activation during Drosophila heart development.
@en
P2093
P2860
P356
P1433
P1476
D-mef2 is a target for Tinman activation during Drosophila heart development.
@en
P2093
P2860
P304
P356
10.1093/EMBOJ/16.3.515
P407
P577
1997-02-01T00:00:00Z