Mechanism by which HLA-DR4 regulates sex-bias of arthritis in humanized mice.
about
Arthritis susceptibility and the gut microbiome.Loss of sex and age driven differences in the gut microbiome characterize arthritis-susceptible 0401 mice but not arthritis-resistant 0402 miceFine specificity of anti-citrullinated peptide antibodies discloses a heterogeneous antibody population in rheumatoid arthritis.DRB1*0402 may influence arthritis by promoting naive CD4+ T-cell differentiation in to regulatory T cells.Epitope analysis of the collagen type V-specific T cell response in lung transplantation reveals an HLA-DRB1*15 bias in both recipient and donor.To B or not to B: role of B cells in pathogenesis of arthritis in HLA transgenic mice.Characterization of a novel and spontaneous mouse model of inflammatory arthritis.The gut microbiome in autoimmunity: Sex matters.Cytokines pre-determined by genetic factors are involved in pathogenesis of Rheumatoid arthritis.Microchimerism: tolerance vs. sensitization.An expansion of rare lineage intestinal microbes characterizes rheumatoid arthritis.Cigarette Smoke Induces Immune Responses to Vimentin in both, Arthritis-Susceptible and -Resistant Humanized Mice.HLA class II molecules influence susceptibility versus protection in inflammatory diseases by determining the cytokine profileHigher Estradiol and Lower Dehydroepiandrosterone-Sulfate Levels Are Associated with Pulmonary Arterial Hypertension in Men.Immunosuppression by co-stimulatory molecules: inhibition of CD2-CD48/CD58 interaction by peptides from CD2 to suppress progression of collagen-induced arthritis in miceB cells as effectors and regulators of sex-biased arthritis.The degree of CD4+ T cell autoreactivity determines cellular pathways underlying inflammatory arthritisCellular and humoral immunity in arthritis are profoundly influenced by the interaction between cigarette smoke effects and host HLA-DR and DQ genes.The long-term marriage between autoimmunity and internal medicine: a homage to Manuel Carlos Dias.CD74/DQA1 dimers predispose to the development of arthritis in humanized mice.Characterization of a novel and spontaneous mouse model of inflammatory arthritis.Suppression of Inflammatory Arthritis by Human Gut-Derived Prevotella histicola in Humanized Mice.Sex Hormones Determine Immune Response
P2860
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P2860
Mechanism by which HLA-DR4 regulates sex-bias of arthritis in humanized mice.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Mechanism by which HLA-DR4 regulates sex-bias of arthritis in humanized mice.
@ast
Mechanism by which HLA-DR4 regulates sex-bias of arthritis in humanized mice.
@en
type
label
Mechanism by which HLA-DR4 regulates sex-bias of arthritis in humanized mice.
@ast
Mechanism by which HLA-DR4 regulates sex-bias of arthritis in humanized mice.
@en
prefLabel
Mechanism by which HLA-DR4 regulates sex-bias of arthritis in humanized mice.
@ast
Mechanism by which HLA-DR4 regulates sex-bias of arthritis in humanized mice.
@en
P2093
P2860
P1476
Mechanism by which HLA-DR4 regulates sex-bias of arthritis in humanized mice.
@en
P2093
Chella S David
Harvinder Luthra
Marie Griffiths
Marshall Behrens
Theodore Trejo
Veena Taneja
P2860
P356
10.1016/J.JAUT.2009.12.007
P577
2010-01-12T00:00:00Z